periodontitis/хипоксия

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[New concept on the mechanism of hypoxia in case of periodontitis].

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In examined groups of healthy people as well as in persons suffering from a periodontitis of a different severity-degree with concomitant affections and without them the PO2 in the blood is defined by the concentration of K+ and Cl- in erythrocytes and in blood as well as by the relation between the

The role of psychologic stress-induced hypoxia-inducible factor-1α in rat experimental periodontitis.

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BACKGROUND Studies have shown that psychologic stress plays a significant role in the outcome of many diseases. The present study is designed to investigate the effect of stress on experimental ligature-induced periodontal disease in rats by means of a variable moderate chronic stress

Expression of RUNX2 and MDM21 in rats with periodontitis under chronic intermittent hypoxia.

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OBJECTIVE To discuss the expression of RUNX2 and MDM21 in rats with periodontitis under the chronic intermittent hypoxia. METHODS A total of 32 SD healthy rats were randomly divided into four groups, with 8 rats in each group. The molecular biological techniques of immunohistochemistry, RT-PCR and

[Effects of interleukin-18 and hypoxia-inducible factor-1α in serum and gingival tissues of rat model with periodontitis exposed to chronic intermittent hypoxia].

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UNASSIGNED This study evaluates the expression of interleukin-18 (IL-18) and hypoxia-inducible factor (HIF)-lα in rat periodontitis model exposed to normoxia and chronic intermittent hypoxia (CIH) environments. The possible correlation between periodontitis and obstructive sleep apnea-hypopnea

Mandibular biomechanical behavior of rats submitted to chronic intermittent or continuous hypoxia and periodontitis

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Background: The aim of this study was to investigate the effects of exposure to continuous (CH) and intermittent (IH) hypoxia on biomechanical properties of the mandible and periodontal tissue of animals submitted to experimental

Effect of non-surgical periodontal treatment on gingival crevicular fluid hypoxia inducible factor-1 alpha, vascular endothelial growth factor and tumour necrosis factor- alpha levels in generalized aggressive periodontitis patients.

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Hypoxia-inducible angiogenic pathway involving hypoxia inducible factor-1 alpha (HIF-1α), vascular endothelial growth factor (VEGF) and tumour necrosis factor- alpha (TNF-α) may regulate several biological processes related to inflammation. The present study aimed to assess the effect

Detection of bacterial diversity in rat's periodontitis model under imitational altitude hypoxia environment.

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OBJECTIVE Oral epidemiologic investigations in China western territory have showed that the immigrants in the plateau have a higher morbidity with periodontitis. To find the possible relationship between the pathogenesis of periodontitis and altitude hypoxia, we designed a periodontitis rat model

[Effects of the activity of superoxide dismutase in blood serum and gingival tissues of rabbit in periodontitis model after hypoxia exposure at high altitude].

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OBJECTIVE To observe the activity of superoxide dismutase (SOD) in rabbit periodontitis model in normoxia and hypoxia environment, and to research the pathogenesis of periodontal diseases at high altitude. METHODS Fourty male rabbits were randomly divided into four groups: Normoxia control group,

A disintegrin-like And metalloproteinase with thrombospondin-1 (ADAMTS-1) levels in gingival crevicular fluid correlate with vascular endothelial growth factor-A, hypoxia-inducible factor-1α, and clinical parameters in patients with advanced periodontitis.

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ADAMTS (A disintegrin-like and metalloproteinase with thrombospondin) are a family of proteinases that are structurally similar to the family of matrix metalloproteinases with critical roles in damage and repair of the extracellular matrix. Their functions are closely related to

Evaluation of apoptosis and hypoxia-related factors in gingival tissues of smoker and non-smoker periodontitis patients.

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Smoking causes pathological changes in all tissues, including gingiva and alveolar bone. The aim of present study was to evaluate apoptotic tissue alterations and tissue destruction in smoker and non-smoker periodontitis patients and healthy

Porphyromonas gingivalis-Derived Lipopolysaccharide Combines Hypoxia to Induce Caspase-1 Activation in Periodontitis.

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Periodontitis is defined as inflammation affecting the supporting tissue of teeth. Periodontal pathogens initiate the disease and induce inflammatory host response. Hypoxia may accelerate the process by producing pro-inflammatory factors. The aim of this study is to investigate the effect of

Hypoxia and collagen crosslinking in the healthy and affected sites of periodontitis patients.

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Present study suggests that diseased sites of periodontitis with stage 3 grade B and C had decreased fibroblast cell density, hypoxia-inducible factor (HIF) and vascular endothelial growth factor (VEGF) expressions while increased inflammatory cell counts compared to both healthy sites of

LPS from P. gingivalis and hypoxia increases oxidative stress in periodontal ligament fibroblasts and contributes to periodontitis.

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Oxidative stress is characterized by an accumulation of reactive oxygen species (ROS) and plays a key role in the progression of inflammatory diseases. We hypothesize that hypoxic and inflammatory events induce oxidative stress in the periodontal ligament (PDL) by activating NOX4. Human primary PDL

A20 inhibits osteoclastogenesis via TRAF6-dependent autophagy in human periodontal ligament cells under hypoxia.

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A20 exerts an anti-osteoclastogenic effect through the inhibition of NF-κB signalling in periodontitis. It also regulates autophagy via ubiquitin modification. This study was aimed at exploring the relationship between A20 and autophagy in anti-osteoclastogenesis in human periodontal

Evaluation of serum ceruloplasmin in aggressive and chronic periodontitis patients.

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BACKGROUND Pro-inflammatory markers are seen to increase in inflammatory diseases like periodontitis. Detecting an increase in these markers is one of the diagnostic modality. One such marker, which can be detected, is the ceruloplasmin. Ceruloplasmin induces hypoxia and generates oxygen radicals at

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