glioma/حمى

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Clinical Outcomes of Wulingsan Subtraction Decoction Treatment of Postoperative Brain Edema and Fever as a Complication of Glioma Neurosurgery.

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Objective. To evaluate the efficacy of Wulingsan subtraction ( WLSS) decoction in the treatment of postoperative brain edema and fever as a complication of glioma neurosurgery. Methods. This retrospective study was conducted at the Department of Neurosurgery of Liaocheng People's Hospital. Patients

[The combined effect of hyperthermia and irradiation on cultured human glioma cell line].

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The effects of hyperthermia (HT) and irradiation (RT) on a cultured human glioma cell line (KC) was examined by a colony formation assay and immuno-histochemical methods using monoclonal antibody (MoAb) against bromodeoxyuridine (BrdU) and MoAb Ki-67. The colony formation assay revealed that HT

Accumulation of cell cycle regulatory proteins, p21 and p27, induced after hyperthermia in human glioma cells.

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It was investigated whether there was a relationship between p53 p21 and p27 induction pathways in the cellular response of glioma cells to hyperthermia. Two glioma cell lines were employed. A-172 cells had the wild-type of p53, and U251 cells had the mutant-type of p53. An adenovirus harbouring

Sensitization to hyperthermia by intracellular acidification of C6 glioma cells.

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Hyperthermia has been introduced as a new modality of treatment for glioma. In these experiments, the cytotoxicity of hyperthermia in C6 glioma cells was enhanced by increasing the intracellular acidity with amiloride and/or 4,4'-diisothiocyanatostilbene-2,2' disulfonic acid (DIDS). Intracellular pH

Induction of differentiation by radiation and hyperthermia in neuroblastoma-glioma hybrid cells.

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The effects of either radiation or hyperthermia on the differentiation potential of NG108-15, a neuroblastoma-glioma hybrid cell line, were studied. After radiation and hyperthermia, the outgrowth of neurites from NG108-15 cells was potentiated, and polarizing current and voltage pulses induced a

The Design of Glioma Hyperthermia Instrument Based on Fuzzy-PID.

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Fuzzy-PID control is introduced to glioma Hyperthermia, the system structure chart of Hyperthermia is given, Fuzzy-PID arithmetic, control rules and the time response figure are illuminated in detail. The simulation figure of the heating field characteristic and circuit of temperature measure are

Hyperthermia enhanced chemosensitivity of human malignant glioma cells.

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In an effort to overcome chemoresistance of human malignant glioma cells, the modulation of drug-induced cell death by hyperthermia was assessed in 4 human malignant glioma cells lines, LN-18, LN-229, T98G and U87MG. Compared to normothermic conditions, pulsed 24 h drug exposure enhanced the

Applications of magnetoliposomes with encapsulated doxorubicin for integrated chemotherapy and hyperthermia of rat C6 glioma.

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There is substantial evidence regarding enhanced antitumor cytotoxicity of selected chemotherapeutic agents by appropriate heat exposure (40-44°C). Based upon these results, the integration of hyperthermia as an additional treatment modality given simultaneously with systemic chemotherapy is

Enhancement of ACNU treatment of the BT4An rat glioma by local brain hyperthermia and intra-arterial drug administration.

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To evaluate the role of intra-arterial (i.a.) chemotherapy, intravenous (i.v.) chemotherapy, and local brain hyperthermia in the treatment of gliomas, the effect of i.v. versus i.a. drug delivery, with or without local brain hyperthermia, was evaluated in BD IX rats with BT4An gliomas implanted in

[The use of an early postoperative interstitial-hyperthermia combination therapy in malignant gliomas].

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BACKGROUND The survival of people suffering from malignant gliomas (WHO level III and IV) is predominantly limited by local progress in the primary tumor region. Interstitial hyperthermia combined with radiotherapy or chemotherapy is one approach for the intensification of local therapy. It is

In vitro response of human glioblastoma and canine glioma cells to hyperthermia, radiation, and chemotherapy.

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Little is known about the sensitivity of human glioblastoma cells to hyperthermia alone and in combination with other therapies. We carried out in vitro cell survival studies on the human glioblastoma cell line U-87MG and our model canine glioma canine brain tumor (CBT) cells after multimodality

Adenovirus-mediated transfer of p53 augments hyperthermia-induced apoptosis in U251 glioma cells.

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OBJECTIVE Hyperthermia kills glioma cells by inducing apoptosis and is thereby an effective therapeutic modality for the treatment of malignant gliomas. However, cells harboring mutated p53 are refractory to hyperthermia-induced apoptosis. In this study, we assessed whether or not adenovirus

Proliferative potential and apoptosis in rat glioma cell lines after hyperthermia.

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The proliferative potential of cultured rat glioma cells (C6 and 9L) was evaluated after hyperthermia using immunohistochemical staining with bromodeoxyuridine (BrdU) and Ki-67 monoclonal antibodies. Apoptosis was assessed by in situ end-labeling of deoxyribonucleic acid breaks. Both BrdU and Ki-67

Concurrent hyperthermia and re-irradiation for recurrent high-grade gliomas.

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Salvage therapy for recurrent high grade gliomas (HGG) includes surgery, radiotherapy and chemotherapy, however, standard treatment does not exist. We evaluated the tolerability and efficacy of re-irradiation (re-RT) with hyperthermia (HT) for patients with recurrent HGG. From September 2010 to July

Enhanced delivery of a monoclonal antibody F(ab')2 fragment to subcutaneous human glioma xenografts using local hyperthermia.

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The purpose of this study was to investigate the effects of tumor-localized hyperthermia at 42 degrees C on the tissue distribution of radioiodinated monoclonal antibody F(ab')2 fragments. Paired-label biodistribution measurements were performed in athymic mice bearing D-54 MG human glioma

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