calcinosis/بوتاسيوم

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مقالاتالتجارب السريريةبراءات الاختراع

اختر نوع الفرز

14 النتائج

Ultrastructural investigation of microcalcification and the role of oxygen-glucose deprivation in cultured rat hippocampal slices.

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Intracellular calcium accumulation is associated with cell death in several neuropathological disorders including brain ischemia, but the exact mechanisms of calcification need to be clarified. We used organotypic hippocampal slice culture - cultures subjected to oxygen-glucose deprivation (OGD)

Early manifestation of nephropathy in rats with arterial calcinosis.

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In vascular smooth muscle, calcium overload is a highly pathogenic event, which increases with advancing age. An increase in the calcium content of arterial wall may be produced in rats by treatment with vitamin D3. The aim of this study was to evaluate the renal clearance of sulfanilamide (a model

Severe vascular disturbance in a case of familial brain calcinosis.

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Here we present the first neuropathological study of a case of autosomal dominant brain calcinosis in a family followed through five generations. The 71-year-old female who came to autopsy had unusually severe and extensive bilateral brain calcifications. The process appeared to start with

Enzootic calcinosis in sheep: soil-plant-animal relationship.

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Chemical compositions of soil, water, feeds, and forages on a farm where sheep were affected with calcinosis (experimental site) and a farm with sheep not affected with calcinosis (control site) were determined. The study lasted 1 year. It was observed that water from the experimental site had

Spatiotemporal Progression of Microcalcification in the Hippocampal CA1 Region following Transient Forebrain Ischemia in Rats: An Ultrastructural Study.

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Calcification in areas of neuronal degeneration is a common finding in several neuropathological disorders including ischemic insults. Here, we performed a detailed examination of the onset and spatiotemporal profile of calcification in the CA1 region of the hippocampus, where neuronal death has

[Calcinosis of the meniscus. Morphologic and roentgenographic findings for zonal classification].

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Primary and secondary meniscal chondrocalcinoses lead to typical changes in X-ray pictures of isolated menisci. Combined X-ray, light and electron microscopic examination of the four menisci in 70 autopsy cases showed calcifications in 13 cases (18.6%). Three types of calcification could be

Ultrastructural study of the long-term development of two experimental cutaneous calcinoses (topical calciphylaxis and topical calcergy) in the rat.

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Skin calcification induced by topical calciphylaxis was provoked by a subcutaneous injection of iron chloride in rats previously sensitized by dihydrotachysterol. A cutaneous topical calcergy was induced by an injection of potassium permanganate. An electron-microscopical study of the long-term

[Etiology, therapy and prophylaxis of bovine parturient paresis (hypocalcaemia)]

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Due to the sudden increase of calcium demand at the onset of lactation many high yielding dairy cows experience a certain level of hypocalcaemia following parturition. The incidence of hypocalcaemia (parturient paresis) increases with age but also depends on many other factors such as the acid-base

Speculations on hormonal controls of magnesium homeostasis: a hypothesis.

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Magnesium deficiency (MD) may be either latent: i.e. without disturbances in blood Mg, calcium and intracellular potassium (Ki) or patent, with a rich symptomatology: i.e. with hypomagnesemia, hypocalcemia and decreased Ki. These two aspects are also found with magnesium excess. The discrepancy

Bullous skin reaction seen after extravasation of calcium gluconate.

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Intravenous (IV) calcium is usually given to temporarily treat the effects of hyperkalaemia on muscle and heart. When extravasation of a calcium gluconate infusion occurs, there may be rapid and marked swelling and erythema, with signs of soft-tissue necrosis or infection, and ensuing extensive

Renal Tubular Acidosis.

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Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which

[Familial case of Fahr's disease].

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The article describes two cases of a rare disorder of potassium metabolism (intracerebral calcinosis, or Fahr's disease) in a 39-year-old male and his 60-year-old mother. It is noteworthy that the diagnosis of Fahr's disease was made during life on the basis of the clinical picture and CT findings.

Non-specific induction of mineralization of incisor pulp in the rat.

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Mineralized intrapulpal lesions were readily formed in the rat incisor after a single intrapulpal injection of the calcergen potassium permanganate, as well as after saline. Besides that there occurred irregular secondary dentin apposition and hard tissue overbridging at the injection site. The

Phytate (Myo-inositol hexakisphosphate) inhibits cardiovascular calcifications in rats.

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Calcification is an undesirable disorder, which frequently occurs in the heart vessels. In general, the formation of calcific vascular lesions involves complex physicochemical and molecular events. Calcification (hydroxyapatite) is initiated by injury and is progressed by promoter factors and/or the

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