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estradiol 17 beta/атрофия
Линкът е запазен в клипборда
Страница 1 от 40 резултата
Quantification of rainbow trout (Oncorhynchus mykiss) zona radiata and vitellogenin mRNA levels using real-time PCR after in vivo treatment with estradiol-17 beta or alpha-zearalenol.
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Estrogen receptor-mediated induction of zona radiata (ZR) and vitellogenin (VTG) mRNA and protein in rainbow trout (Oncorhynchus mykiss) was compared to assess their utility as biomarkers for exposure to estrogenic compounds. Partial sequences of rainbow trout ZR and beta-actin were cloned by
Estradiol-17 beta as an initiation modifier for radiation-induced mammary tumorigenesis of rats ovariectomized before puberty.
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This investigation evaluated the roles of estradiol-17 beta, progesterone and prolactin in the initiation of mammary tumorigenesis by irradiation. Sixty day old Wistar-MS rats ovariectomized bilaterally at 23 days of age were injected daily with olive oil, estradiol-3-benzoate (E2B), progesterone or
Comparison of the effects of gossypol, estradiol-17 beta and testosterone compensation on male rat reproductive organs.
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The effects of gossypol acetic acid (GAA) were compared with those induced by estradiol-17 beta (E2), testosterone, and a combination of these steroids. GAA was administered s.c. to adult rats at doses of 25 to 30 mg . kg BW . day for 30 days, while steroids in polydimethylsiloxane tubing of various
Differential response of testis and serum gonadotropins to testosterone in rats treated with a gonadotropin releasing hormone antagonist or estradiol 17 beta.
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Adult rats treated with a GnRH antagonist (Ac D2Nal1, D4Cl Phe2, DTrp3, DArg6, DAla10 GnRH; code: 103-289-10, National Institutes of Health, USA) for 5 weeks (250 micrograms/kg b.w) showed multiple degrees of impairment and atrophy of the genital organs concomitant with decreased serum levels of
3,3 diindolylmethane leads to apoptosis, decreases sperm quality, affects blood estradiol 17 β and testosterone, oestrogen (α and β) and androgen receptor levels in the reproductive system in male rats.
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3,3 Diindolylmethane (DIM) is a major digestive product of indole-3 carbinol, obtained from Brassica family vegetables such as broccoli, cabbage and Brussels sprouts. This study aimed to investigate the effects of DIM on sperm parameters, histological structures of testicular tissues, blood
Markers of atresia in ovarian follicular components from rhesus monkeys treated with estradiol-17 beta.
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A new model for the investigation of atresia in rhesus monkeys is presented. This model is based upon the reliable induction of an atresia-like process in the dominant preovulatory follicle (DF) by estradiol-17 beta administered subcutaneously via Silastic capsules for 24 h. Data obtained from
Effects of estradiol-17 beta on reproduction in adult male rats.
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Estradiol-17 beta (E2-17 beta) treatment induced dose-related reduction of sperm population and eventual azoospermia, atrophy of accessory organs, and suppressed serum LH and testosterone levels. In spite of all these alterations mating behavior was not affected, although the number of implantation
Endocrine changes associated with cystic ovarian degeneration in sows.
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Endocrine changes in sows with ovarian cysts were determined to be comparable with those in sows in diestrus. Blood samples were collected at frequent intervals from 5 sows affected with cystic ovaries. Concentrations of serum luteinizing hormone, progesterone, estradiol-17 beta, and cortisol were
Depletion of [3H]methyltrienolone cytosol binding in glucocorticoid-induced muscle atrophy.
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The present study was undertaken to determine cytosol binding properties of [3H]methyltrienolone, a synthetic androgen, in comparison with [3H]dexamethasone, a synthetic glucocorticoid, under conditions of glucocorticoid excess in skeletal muscle. Male hypophysectomized rats received either seven
A rapid method for the isolation and culture of endometrial epithelial cells responsive to estradiol.
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Epithelial endometrial cells were isolated from normal and ovariectomized adult rats. The procedure involves successively (1) perfusion of the uterine vasculature with calcium-free Hanks' solution, (2) perfusion with 0.05% collagenase and 0.1% hyaluronidase in calcium-free Hanks' solution, (3)
Sex inversion of sexually immature honeycomb grouper (Epinephelus merra) by aromatase inhibitor.
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Previous studies have shown that estrogen plays an important role in sex change of protogynous honeycomb grouper, and that the treatments with aromatase inhibitor (AI) cause estrogen depletion and complete sex inversion of pre-spawning females into functional males. In the present study, we examined
Preimplantation embryo development and serum steroid levels in immature rats induced to ovulate or superovulate with pregnant mares' serum gonadotropin injection or follicle-stimulating hormone infusions.
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Follicular stimulation protocols using pregnant mares' serum gonadotropin (PMSG) or a follicle-stimulating hormone (FSH) preparation were compared to evaluate the yield and quality of embryos obtained from immature rats. Rats received a superovulatory dose of PMSG (40 IU), a nonsuperovulatory dose
Effects of androgen and antiandrogen treatment on canine prostatic arginine esterase.
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A series of experiments were conducted to investigate the regulation of the primary secretory protein of the canine prostate, arginine esterase, by androgens and/or new antiandrogen under development. In the first experiment, castration decreased (P less than 0.05) prostatic arginine esterase levels
Androgen-supported estrogen-enhanced epithelial proliferation in the prostates of intact Noble rats.
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Using a stathmokinetic in vivo metaphase-arrest technique, we studied cell proliferation and histological changes in the ventral (VP) and dorsolateral (DLP) prostate lobes of intact Noble (Nb) rats following a 16 week treatment with testosterone (T) or 5 alpha-dihydrotestosterone (DHT) administered
Morphological pattern elicited by agents affecting spermatogenesis by stimulation.
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A variety of agents (clomiphene citrate, cyproterone acetate, estradiol 17-beta, medrogestone, medroxyprogesterone, and methoxychlor) thought to disrupt the normal pathway by which hormones stimulate the testis were tested in rats to determine the early morphologic changes in the testis. After
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