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[In-vitro-effects of cocaine in skeletal muscle specimens of patients susceptible to malignant hyperthermia].

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OBJECTIVE The abuse of cocaine can cause serious medical complications like tachycardia, rhabdomyolysis, and hyperthermia. Because of the clinical similarities, it has been suggested that cocaine might be a trigger of malignant hyperthermia (MH). Therefore, aim of this study was to investigate the

Hyperthermia in sauna is unable to increase the plasma levels of ACTH/cortisol, beta-endorphin and prolactin in cocaine addicts.

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In order to establish possible different reactions between normal subjects and cocaine addicts to short term exposure to heat, thermal, cardiovascular and pituitary hormonal responses to hyperthermia in sauna were measured in 8 male cocaine addicts (studied after 14 days of abstinence) and in 8 age

Rhabdomyolysis and hyperthermia after cocaine abuse: a variant of the neuroleptic malignant syndrome?

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Rhabomyolysis with myoglobinuria has been added relatively recently to the neurologic complications associated with the increased use of cocaine and the introduction of its alkaloid form (crack). This retrospective study reports our experience with 14 patients who presented with rhabdomyolysis after

Methanandamide attenuates cocaine-induced hyperthermia in rats by a cannabinoid CB1-dopamine D2 receptor mechanism.

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Evidence implicates anandamide in dopamine-related cocaine function. In the present study, we investigated the effect of methanandamide (5 mg/kg, i.p.), a stable anandamide analog, on the hyperthermia and hyperactivity induced by a fixed dose of cocaine (15 mg/kg,i.p.). Cocaine administered to rats

Risperidone and 5-HT2A receptor antagonists attenuate and reverse cocaine-induced hyperthermia in rats

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Background: Cocaine (benzoylmethylecgonine) is one of the most widely used illegal psychostimulant drugs used worldwide, and mortality from acute intoxication is increasing. Suppressing hyperthermia is effective in reducing

Enhancement of cocaine-induced hyperthermia fails to elicit neurotoxicity.

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The neurotoxic potential of cocaine when administered under conditions conducive to the initiation of hyperthermia was investigated. Rats were administered cocaine at ambient temperatures of 22 degrees C or 30 degrees C. To determine the thermal response, body temperatures were measured every 30 min

Cocaine abuse with hyperthermia, seizures and fatal complications.

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The case of a previously-healthy woman who developed marked hyperthermia after the intravenous self-administration of cocaine is described. Status epilepticus and a prolonged period of hypotension were followed by the development of rhabdomyolysis, disseminated intravascular coagulation, acute renal

Hyperthermia, rhabdomyolysis, and myoglobinuric renal failure after recreational use of cocaine.

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A patient had hyperthermia, rhabdomyolysis, and myoglobinuric renal failure after the recreational use of cocaine. We attribute this to the pyrogenic properties of cocaine and environmental factors. The use of cocaine during summer months or in areas with high ambient temperatures should alert

Mechanism of cocaine-induced hyperthermia in humans.

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BACKGROUND The lethal effects of cocaine are unique among those of other illicit drugs because cocaine has the propensity to cause hyperthermia. The traditional view is that cocaine causes a hypermetabolic state with increased heat production. However, because cocaine-induced hyperthermia occurs

Effect of cocaine on the contracture response to 1% halothane in patients undergoing diagnostic muscle biopsy for malignant hyperthermia.

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Two case reports have cited the recreational use of cocaine as possible trigger of a malignant hyperthermia (MH) crisis. We evaluated whether toxic concentrations of cocaine altered the in vitro muscle response to halothane during contracture tests for MH. Twenty-two patients were studied. Muscle

Fatal malignant hyperthermia associated with recreational cocaine and ethanol abuse.

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Malignant hyperthermia is an often lethal hypermetabolic crisis state precipitated by a variety of pharmacological and environmental triggers in genetically susceptible persons. The present report documents, by medical history and necropsy, a fatal malignant hyperthermic crisis in a 20-year-old man

Cocaine-induced agitated delirium with associated hyperthermia: a case report.

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BACKGROUND "Cocaine-induced agitated delirium with associated hyperthermia" is a rare, almost uniformly fatal syndrome. The incidence of the disease is not known, however, it is believed to have markedly increased since the late 1980s with widespread popularity of crack cocaine. OBJECTIVE Recent

Hyperthermia sensitizes rats to cocaine's proconvulsive effects and unmasks EEG evidence of kindling after chronic cocaine.

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In phase I, 64 male and female Sprague-Dawley rat siblings from 8 litters were divided equally among 4 treatment groups; saline plus normothermia (S37), saline plus hyperthermia (S45), cocaine (30 mg/kg) plus normothermia (C37), and cocaine plus hyperthermia (C45) and treated daily from 45-60 days

Pharmacological and behavioral determinants of cocaine, methamphetamine, 3,4-methylenedioxymethamphetamine, and para-methoxyamphetamine-induced hyperthermia.

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BACKGROUND Cocaine, methamphetamine, 3,4-methylenedioxymethamphetamine (MDMA, ecstasy), and para-methoxyamphetamine (PMA) disrupt normal thermoregulation in humans, with PMA being associated with more severe cases of hyperthermia. Harm minimization advice on how to prevent overheating depends on

[Rhabdomyolysis syndrome and hyperthermia secondary to cocaine and/or ecstasy consumption. Report of 2 new cases and review of the problem].

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We describe two cases of hyperthermia and rhabdomyolysis related to cocaine and/or ecstasy consumption. The clinical and laboratory findings are characteristic of hyperthermia syndrome in both cases. The first patient consumed cocaine, ecstasy and alcohol and afterwards suffers from hyperthermia and

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