osteonecrosis/وذمة

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Histological findings of bone marrow edema pattern on MRI in osteonecrosis of the femoral head.

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Sequential magnetic resonance imaging (MRI) was performed on a 38-year-old woman with systemic lupus erythematosus who had received corticosteroid and had developed non-traumatic osteonecrosis of the femoral head. The initial MR finding was a band lesion on the T1-weighted image, which had been

Infusion, core decompression, or infusion following core decompression in the treatment of bone edema syndrome and early avascular osteonecrosis of the femoral head.

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An increase in interstitial fluid is an expression of bone marrow edema (BME) and osteonecrosis (ON). The exact pathogenetic processes still remain unknown. Treatment options are mainly symptomatic with core decompression as surgical golden standard with immediate pain relief. Recently, it has been

Osteonecrosis, transient osteoporosis, and transient bone marrow edema: current concepts.

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Osteonecrosis, transient osteoporosis, and transient bone marrow edema are closely related diseases that may have an overlapping clinical and radiographic presentation, thus creating difficulty in establishing a diagnosis. Close scrutiny of MR images may aid in distinguishing the pattern of

[Differences between transient osteoporosis of the hip and bone marrow edema associated with osteonecrosis of the femoral head].

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OBJECTIVE To explore the differences between transient osteoporosis of the hip (TOH) and bone marrow edema (BME) associated with osteonecrosis of the femoral head (ONFH) in terms of clinical practice and imaging. METHODS From January 2006 to February 2008, 5 hips of TOH in 5 cases (1 male and 4

[Study on correlation between pain grading, stage of necrosis and bone marrow-edema in nontraumatic osteonecrosis of femoral head].

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OBJECTIVE To explore the correlation between pain grading, stage of necrosis and bone marrow edema (BME) in nontraumatic osteonecrosis of femoral head (NONFH) so as to strengthen understandings about clinical significance of BME in NONFH. METHODS From October 2004 to October 2006, 97 patients (149

MR imaging of avascular necrosis and transient marrow edema of the femoral head.

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The value of high spatial resolution and contrast material-enhanced magnetic resonance (MR) imaging was assessed in 69 patients with either femoral head avascular necrosis (AVN) or transient bone marrow edema lesions. An AVN lesion was typically a well-demarcated epiphyseal area of variable signal

Acute bone pain following renal transplantation: differentiation between benign bone edema and avascular necrosis.

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Two patients are reported who presented within the first 3 months posttransplantation with acute bone pain where serial magnetic resonance imaging (MRI) allowed differentiation between bone edema, which resolved spontaneously, and avascular necrosis (AVN) requiring core decompression. Case 1 had

[Diagnosis and treatment of bone marrow edema syndrome of the hip and differential diagnosis from avascular necrosis of femoral head].

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OBJECTIVE To observe the diagnosis and treatment of bone marrow edema syndrome and summarize its features, mechanisms and its differences from avascular necrosis of femoral head. METHODS From 2004.1, 19 patients (12 patients were males and 7 patients were females, with a mean age of (46.70 +/-

Study on correlation between bone marrow edema, stage of necrosis and area ratio of necrosis with the hip pain grading in nontraumatic osteonecrosis of the femoral head.

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The objective of this study was to explore the correlation between bone marrow edema, stage of necrosis, and area ratio of necrosis with the hip pain grading in non-traumatic osteonecrosis of the femoral head. Bone marrow edema grading at all levels and the hip pain grade differences were

Avascular necrosis of bone following short-term dexamethasone therapy for brain edema. Case report.

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The authors present a case of avascular necrosis of both femoral and humeral heads which developed after short-term steroid treatment for brain edema. A vascular necrosis of bone may develop after short-term as well as after maintenance steroid therapy. Early diagnosis with bone scanning and

Coagulation abnormalities in osteonecrosis and bone marrow edema syndrome.

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The aim of this review was to provide information about the variety of thrombophilic and hypofibrinolytic markers that are possible risk factors for the development of osteonecrosis and bone marrow edema syndrome. A total of 48 parameters were identified in 45 studies that included 2163 patients.

Calcium dobesilate (Doxium®) in bone marrow edema syndrome and suspected osteonecrosis of the hip joint - A case series

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Osteonecrosis of the femoral head is a disease that leads to gradual degeneration of the hip joint causing immobility and a devastating impairment of quality of life. Early stage disease is characterised by a bone marrow edema of the proximal femur, a sight it shares with bone marrow edema syndrome.

[Craniocerebral trauma and aseptic osteonecrosis. Steroid-induced sequelae after therapy of brain edema].

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In this publication we present three cases of avascular osteonecrosis (AON) of the femoral head and the talus in adolescent and young adult patients following short-term, high-dose steroid therapy for cerebral trauma. All patients were proven to be free of other risk factors for AON. The latency

Magnetic resonance perfusion and diffusion imaging characteristics of transient bone marrow edema, avascular necrosis and subchondral insufficiency fractures of the proximal femur.

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OBJECTIVE To evaluate magnetic resonance (MR) perfusion and diffusion imaging characteristics in patients with transient bone marrow edema (TBME), avascular necrosis (AVN), or subchondral insufficiency fractures (SIF) of the proximal femur. METHODS 29 patients with painful hip and bone marrow edema

Transient bone marrow edema syndrome progressing to avascular necrosis of the hip - a case report and review of the literature.

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Transient bone marrow edema syndrome (TMES) is a rare disease of unknown etiology. Diagnosis is made by exclusion. There is still controversy as to whether TMES is considered to be a reversible form of avascular necrosis (AVN), a disease entity of its own or a form of non-traumatic algodystrophy. We

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