angioedema/وذمة

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Hereditary angioneurotic edema: clinical and laboratory findings in 58 subjects.

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An in-depth analysis of clinical and laboratory findings in 58 patients affected by hereditary angioneurotic edema (HANE) is reported with special focus on problems related to the diagnosis of the disorder. The functional C1 inhibitor (C1INH) assay is the method of choice in the diagnosis of HANE,

Association of Sjögren's syndrome with hereditary angioneurotic edema: report of a case.

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A case of hereditary angioneurotic edema (HANE) associated with Sjögren's syndrome is presented. One of the members of a pedigree of HANE due to deficiency of C1 inhibitor (C1INH) had a positive titer for anti-SS-A and anti-SS-B antibodies in the serum, complaining of symptom of dry eyes and dry

Contiguous deletion and duplication mutations resulting in type 1 hereditary angioneurotic edema.

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Mutations that cause low antigenic and functional levels of C1 inhibitor protein result in type 1 hereditary angioneurotic edema. This disease is characterized by episodic edema leading to considerable morbidity and sometimes death. We present here two novel mutations in the reactive center coding

[Clinical contribution to the problem of correlations between hereditary angioneurotic edema and pregnancy].

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Hereditary angioneurotic edema (HAE) is an autosomal dominant disease caused by a deficiency of a complement regulatory protein, the C1INH.HAE is clinically characterized by recurrent, self-limited attacks of edema involving the extremities, face, upper respiratory tract or gastrointestinal tract.

Plasmapheresis in hereditary angioneurotic edema and systemic lupus erythematosus.

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A patient had hereditary angioneurotic edema coincident with systemic lupus erythematosus. This condition resulted in loss of hair, arthralgia, leukoplakia, collitis, and a nephrotic syndrome due to proliferative glomerulonephritis. The renal lesion was resistant to treatment with high-dose

A tracheostomy obturator for a patient with hereditary angioneurotic edema.

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This article describes a technique for fabrication of a custom tracheostomy obturator for a patient with hereditary angioneurotic edema and a permanent tracheostoma. The impression technique and fabrication of a custom tracheostoma valve retainer for a laryngectomy patient closely mirrors the

[Facial edema as an earlier presenting sign of giant cell arteritis. Possible relationship with angioedema].

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Giant cell arteritis (GCA) is a chronic granulomatous vasculitis of unknown etiology occurring in the elderly. New-onset headache, scalp tenderness, jaw claudication, temporal artery abnormalities on physical examination, visual symptoms and associated polymyalgia rheumatica represent the most

A case of hereditary angioneurotic edema associated with systemic lupus erythematosus.

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A pedigree of C1 inhibitor (C1 INH) deficiency associated with positive LE cell and an elevated titer of DNA antibodies and antinuclear factor (ANF) and nephropathy was presented. The proband of this family was diagnosed as having definite systemic lupus erythematosus (SLE) after a clinical course

[Acquired angioneurotic edema. Clinical and biological characteristics in 9 patients].

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OBJECTIVE Angioneurotic edema (AE) is a rare but severe disease. Hereditary AE is the more well-known form. The acquired form is exceptional: the symptoms are the same but there are some biologic and treatment differences. We investigated the clinical and biochemical features in nine patients with

Synthesis of C1 inhibitor in fibroblasts from patients with type I and type II hereditary angioneurotic edema.

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Patients with hereditary angioneurotic edema (HANE) have serum levels of functionally active inhibitor of the first component of complement (C1 INH) between 5 and 30% of normal, instead of the 50% expected from the single normal allele. Increases in rates of catabolism have been documented in

A capillary hemostasis mechanism regulated by sympathetic tone and activity via factor VIII or von Willebrand's factor may function as a "capillary gate" and may explain angiodysplasia, angioneurotic edema, and variations in systemic vascular resistance.

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The sympathetic nervous system (SNS) may directly or indirectly govern the release of von Willebrand's factor (VWB), which may be a hormone, from the vascular endothelium, which may be a gland. VWB is known to stabilize the labile VIIIC component of Factor VIII complex, and may thereby determine its

Angioneurotic edema following ingestion of iocetamic acid.

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A case of angioneurotic edema involving the left upper extremity following iocetamic acid ingestion is reported. This is the fifth reported case of a severe cutaneous reaction following ingestion of this material. The author suggests that iocetamic acid not be used in outpatients.

Hereditary angioneurotic edema in the puerperium. A case report.

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Hereditary angioneurotic edema is a potentially fatal condition. Below is described the first reported case of the initial presentation of the condition precipitated by vaginal delivery.

Recurrent colocolic intussusception in a child with hereditary angioneurotic edema: reduction by air enema.

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The authors discuss a case of hereditary angioneurotic edema in a child presenting with recurrent episodes of colocolic intussusception, each successfully reduced by air enema. Although additional manifestations of vasogenic edema are potential risks of the procedure, these were not encountered.

Hereditary angioneurotic edema and Charcot-Marie-Tooth disease in the same family.

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In one family two genetic diseases were transmitted as autosomal dominant traits; hereditary angioneurotic edema was inherited from the paternal side and Charcot-Marie Tooth disease from the maternal side of the family. The conditions occurred separately in 8 and 11 members respectively and together

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