liver cirrhosis/نيكوتين

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Liver cirrhosis, tobacco, alcohol, and cancer among blacks.

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Attributes of age, tobacco use, and alcohol consumption were studied in order to elucidate their roles in the increased risks of blacks for selected neoplasms. Black cancer patients with and without liver cirrhosis were compared by cancer sites, age, tobacco usage, and alcohol consumption.

[Geographical relations between mortality from cancer of the esophagus, cirrhosis of the liver, alcohol and tobacco: case of the Province of Quebec].

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This study concerns the geographical relationships between alcohol consumption per capita; smoking and death from cirrhosis of the liver and cancer of the oesophagus in persons of 15 years old and over in Quebec. First, the geographical distribution of variables is analysed. Secondly, we have used

Liver cirrhosis mortality, alcohol consumption and tobacco consumption over a 62 year period in a high alcohol consumption country: a trend analysis.

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BACKGROUND The relationship between alcohol consumption and liver cirrhosis mortality has been revealed by data from several different countries. However, the impact of tobacco smoking on liver cirrhosis has not been considered. The aim of this study was to estimate trends in liver cirrhosis

Alcohol, tobacco and age factors in the relative frequency of cancer among males with and without liver cirrhosis.

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Causes of death among workers in the tobacco industry.

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The number of deaths by cause, race, and sex among 2,709 workers (1,003 white males, 789 black males, 517 white females, and 400 black females) in the tobacco industry identified from obituary listings of the Tobacco Workers' International Union from 1957 to 1978 were compared to expected numbers

Nicotine metabolism in liver microsomes from rats with acute hepatitis or cirrhosis.

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Nicotine exerts a number of physiological effects. Nicotine is absorbed through the lungs with smoking and is rapidly metabolized in humans. Although it is mainly metabolized in the liver, the effects of liver injuries on nicotine metabolism are not clear. The purpose of this study was to clarify

Roles of alcohol and tobacco exposure in the development of hepatocellular carcinoma.

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The purpose of this report is to summarize the roles of alcohol and tobacco exposure in the development of hepatocellular carcinoma (HCC). Chronic heavy alcohol exposure is a major risk factor for HCC, which is the most frequent type of liver cancer. Alcohol ingestion may initiate and or promote the

Tobacco Smoking Is Not Associated With Accelerated Liver Disease in Human Immunodeficiency Virus-Hepatitis C Coinfection: A Longitudinal Cohort Analysis.

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Background. Tobacco smoking has been shown to be an independent risk factor for liver fibrosis in hepatitis C virus (HCV) infection in some cross-sectional studies. No longitudinal study has confirmed this relationship, and the effect of tobacco exposure on liver fibrosis in human immunodeficiency

Is tobacco a risk factor for chronic pancreatitis and alcoholic cirrhosis?

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In a case control study alcohol intake and tobacco use were assessed between 1975 and 1987 in 103 male patients suffering from alcoholic cirrhosis of the liver, in 145 patients with chronic pancreatitis, and in 264 control subjects. The patients with chronic pancreatitis were significantly younger

Nicotine induces fibrogenic changes in human liver via nicotinic acetylcholine receptors expressed on hepatic stellate cells.

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OBJECTIVE Cigarette smoke (CS) may cause liver fibrosis but possible involved mechanisms are unclear. Among the many chemicals in CS is nicotine - which affects cells through nicotinic acetylcholine receptors (nAChR). We studied the effects of nicotine, and involved pathways, on human primary

Detrimental effects of nicotine on thioacetamide-induced liver injury in mice.

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OBJECTIVE Nicotine exerts a number of physiological effects. The purpose of this study was to determine the effects of nicotine on thioacetamide (TAA)-induced liver fibrosis in mice. METHODS For in vivo experiments, hepatic fibrosis was induced by TAA (0.25 g/kg, i.p.) three times a week for 6

Protective effects of coffee against oxidative stress induced by the tobacco carcinogen benzo[α]pyrene.

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Coffee consumption has been epidemiologically associated with a lower risk for liver cirrhosis and cancer. UDP-glucuronosyltransferases (UGT1A) catalyze the detoxification of reactive metabolites thereby acting as indirect antioxidants. Aim of the study was to examine UGT1A regulation in response to

Blood lead level is associated with advanced liver fibrosis in patients with non-alcoholic fatty liver disease: A nationwide survey (NHANES 2011-2016).

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Non-Alcoholic Fatty Liver Disease (NAFLD) is linked to obesity and metabolic syndrome, but increasing evidence also implicates environmental toxins. In this study, we aim to show that in elevated blood Lead levels in NAFLD patients result in worsening liver

[Dyspnea in a 45-year-old man with liver cirrhosis].

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METHODS During evaluation for a liver transplantation in a 45-year-old man with alcoholic liver he complained of exertional dyspnea. He had grade 3 chronic obstructive pulmonary disease (COPD) after nicotine abuse of 50 pack-years. One and a half years earlier the patient had been treated for

[Cirrhosis of the liver as a precancerous condition].

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Cirrhosis of the liver can be regarded as premalignant state, since more than 80 percent of hepatocellular carcinoma (HCC) in the western world develop in a cirrhotic liver. The risk to develop this malignancy depends on the activity of the underlying cirrhosis, its etiology and the duration of the

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