hyperammonemia/نوبة

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Hyperammonemia as a Marker of Subclinical Seizures After Traumatic Cardiac Arrest.

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This report details the presence of hyperammonemia in a patient who sustained cardiac arrest after a traumatic amputation. Serum ammonia levels may rise due to numerous etiologies; however, few reports detail its usefulness in diagnosing subclinical seizures. In this case, we successfully utilized

Transient hyperammonemia associated with postictal state in generalized convulsion.

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Previous studies revealed that transient hyperammonemia was noted after generalized convulsion. This study was undertaken to analyze the association between postictal state and serum ammonia levels. Adult patients presenting to the emergency department with seizures were included. Serum ammonia and

Hyperammonemia is associated with generalized convulsion.

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OBJECTIVE The possibility that hyperammonemia may be associated with generalized convulsion (GC) was retrospectively investigated. METHODS Subjects comprised 17 patients with GC who were transported to our department and underwent analysis of serum biochemistry, including ammonia, since October

Hyperammonemia in idiopathic epileptic seizure.

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OBJECTIVE It is known that patients with convulsion often present hyperammonemia. The elevation of ammonia levels in convulsion is considered to occur along with extensive muscle contractions, but the details remain unclear. In emergency pathologies, such as cardiopulmonary arrest or hemorrhagic

Hyperinsulinism-hyperammonemia Syndrome in an Infant with Seizures.

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Hyperinsulinism-hyperammonemia syndrome (HI/HA) is the second most common form of persistent hyperinsulinemic hypoglycemia of infancy (PHHI). The main clinical characteristics of HI/HA syndrome are repeated episodes of symptomatic hypoglycemia, but not usually severe. Consequently, children with

Seizures and diagnostic difficulties in hyperinsulinism-hyperammonemia syndrome.

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Hyperinsulinism/hyperammonemia (HI/HA) syndrome is a rare disorder presented with recurrent hypoglycemia and elevated serum ammonia, which may lead to development delays, permanent neurologic damages, if it remains underdiagnosed. It is caused by activating mutations in the GLUD1 gene which encodes

Inhibition of acute hyperammonemia-induced convulsions by systemically administered gamma aminobutyric acid in rats.

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The present study has investigated the effects of intraperitoneally administered gamma aminobutyric acid (GABA) on ammonium chloride-induced hyperammonemia and convulsions in rats. Systemically administered GABA did not alter the concentration of GABA in the brain of control as well as

Intractable absence seizures in hyperinsulinism-hyperammonemia syndrome.

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A girl with intractable absence seizures and facial myoclonia at age 7 years was eventually diagnosed with hyperinsulinism-hyperammonemia syndrome because of hypoglycemia, hyperinsulinism, hyperammonemia, and the results of an oral l-leucine loading test. Her seizures occurred even during periods of

Transient hyperammonemia in seizures: a prospective study.

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OBJECTIVE To investigate the incidence and duration of transient hyperammonemia in seizures and to verify the significant confounders related to transient hyperammonemia in seizures. METHODS One hundred twenty-one noncirrhotic adult patients with seizures admitted to the emergency department were

Postictal transient hyperammonemia as an indicator of seizure disorder.

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OBJECTIVE Transient hyperammonemia can occur after episodes of seizure. To verify that transient hyperammonemia is a unique finding in seizure patients, we compared the change in blood ammonia concentration during the postictal period with that of other patients who suffered consciousness

Familial hyperinsulinism-hyperammonemia syndrome in a family with seizures: case report.

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Hyperinsulinism-hyperammonemia (HI/HA) syndrome is the second most frequent cause of congenital hyperinsulinism (CHI) and it is characterized by recurrent symptomatic hypoglycemia and persistent hyperammonemia. We describe the familial case of a 2-year-old child and her 32-year-old mother who,

Risk Factors Associated With Hyperammonemia Following Unprovoked Convulsive Seizures

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This report describes a unique case of recurrent transient hyperammonemia (THA) following a first-time occurrence of generalized tonic-clonic seizure in a young adult, who went on to develop post-stroke epilepsy. Although this phenomenon has been described in recent literature, we report not only

Generalized convulsion resulted in hyperammonemia during treatment with tranexamic acid for hemoptysis.

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BACKGROUND Tranexamic acid is commonly used to treat various kinds of bleeding disorders. It has been shown to cause severe convulsions in animal experiments. OBJECTIVE We report a patient who experienced a single convulsive seizure that resulted in transient hyperammonemia during treatment with

Valproate-induced hyperammonemia and seizures: perioperative concerns.

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Hyperornithinemia, hyperammonemia, and homocitrullinuria. A new disorder of amino acid metabolism associated with myoclonic seizures and mental retardation.

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