uremia/ألبيومين

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Underestimation of serum albumin by the bromcresol purple method and a major endogenous ligand in uremia.

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In hemodialyzed patients, the serum albumin concentration determined by the bromcresol purple (BCP) method was lower than that determined by an immunological method. The degree of underestimation appeared to be well correlated to the serum concentration of

Alteration of plasma albumin in relation to decreased drug binding in uremia.

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The binding of sulfadiazine in the plasma of normal and uremic subjects was measured by means of an ultrafiltration technique. Patients with uremia had reduced binding of sulfadiazine due to decreased albumin-binding capacity. No conclusive evidence was found to suggest that reduced drug binding in

Biophysical insight into furosemide binding to human serum albumin: a study to unveil its impaired albumin binding in uremia.

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Exogenous substances like drugs, when absorbed, enter into the circulatory system and bind reversibly and extensively to human serum albumin (HSA). But transport of various drugs like a diuretic, furosemide (FUR), via albumin in uremia is seriously compromised due to accumulation of uremic toxins.

Serum fructosamine in uraemia, myeloma and acute inflammatory disorders--relationship to serum glucose and albumin levels.

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There has been considerable interest in the serum fructosamine assay as a measure of glycated serum proteins. We have measured serum fructosamine in three groups of patients--those with uraemia; those with multiple myeloma; and those with acute inflammatory conditions--none of whom were known to

Effects of long-term low protein diet on albumin metabolism in chronic uremia.

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The effects of long-term low protein diet on albumin metabolism of uremic patients were evaluated. Studies were performed on 62 patients divided into two groups depending on the duration of the diet (35 subjects from 6 to 30 days, 27 subjects from 6 months to 5 years). All patients received a diet

Mass spectrometric monitoring of albumin in uremia.

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BACKGROUND Advanced glycation end products (AGEs) are a novel class of uremic toxins. In plasma, they are present in proteins and also in low molecular mass peptides. AGE-modified peptides are thought to bind and modify plasma proteins. Monitoring of the consequent increase in molecular mass of

Albumin is the major plasma protein target of oxidant stress in uremia.

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BACKGROUND Patients with uremia are exposed to increased oxidative stress. Examination of the oxidation of individual plasma proteins may be useful in establishing specific pathways of oxidative stress in vivo and in determining functional consequences of oxidant stress exposure. We therefore

The incorporation of ammonia nitrogen into albumin in man: the effects of diet, uremia and growth hormone.

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15 N-ammonium chloride was given orally to 17 individuals in 20 studies. Retention of 15N by those with normal renal function was inversely proportional to the protein intake; enrichment of albumin with 15N increased during protein restriction. Protein restriction appeared to be a more potent

Underestimation of albumin content by bromocresol green, induced by drug displacers and uremia.

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Phenylbutazone and clofibric acid, two drugs strongly bound to human albumin, produce low readings of albumin content in serum when the bromocresol green immediate reaction is used. This abnormality is observed at drug concentrations within the range obtained during therapeutic use, and tends to be

A biophysical insight into structural and functional state of human serum albumin in uremia mimic milieu.

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In chronic kidney diseases (CKD), uremic toxins accumulate in the blood plasma of patients and interact with human serum albumin (HSA) and thus impaired its role as carrier protein. Present study shows in vitro effect of carboxy-4-methyl-5-propyl-2-furanpropanoic acid (CMPF), indoxyl sulfate (IS),

Albumin binding in uraemia: quantitative assessment of inhibition by endogenous ligands and carbamylation of albumin.

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The binding capacity of human serum albumin (HSA) for small acidic molecules is known to be reduced in chronic renal failure (CRF). The contribution of competitive inhibition by accumulated endogenous ligands and of structural changes in HSA has now been evaluated. In a fluorimetric in vitro assay

Interaction of 3-carboxy-4-methyl-5-propyl-2-furanpropanoic acid, an inhibitor of plasma protein binding in uraemia, with human albumin.

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The furan dicarboxylic acid 3-carboxy-4-methyl-5-propyl-2-furanpropanoic acid (5-propyl FPA) accumulates in uraemic plasma and is a potent inhibitor of the binding of other anionic ligands to albumin. The interaction of 5-propyl FPA with human albumin has been investigated by equilibrium dialysis at

Decreased drug binding in uraemia: effect of indoxyl sulphate and other endogenous substances on the binding of drugs and dyes to human albumin.

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Ten uraemic metabolites, alone or in combination, have been investigated by equilibrium dialysis for their effect on the binding of methyl red, methyl orange, 2-(4'-hydroxybenzeneazo)benzoic acid (HABA), phenytoin and L-tryptophan to human albumin (HSA). Indoxyl sulphate emerges as a substance

Abnormal conformation of albumin in uraemia.

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A possible role for cyanate in the albumin binding defect of uremia.

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