alcoholism/phosphatase

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Prostatic acid phosphatase, aspermia, and alcoholism in rape cases.

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In a few alleged rape cases, examination of vaginal secretions will be negative for spermatozoa but positive for significant levels of prostatic acid phosphatase. These laboratory results can occur in cases in which the accused is known to have sired children. The most common etiologic factors for

Association of Protein Phosphatase PPM1G With Alcohol Use Disorder and Brain Activity During Behavioral Control in a Genome-Wide Methylation Analysis.

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OBJECTIVE The genetic component of alcohol use disorder is substantial, but monozygotic twin discordance indicates a role for nonheritable differences that could be mediated by epigenetics. Despite growing evidence associating epigenetics and psychiatric disorders, it is unclear how epigenetics,

Ileum brush border alkaline phosphatase activity in an experimental model of chronic alcoholism after small bowel proximal resection in the rat.

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Literature reports that chronically ingested ethanol induces changes in the morphology of the small bowel mucous membranes. It has a topical toxic effect on the epithelium of the proximal jejunum and a blood-borne effect on the epithelium of the ileum because its absorption is almost complete in the

Protein Tyrosine Phosphatase β/ζ and alcohol use disorder: A Commentary.

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The need to find new pharmacological targets for treating alcohol use disorder (AUD) has been an extensive effort in alcohol research. Changes in immune function due to AUD may represent an exploitable target for developing new medications to treat AUD, since the cytotoxic effect of alcohol has

[Gamma-glutamyl-transpeptidase, alkaline phosphatase and median erythrocyte volume in the diagnosis of chronic alcoholism].

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Disulfiram reactivates latent HIV-1 expression through depletion of the phosphatase and tensin homolog.

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OBJECTIVE Disulfiram (DSF), an inhibitor of acetaldehyde dehydrogenase that is used for the treatment of alcoholism, was shown to reactivate latent HIV-1 expression in a primary cell model of virus latency and is currently being assessed in a clinical trial for its potential to deplete the latent

Inhibition of protein phosphatase 1 reverses alcohol-induced ciliary dysfunction.

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Airway mucociliary clearance is a first-line defense of the lung against inhaled particles and debris. Among individuals with alcohol use disorders, there is an increase in lung diseases. We previously identified that prolonged alcohol exposure impairs mucociliary clearance, known as alcohol-induced

An association study revealed substantial effects of dominance, epistasis and substance dependence co-morbidity on alcohol dependence symptom count.

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Alcohol dependence is a complex disease involving polygenes, environment and their interactions. Inadequate consideration of these interactions may have hampered the progress on genome-wide association studies of alcohol dependence. By using the dataset of the Study of Addiction: Genetics and

Population pharmacokinetics of extended-release injectable naltrexone (XR-NTX) in patients with alcohol dependence.

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OBJECTIVE Injectable extended-release naltrexone (XR-NTX; Vivitrol) has recently been approved for the treatment of alcohol dependence. A population pharmacokinetic (PPK) analysis examined the possibility of altered pharmacokinetics for naltrexone and its primary metabolite, 6beta-naltrexol, in

Role of Striatal-Enriched Tyrosine Phosphatase in Neuronal Function.

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Striatal-enriched protein tyrosine phosphatase (STEP) is a CNS-enriched protein implicated in multiple neurologic and neuropsychiatric disorders. STEP regulates key signaling proteins required for synaptic strengthening as well as NMDA and AMPA receptor trafficking. Both high and low levels of STEP

Neurologic complications of alcoholism.

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Neurologic and myopathic complications of alcoholism are multiple and diverse, affecting both the central and peripheral nervous systems. In the ED, initial concern is for diagnosing readily reversible causes and ruling out possible life- or limb-threatening etiologies. A rapid assessment of the

Gamma-glutamyltransferase, aspartate aminotransferase and alkaline phosphatase as markers of alcohol consumption in out-patient alcoholics.

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Serum activity of gamma-glutamyltransferase, aspartate aminotransferase and alkaline phosphatase were determined in 316 patients attending an out-patients clinic for treatment of alcoholism. The activity of gamma-glutamyltransferase was raised in 34% and that of aspartate aminotransferase and

Alcoholic liver disease presenting with marked elevation of serum alkaline phosphatase. A combined clinical and pathological study.

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Twenty patients with longstanding alcoholism and biopsy-proven alcoholic liver disease presented with marked elevation of serum alkaline phosphatase (in excess of four times the upper limit of normal). None had a past or present history to suggest pancreatitis or biliary tract disease, nor had any

Screening for disulfiram-induced liver test dysfunction in an inpatient alcoholism program.

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The objective of this study was to report the frequency of disulfiram-related elevations of four commonly used hepatic screening chemistries using a retrospective record review design. An inpatient alcoholism program was selected for the setting. Patients who had initial laboratory values within the

Investigation of DUSP8 and CALCA in alcohol dependence.

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Evidence for genetic linkage to alcohol dependence was found on chromosome 11p15.5 from an autosome-wide scan in a Southwestern Native American population. The purpose of this study was to identify genes that may underlie this linkage signal. Two genes, calcitonin/calcitonin-related polypeptide

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