bone resorption/نيكوتين

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The role of HIF-1α in nicotine-induced root and bone resorption during orthodontic tooth movement

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Background: In orthodontic tooth movement (OTM), pseudo-inflammatory processes occur that are similar to those of nicotine-induced periodontitis. Previous studies have shown that nicotine accelerates OTM, but induces periodontal bone loss

Nicotine affects bone resorption and suppresses the expression of cathepsin K, MMP-9 and vacuolar-type H(+)-ATPase d2 and actin organization in osteoclasts.

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Tobacco smoking is an important risk factor for the development of several cancers, osteoporosis, and inflammatory diseases such as periodontitis. Nicotine is one of the major components of tobacco. In previous study, we showed that nicotine inhibits mineralized nodule formation by osteoblasts, and

Morpho-histological assessment of the periodontal support structures under the action of excessive occlusal forces and under the influence of nicotine.

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Smoking and occlusal trauma are two factors that can interfere with bone homeostasis. The aim of this study was to evaluate the histocellular changes occurring in the periodontal ligament and alveolar bone during the action of excessive occlusal forces, and to assess the influence of nicotine on the

[Negative effects of tobacco on the periodontium in a Moroccan population].

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OBJECTIVE Several studies have reported an association between smoking and periodontal destruction. The aim of this study was to investigate the harmful effect of tobacco on the periodontal condition of a Moroccan population. METHODS It was an exposed-non exposed study of 42 patients: 21 smokers and

[Impact of tobacco use on the periodontium--an update. Part 2: Clinical and radiographic changes in the periodontium and effects on periodontal and implant therapy].

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This third part of a series of publications from the Swiss task force "Smoking--Intervention in the private dental office" on the topic "tobacco use and dental medicine" describes the clinical and radiographic changes of the periodontium within smokers as well as the consequences of tobacco use on

Effect of nicotine on orthodontic tooth movement in rats.

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BACKGROUND Although the detrimental health effects of cigarettes are well known, many people, including some orthodontic patients, continue to smoke. Nicotine can affect bone resorption and apposition. Therefore, the aim of this study was to investigate the effect of nicotine on orthodontic tooth

Effects of nicotine administration and nicotine cessation on bone histomorphometry and bone biomarkers in Sprague-Dawley male rats.

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Nicotine is a major alkaloid of tobacco, which can increase free radical formation, leading to osteoporosis. The effects of nicotine administration and cessation on bone histomorphometry and biomarkers were studied in 28 Sprague-Dawley male rats. Rats aged 3 months and weighing 250-300 g were

Effects of nicotine on bone during orthodontic tooth movement in male rats. Histological and immunohistochemical study.

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OBJECTIVE To investigate the effects of nicotine on orthodontic tooth movement and accompanying histological and immunohistochemical changes in rats. METHODS An experimental study conducted at King Abdulaziz University, Jeddah, Saudi Arabia between 2013 and 2014. Thirty-two rats randomly divided

Effect of Environmental Tobacco Smoke on COX-2 and SHP-2 Expression in a Periodontitis Rat Model

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Objectives: To investigate the effects of environmental tobacco smoke (ETS) on the inflammatory process of periodontitis by evaluating bone loss and the expression of cyclooxygenase-2 (COX-2) and Src homology phosphotyrosyl phosphatase 2

Lipopolysaccharide enhances the production of nicotine-induced prostaglandin E2 by an increase in cyclooxygenase-2 expression in osteoblasts.

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Previous studies have indicated that lipopolysaccharide (LPS) from Gram-negative bacteria in plaque induces the release of prostaglandin E(2) (PGE(2)), which promotes alveolar bone resorption in periodontitis, and that tobacco smoking might be an important risk factor for the development and

Harmful lifestyles on orthopedic implantation surgery: a descriptive review on alcohol and tobacco use.

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Alcohol abuse and smoking habits have adverse effects on bone health and are a risk factor for osteoporosis, fractures and impaired fracture repair. Osteointegration processes around implanted biomaterials involve a coordinated cascade of complex events that are very similar to those occurring

Influence of nicotine on orthodontic tooth movement: A systematic review of experimental studies in rats.

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OBJECTIVE The objective of this systematic review was to assess the impact of nicotine administration on orthodontic tooth movement (OTM). METHODS A systematic search was conducted in PubMed, Scopus, EMBASE, MEDLINE (OVID) and Web of Knowledge databases and the Preferred Reporting Items for

Nicotine favors osteoclastogenesis in human periodontal ligament cells co-cultured with CD4(+) T cells by upregulating IL-1β.

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Periodontitis, which is the main cause of tooth loss, is one of the most common chronic oral diseases in adults. Tooth loss is mainly a result of alveolar bone resorption, which reflects an increased osteoclast formation and activation. Osteoclast formation in periodontal tissue is a multistep

Increased bone resorption in moderate smokers with low body weight: the Minos study.

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Tobacco was found to be a risk factor for osteoporosis, mainly in postmenopausal women. We studied the effect of smoking on bone mineral density (BMD) and bone turnover in a cohort of 719 men, aged 51-85 yr, composed of 83 current smokers, 405 former smokers, and 231 men who never smoked. Most

The bone density of female twins discordant for tobacco use.

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BACKGROUND Smoking is recognized as a risk factor for vertebral, forearm, and hip fractures. Since bone density is an important determinant of bone strength, we conducted a study to ascertain whether a deficit in bone density is associated with tobacco use and, if so, to identify the responsible

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