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antidiuretic/инфаркт
Линкът е запазен в клипборда
Страница 1 от 61 резултата
The syndrome of inappropriate antidiuretic hormone secretion associated with acute myocardial infarction.
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A 72-year-old man developed the syndrome of inappropriate antidiuretic hormone secretion after sustaining an acute myocardial infarction. Other documented causes of this syndrome were excluded, and this case is therefore reported as a new association.
[Behavior of urinary gonadostimulins and antidiuretic activity in the post-infarct period].
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[Hyponatremia and syndrome of inappropriate antidiuretic hormone secretion in patient with cerebral infarction].
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[Oral administrated antidiabetics in diabetic patients with heart failure/myocardial infarct. Antidiuretic effect].
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Atrial natriuretic peptide in patients with acute myocardial infarction without functional heart failure.
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The purpose of the present study was to measure plasma levels of atrial natriuretic peptide (ANP) in patients with acute myocardial infarction without heart failure, and also to assess the temporal sequence of changes of plasma ANP during the first hours of recovery from myocardial infarction. The
Vascular and renal effects of vasopressin and its antagonists in conscious rats with chronic myocardial infarction; evidence for receptor shift.
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Acute myocardial infarction evokes activation of, among others, the arginine-vasopressin system, resulting in vasoconstriction and fluid retention. In the present study, the vasoconstrictor and antidiuretic effects of vasopressin were examined in vivo in conscious rats with chronic myocardial
Renal function after myocardial infarction and cardiac arrest in rats: role of ANP-induced albuminuria?
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Renal function was measured by clearance technique before and after acute myocardial infarction (MI) induced by left coronary artery ligation in male Sprague-Dawley rats. The animals were anaesthetized with halothane-nitrous oxide, paralysed with pancuronium and artificially ventilated. All
Acute myocardial infarction activates magnocellular vasopressin and oxytocin neurons.
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Myocardial infarction (MI) is a leading cause of death worldwide. For those who survive the acute insult, the progressive dilation of the ventricle associated with chronic heart failure is driven by an adverse increase in circulating levels of the antidiuretic hormone, vasopressin, which is secreted
[Granulomatous angiitis of the central nervous system complicated by the syndrome of inappropriate antidiuretic hormone].
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We report an autopsy case of granulomatous angiitis of the central nervous system (GANS) complicated by the syndrome of inappropriate antidiuretic hormone (SIADH). A 88-year old female was admitted because of progressive mental deterioration, fever, and vomiting. A computed tomogram disclosed
[Neuroleptic malignant syndrome associated with the syndrome of inappropriate secretion of antidiuretic hormone].
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We reported a case of neuroleptic malignant syndrome (NMS) associated with the syndrome of inappropriate secretion of antidiuretic hormone (SIADH). A 71-year-old woman, who had been diagnosed as hypertension and multiple cerebral infarction, was given sulpiride 150 mg daily for depressive state.
Malignant Middle Cerebral Artery Infarction Due to Hyponatremia Following Traumatic Brain Injury: A Case Report.
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BACKGROUND Development of syndrome of inappropriate antidiuretic hormone secretion or cerebral salt wasting has been commonly noted in post-traumatic brain injury, and this condition may lead to hyponatremia resulting in cerebral edema and possible cerebral herniation. However, the predominant
Transient body fluid accumulation and enhanced NKCC2 expression in gerbils with brain infarction.
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BACKGROUND
Enhanced expression of a kidney-specific sodium co-transporter (NKCC2: Na-K-2Cl co-transporter) in the thick ascending limb of Henle has been identified in rat models of congestive heart failure and liver cirrhosis, suggesting that high NKCC2 expression underlies edema formation. An
Spontaneous infarction in pituitary tumors: neurologic and therapeutic aspects.
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In addition to progressive endocrine dysfunction and progressive visual loss, pituitary neoplasms may annouce their presence by the more catastrophic alternative of spontaneous tumor infarction. In two patients reported, illness due to the spontaneous infraction of pituitary tumors was heralded by
Symptomatic hyponatremia following lateral medullary infarction: a case report.
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BACKGROUND
Hyponatremia has been reported from patients with severe neurological disease, and the syndrome of inappropriate secretion of antidiuretic hormone and cerebral salt wasting syndrome are the two main etiologies of hyponatremia after brain injury. Here we describe a patient with a lateral
Selective traumatic infarction of the human anterior hypothalamus. Clinical anatomical correlation.
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A case of assult with bilateral manual avulsion of the eyes was followed by highly selective infarction of the anterior hypothalamus. The hypothalamic infarction occurred as a result of avulsion of part of the optic chiasm together with the anterior perforating arteries passing through it; Following
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