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Anticonvulsant effects of thiamine on pentylenetetrazole-induced seizure in mice.

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OBJECTIVE Thiamine serves as a cofactor for several enzymes involved in brain function and neurotransmitters biosynthesis. Thiamine-dependent enzymes are important for oxidant stress defenses. Several studies have reported that thiamine deficiency in the central nervous system reduces seizure

Effect of thiamine tetrahydrofurfuryl disulfide on audiogenic seizures in DBA/2J mice.

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Weanling mice of the DBA/2J strain are spontaneously audiogenically seizure prone between 19 and 24 days. Thereafter, susceptibility declines rapidly within the next 7-10 days. It was found that thiamine tetrahydrofurfuryl disulfide (TTFD) significantly delayed the natural disappearance of seizure

Lead induced thiamine deficiency in the brain decreased the threshold of electroshock seizure in rat.

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Many neurological disorders that occur frequently in lead intoxicated animals, have also been observed in thiamine deficient animals. To test whether lead intoxication could decrease the thiamine status and thresholds of electroshock seizure in rats, 3-week-old Wistar rats were treated with lead or

Brain ammonia during convulsions in thiamine-deficient mice.

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[ACTIVITY OF TISSUE TRANSAMINASES IN CONVULSIONS PRODUCED WITH THIAMINE].

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AN INVESTIGATION ON THE THIAMINE CONTENT OF MOTHER'S MILK IN RELATION TO INFANTILE CONVULSIONS.

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Experimentally induced thiamine deficiency in beagle dogs: clinical observations.

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Twenty-three 2- to 5-month-old Beagle dogs were fed a purified thiamine-deficient ration (2 to 3 micrograms of thiamine/100 g of ration) at a rate of 40 to 70 g/kg of body weight/day depending on age. Eleven dogs were used as principles, 6 as pair-fed controls, and 6 as ad libitum-fed controls.

Persistence of habituation deficits after neurological recovery from severe thiamine deprivation.

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Rats were fed a thiamine-deficient diet for 4 weeks and injected daily with pyrithiamine during the last two weeks of the diet. This regime induced severe neurological anomalies such as ataxia, loss of righting reflex and visual place reflex, and finally full tonic-clonic seizures. These symptoms

Effect of a deficiency of thiamine on brain pyruvate dehydrogenase: enzyme assay by three different methods.

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A simple and rapid method based on the NADH-linked reduction of a tetrazolium dye was described for the determination of pyruvate dehydrogenase activity in rat brain homogenates. The method (method 3) gave a value of 36.06 +/- 1.24 nmol of pyruvate utilised/min/mg of whole brain protein. This value

Thalamic Proteome Changes and Behavioral Impairments in Thiamine-deficient Rats.

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Thiamine deficiency (TD) has been used as an experimental model in rodents to study the molecular mechanisms of neurodegeneration and its association with behavioral changes. The aims of the present study were to investigate the spatial cognitive performance of pyrithiamine-induced thiamine

[Effect of pyrithiamine on rat sciatic nerve. (II) Morphological changes during the last stages of thiamine deficiency (author's transl)].

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We observed under light and electron microscopes morphological changes in the rat sciatic nerve during the last stages of a thiamine deficient state as induced by pyrithiamine (PT: 50 microgram/100g X 11 days) and thiamine deficient diet (TDD), in which rats experienced severe tetanic convulsions.

Biotin-Thiamine-Responsive Basal Ganglia Disease

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Clinical characteristics: Biotin-thiamine-responsive basal ganglia disease (BTBGD) may present in childhood, early infancy, or adulthood. Prompt administration of biotin and thiamine early in the disease course results in partial

Thiamine deficiency in a dog associated with exclusive consumption of boiled sweet potato (Ipomoea batatas): Serial changes in clinical findings, magnetic resonance imaging findings and blood lactate and thiamine concentrations

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Thiamine (vitamin B₁ ) is an essential nutrient that significantly influences ATP production in the body. It needs to be supplemented consistently through an exogenous source to prevent deficiency; however, it is easily affected by a variety of mitigating factors. Additionally, thiamine

Thiamine deficiency in a cat: resolution of MRI abnormalities following thiamine supplementation.

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Thiamine (vitamin B(1)) is an essential component of a number of metabolic pathways and thiamine deficiency results in a progressive encephalopathy in both humans and animals. Confirming thiamine deficiency is problematic and relies on demonstrating reduced red blood cells transketolase activity, or

[Subacute encephalopathy with epileptic seizures in a patient with chronic alcoholism (SESA syndrome)].

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Subacute encephalopathy with seizures in alcoholics (SESA syndrome) is a rare disease entity following chronic alcohol ingestion. It is quite distinct from alcohol withdrawal syndromes, such as delirium, withdrawal seizures or CNS complications of alcohol, such as Wernicke-Korsakow syndrome, central

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