rhabdomyolysis/نوبة

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Case report: rhabdomyolysis following grand mal seizures presenting as a delayed and increasingly dense nephrogram.

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Rhabdomyolysis as a result of major trauma is a well recognized cause of acute renal failure. Non-traumatic rhabdomyolysis causing transient renal impairment may occur following generalized convulsions. We present a case in which rhabdomyolysis following epilepsy was first indicated at urography by

Doxylamine toxicity: seizure, rhabdomyolysis and false positive urine drug screen for methadone.

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The present report highlights the possible adverse effects of doxylamine, a common over the counter sleep aid. Doxylamine is an antihistamine that at toxic doses can cause anticholinergic effects, including seizures, rhabdomyolysis and death. The following case describes a patient with doxylamine

A patient using ziprasidone with polydipsia, seizure, hyponatremia and rhabdomyolysis.

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We aimed to report a case with rhabdomyolysis related to hyponatremia and/or its correction. A 32-year-old male schizophrenic patient on ziprasidone treatment was admitted to the hospital following a seizure. Patient had primary polydipsia and secondarily developed hyponatremia. After the correction

CT of rhabdomyolysis associated with malignant hyperthermia and seizures.

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Computed tomography evaluation in rhabdomyolysis can reveal muscle necrosis and calcification. We report the computed tomography findings in acute rhabdomyolysis, secondary to hyperthermia and seizures.

Isoniazid-induced seizures with secondary rhabdomyolysis and associated acute renal failure in a dog.

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Isoniazid-induced seizures resulted in rhabdomyolysis and associated acute renal tubular necrosis in a dog. Rhabdomyolysis and myoglobinuric renal failure, although recognised in the dog, are reported infrequently as a consequence of seizures. The clinical presentation of isoniazid toxicity in a dog

[Rhabdomyolysis related to statin and seizures: report of 3 cases].

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OBJECTIVE To investigate the clinical features and prognosis of rhabdomyolysis related to seizure attacks and use of statin. METHODS The medical records of 3 patients with established diagnosis of rhabdomyolysis were analyzed and the related literatures were reviewed. RESULTS All the 3 patients had

Fluoxetine overdose in a teenager resulting in serotonin syndrome, seizure and delayed onset rhabdomyolysis.

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A 14-year-old young adult took an overdose of 1.2 g of fluoxetine, a selective serotonin reuptake inhibitor (SSRI) that he had been prescribed for depression. He had a generalised tonic/clonic seizure at 6 hours postingestion.After the seizure, he developed signs consistent with serotonin syndrome:

Seizures, Systemic Inflammatory Response, and Rhabdomyolysis Associated With Laboratory-Confirmed 2C-I and 25-I Exposure.

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The 2C drugs are hallucinogenic phenethylamines. They and their n-benzyloxymethyl analogs have become popular as "legal highs," and significant toxicity has been attributed to their use. We report on a case of seizures, systemic inflammatory response, and rhabdomyolysis associated with

Severe, self-limiting lactic acidosis and rhabdomyolysis accompanying convulsions.

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A 26 year old man with no previous history of convulsions presented in status epilepticus and severe lactic acidosis. He regained consciousness and the acidosis resolved after several hours of conservative management without intravenous bicarbonate, but he developed severe myalgia associated with

[Acute renal failure due to rhabdomyolysis in McArdle's disease following binge drinking with seizures].

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METHODS A 36-year-old gardener was admitted for tonic-clonic seizures after binge drinking. The next days he developed massive rhabdomyolysis with acute renal failure. Past medical history was unremarkable except for a similar episode of acute renal failure 14 years ago. At that time he had consumed

Acute renal failure resulting from rhabdomyolysis following a seizure.

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A 30-year-old Indian man presented with progressive renal impairment following a seizure. The complaint of troublesome back pain led to the suspicion of rhabdomyolysis being the cause of acute renal failure. The diagnosis of rhabdomyolysis was supported by a markedly elevated serum creatinine

General convulsions and rhabdomyolysis. Case reports.

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Two patients who developed rhabdomyolysis secondary to generalized seizures are presented. Minor traumas of skeletal muscles due to seizures may be associated with myoglobinuria and the development of acute renal failure. The diagnosis rhabdomyolysis may easily be confirmed by following serum

Deep vein thrombosis complicating severe hypernatremia, rhabdomyolysis, and acute renal failure in a patient with untreated seizure disorder.

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We report a 22-year-old male patient with untreated seizure disorder, presenting with increased frequency of seizures followed by encephalopathy. Laboratory evaluation showed severe hypernatremia (175 meq/l sodium), rhabdomyolysis, and acute renal failure (ARF). Excessive insensible water loss in

Seizures, ventricular tachycardia, and rhabdomyolysis as a result of ingestion of venlafaxine and lamotrigine.

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Few cases of overdoses have been described involving venlafaxine, lamotrigine, or a combination of the two agents. We describe a combined venlafaxine and lamotrigine ingestion in a patient presenting with a seizure, ventricular tachycardia, and rhabdomyolysis. We conclude that patients with

Acute renal failure due to rhabdomyolysis following a seizure.

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Acute renal failure, oliguric or nonoliguric, is the most common complication of rhabdomyolysis. Rhabdomyolysis should be suspected in patients presenting with states of increased muscular activity, such as seizures, agitation, strenuous muscle exercise, or dystonia. We report an adult who developed

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