cocaine/نزف

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Refractile foreign material deposits and alveolar hemorrhage in crack cocaine smoker.

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Recreational use of alkaloid free-base cocaine, also known as crack cocaine, has reached epidemic proportions in the United States. Inhalation of crack cocaine is known to cause a variety of pulmonary complications. Herein we present a case of diffuse alveolar hemorrhage (DAH) and particulate

Diffuse alveolar hemorrhage temporally related to cocaine smoking.

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Previous reports of respiratory complications from cocaine abuse have focused on pulmonary barotrauma or a reduction in carbon monoxide diffusing capacity. We report a patient who developed life-threatening alveolar hemorrhage following repeated inhalation of alkaloid cocaine.

Increased incidence of intraventricular hemorrhage and developmental delay in cocaine-exposed, very low birth weight infants.

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This study sought to determine whether very low birth weight (VLBW) infants (< 1500 gm) with fetal cocaine exposure differed from non-cocaine-exposed VLBW infants in incidence of neonatal medical complications and in later developmental outcome. Forty-one cocaine-exposed, VLBW infants, followed in a

Cocaine-induced intracerebral hemorrhage in a patient with cerebral amyloid angiopathy.

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Intracerebral hemorrhage (ICH) is a well-recognized complication of recreational cocaine use. The precise mechanism of the cocaine-induced hemorrhagic event is unclear, although multiple factors have been implicated. We report a case of a 62-year-old woman who suffered left parieto-occipital ICH

Intralabyrinthine haemorrhage following cocaine consumption.

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The origin of cochleovestibular deficits remains hypothetical, a viral attack or a circulatory disorder being the two most frequently reported hypotheses. We report the case history of a patient suffering from a cochleovestibular deficit after consumption of cocaine, a drug known for its vascular

[Intraparenchymal cerebral hemorrhage and cocaine].

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Medical problems related to cocaine consumption are not rare in our environment; however, to this respect, descriptions in the spanish literature are scarce. We present three cases of intracerebral hemorrhage which occurred after cocaine consumption (intranasal route in two cases and intravenous

Alveolar hemorrhage associated with cocaine consumption.

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Cocaine is an illegal drug; its abuse and toxicity are a public health problem due to its high morbidity and mortality. Cocaine can affect the cardiovascular, central nervous and respiratory systems. The case of a 42-year-old male without history of chronic or degenerative diseases, but previous

Multiple intracerebral hemorrhages after smoking "crack" cocaine.

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After smoking "crack" cocaine and consuming large quantities of ethanol, a 36-year-old man developed multiple, bilateral, deep, and superficial cerebral hematomas. He was hypertensive for several days, but angiography revealed no evidence of vascular malformation or vasculitis. The multifocality of

Retinal hemorrhages associated with in utero exposure to cocaine. Experimental and clinical findings.

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BACKGROUND The use of drugs of abuse--e.g., cocaine--during pregnancy has been associated with abnormalities of the visual system. The authors studied the effects of prenatal exposure to drugs of abuse, especially cocaine, on the vascular system of the retina in newborn infants and in an

Spontaneous perinephric haemorrhage and acute renal failure in pregnancy due to cocaine intoxication.

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Cocaine abuse may contribute to the diverse forms of renal injury. We report a case of a pregnant woman who developed a large subcapsular renal haematoma after cocaine intoxication at 18-week gestation. She stabilized on conservative management and presented again at 29-week gestation with

Cocaine-induced intratumoral hemorrhage: case report and review of the literature.

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A unique case is described of a 28-year-old man who had seizures and a hemorrhage within a previously unsuspected cerebellopontine angle schwannoma within minutes of nasally inhaling cocaine. A review of the literature on cocaine-induced seizures and cocaine-induced cerebral hemorrhage is also

Absence of acute cerebral vasoconstriction after cocaine-associated subarachnoid hemorrhage.

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BACKGROUND Cocaine use has been associated with neurovascular complications, including arterial vasoconstriction and vasculitis. However, there are few studies of angiographic effects of cocaine on human cerebral arteries. Information on these effects could be obtained from angiograms of patients

[Stroke and intracranial hemorrhage after cocaine abuse].

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Cocaine abuse as a risk factor for acute cerebrovascular events has received too little attention, in particular in young patients. Cocaine hydrochloride causes mainly intracerebral and subarachnoidal bleeding, while crack (freebase) causes intracranial hemorrhage and ischemic infarctions with equal

Intravenous cocaine abuse and subarachnoid haemorrhage: effect on outcome.

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A retrospective study of subarachnoid hemorrhage associated with intravenous cocaine injection was undertaken in a large urban hospital. Patients who used intravenous cocaine had significantly poorer outcomes when compared with subarachnoid haemorrhage patients with no known exposure to the drug.

[Cocaine abuse as precipitating factor of tumoral bleeding in an oligoastrocitoma].

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Cocaine abuse has been associated with a variety of intracranial haemorrhagic disorders, such as intracerebral, subdural or subarachnoidal haemorrhage. Frequently, these patients harbour underlying vascular malformations, like cerebral aneurysms or arteriovenous malformations (AVM). To the best of

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