neuromyelitis optica/نوبة

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Epileptic seizures in Japanese patients with multiple sclerosis and neuromyelitis optica.

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OBJECTIVE To examine the incidence and clinical features of epileptic seizures in Japanese patients with multiple sclerosis (MS) and neuromyelitis optica (NMO). METHODS We reviewed medical records of all patients who visited the Neurology Clinic in Utano National Hospital between January and

Acute epileptic seizures in myelin oligodendrocyte glycoprotein encephalomyelitis and neuromyelitis optica spectrum disorder: A comparative cohort study.

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BACKGROUND Little is known about the incidence and characteristics of acute epileptic seizures in myelin oligodendrocyte glycoprotein encephalomyelitis (MOG-EM) and neuromyelitis optica spectrum disorder (NMOSD). In this study, we compared the incidence and characteristics of acute epileptic

Bilateral extensive lesions of the brain in a patient with neuromyelitis optica manifested with seizure and cognitive impairments.

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A young woman with seizures, visual impairment, and paralysis.

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Anti-MOG encephalomyelitis is a recently described demyelinating, autoimmune disease of the central nervous system, identified by antibodies against myelin oligodendrocyte glycoprotein (MOG).A previously healthy 20-year-old woman was admitted to hospital

Seizures and multiple sclerosis in Chinese patients: a clinical and magnetic resonance imaging study.

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OBJECTIVE Epileptic seizures in Chinese patients with multiple sclerosis (MS) have not been studied extensively. We investigated the clinical, laboratory, and imaging findings for Chinese patients with MS who had experienced seizures. METHODS A total of 93 (57.4%) patients were diagnosed as having

A treatable cause of myelopathy and vision loss mimicking neuromyelitis optica spectrum disorder: late-onset biotinidase deficiency.

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Biotinidase deficiency is characterized by severe neurological manifestations as hypotonia, lethargy, ataxia, hearing loss, seizures and developmental retardation in its classical form. Late-onset biotinidase deficiency presents distinctly from the classical form such as limb weakness and vision

Secondary Hypogammaglobulinemia After Rituximab for Neuromyelitis Optica: A Case Report.

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A 17-year-old male with history of neuromyelitis optica and seizures presented to the pulmonology clinic for evaluation of recurrent pneumonias. He had received rituximab for the past 6 years. Over the past 2 years, he experienced four episodes of pneumonia. In between these episodes, he would

Devic's neuromyelitis optica during pregnancy in a patient with systemic lupus erythematosus.

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Neuropsychiatric forms of systemic lupus erythematosus (SLE) vary, most commonly consisting of seizures, psychiatric disturbances, or focal central nervous deficits. This is a new case of neuromyelitis optica or Devic's syndrome during the course of SLE. Few reports of this association exist in the

Biotinidase deficiency presenting as Neuromyelitis Optica Spectrum Disorder

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Biotinidase deficiency disorder is a rare inherited metabolic disorder with typical neurological manifestations of hypotonia, developmental delay, rashes, seizures, hearing and vision impairment. We present two cases with different and unusual clinical profiles, whose neuroimaging resembled

Current symptomatology in multiple sclerosis and neuromyelitis optica.

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OBJECTIVE Several symptoms and signs are characteristic of multiple sclerosis (MS) such as Lhermitte's sign, Uhthoff's phenomenon and painful tonic seizure. Neuromyelitis optica (NMO) is another inflammatory disease of the central nervous system, and most of the opticospinal form of MS is thought to

A Case of Neuromyelitis Optica Spectrum Disorder Complicated by Posterior Reversible Encephalopathy Syndrome as an Initial Manifestation.

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A 25-year-old woman was admitted to our hospital due to tonic convulsion with severe headache after having experienced symptoms of nausea and vomiting for a month. Brain MRI showed extensive symmetrical lesions in the cortical and subcortical areas of parieto-occipital lobes and basal ganglia,

Aquaporin-4: orthogonal array assembly, CNS functions, and role in neuromyelitis optica.

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Aquaporin-4 (AQP4) is a water-selective transporter expressed in astrocytes throughout the central nervous system, as well as in kidney, lung, stomach and skeletal muscle. The two AQP4 isoforms produced by alternative spicing, M1 and M23 AQP4, form heterotetramers that assemble in cell plasma

Anti-N-methyl-D-aspartate receptor(NMDAR) antibody encephalitis presents in atypical types and coexists with neuromyelitis optica spectrum disorder or neurosyphilis.

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BACKGROUND Anti-N-methyl-D-aspartate receptor (NMDAR) encephalitis is a clinically heterogeneous disorder characterized by epileptic seizures, psychosis, dyskinesia, consciousness impairments, and autonomic instability. Symptoms are always various. Sometimes it presents in milder or incomplete

Atypical presentation of MOG-related disease: Slowly progressive behavioral and personality changes following a seizure.

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Myelin Oligodendrocyte Glycoprotein (MOG) antibodies-related disease is mainly presented with acute disseminated encephalomyelitis (ADEM), recurrent optic neuritis, and neuromyelitis optica spectrum disorders (NMOSDs), however the complete clinical spectrum has not yet been defined. We

Clinical features of neuromyelitis optica in children: US Network of Pediatric MS Centers report.

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OBJECTIVE To compare clinical features of pediatric neuromyelitis optica (NMO) to other pediatric demyelinating diseases. METHODS Review of a prospective multicenter database on children with demyelinating diseases. Case summaries documenting clinical and laboratory features were reviewed by an

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