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Страница 1 от 75 резултата
Raspberry ketone protects against isoproterenol-induced myocardial infarction in rats.
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OBJECTIVE
The cardioprotective role of raspberry ketone (RK) against isoproterenol (ISO)-induced myocardial infarction (MI) in rats was assessed.
METHODS
Rats were randomly divided into Group I - Vehicle control; Group II - Toxic control ISO (85mg/kg, s.c.); Group III, IV and V - RK (50, 100 and
[Behavior of ketone bodies and some carbohydrate metabolism indicators in the blood of patients with cerebral infarction in the earliest and later stages after intravenous glucose loading].
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[Intraoperative acute myocardial infarction during total gastrectomy under general anesthesia combined with thoracic epidural anesthesia].
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A 61-year-old, 54-kg man with gastric cancer was scheduled for total gastrectomy under general anesthesia combined with thoracic epidural anesthesia. Approximately 20 minutes after the start of the operation, the patient developed sudden hypotension and ventricular fibrillation. The cardiac rhythm
The determination of ketone bodies: preanalytical, analytical and physiological considerations.
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The arterial ketone body ratio is calculated as the ratio of arterial levels of acetoacetate/beta-hydroxybutyrate. It correlates with survival in experimental hemorrhagic shock and outcome after liver surgery and myocardial infarction. Procedures for determination of ketone bodies are often
Dietary nonprotein calories and cerebral infarction size in rats.
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OBJECTIVE
Conventional diets may cause hyperglycemia in patients with neurological injuries. The purpose of this study was to examine the effect on the severity of cerebral infarction of replacing carbohydrates as the primary dietary source of nonprotein calories.
METHODS
Sixty-nine Long-Evans rats
Cardiac metabolism as a driver and therapeutic target of myocardial infarction.
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Reducing infarct size during a cardiac ischaemic-reperfusion episode is still of paramount importance, because the extension of myocardial necrosis is an important risk factor for developing heart failure. Cardiac ischaemia-reperfusion injury (IRI) is in principle a metabolic pathology as it is
Imaging the Effects of β-Hydroxybutyrate on Peri-Infarct Neurovascular Function and Metabolism.
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Background and Purpose- Recent evidence suggests great potential of metabolically targeted interventions for treating neurological disorders. We investigated the use of the endogenous ketone body β-hydroxybutyrate (BHB) as an alternate metabolic substrate for the brain in the acute phase of ischemia
Role and therapeutic potential of dietary ketone bodies in lymph vessel growth
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Lymphatic vessels (LVs), lined by lymphatic endothelial cells (LECs), are indispensable for life1. However, the role of metabolism in LECs has been incompletely elucidated. In the present study, it is reported that LEC-specific loss of OXCT1, a key enzyme of ketone body
Empagliflozin, an SGLT2 Inhibitor, Reduced the Mortality Rate after Acute Myocardial Infarction with Modification of Cardiac Metabolomes and Antioxidants in Diabetic Rats.
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The mechanism by which SGLT2 inhibitors reduce cardiac events in diabetic patients remains unclear. Here, we examined the effects of an SGLT2 inhibitor on the acute survival rate after myocardial infarction (MI) in an animal model of type 2 diabetes mellitus (DM) and the possible involvement of
Sirtuin 3 mediates neuroprotection of ketones against ischemic stroke.
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Stroke is one of the leading causes of death. Growing evidence indicates that ketone bodies have beneficial effects in treating stroke, but their underlying mechanism remains unclear. Our previous study showed ketone bodies reduced reactive oxygen species by using NADH as an electron donor, thus
Impact of EMpagliflozin on cardiac function and biomarkers of heart failure in patients with acute MYocardial infarction-The EMMY trial.
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Targeting the glucagon receptor improves cardiac function and enhances insulin sensitivity following a myocardial infarction.
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Critical role of complex III in the early metabolic changes following myocardial infarction.
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OBJECTIVE
The chronically infarcted rat heart has multiple defects in metabolism, yet the location of the primary metabolic abnormality arising after myocardial infarction is unknown. Therefore, we investigated cardiac mitochondrial metabolism shortly after infarction.
RESULTS
Myocardial infarctions
Differential network analysis reveals metabolic determinants associated with mortality in acute myocardial infarction patients and suggest potential mechanisms underlying different clinical scores used to predict death.
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Acute myocardial infarction (AMI) is a leading worldwide cause of death. Risk stratification and management of AMI patients continue to be challenging despite considerable efforts made in the last decades. We present here differential analysis of metabolite-metabolite association networks
Association Between Circulating Ketone Bodies and Worse Outcomes in Hemodialysis Patients.
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BACKGROUND
Cardiovascular disease is the leading cause of morbidity and mortality in patients receiving hemodialysis. Systemic metabolic perturbation is one of the hallmark abnormalities in patients at high cardiovascular risk. We sought to determine the relationship between circulating ketone body
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