wallerian degeneration/албумин

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The blood-nerve barrier in Wallerian degeneration: a sequential long-term study.

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The blood-nerve barrier (BNB) for serum proteins was studied after a crush lesion of the murine sciatic nerve or after transsection with persistent Wallerian degeneration. Using single intraperitoneal injections of biotinylated human albumin, transferrin, IgG, and complement components as tracers,

Wallerian degeneration induces Ia-antigen expression in the rat brain.

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Strong expression of class II major histocompatibility (MHC II, Ia) antigens was observed in areas of Wallerian degeneration following either a cryoinjury to the cerebral and cerebellar cortex or unilateral eye enucleation in adult Wistar and Lewis rats. The Wallerian degeneration was disclosed by

Pathology of lumbar nerve root compression. Part 1: Intraradicular inflammatory changes induced by mechanical compression.

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METHODS This study is to investigate the intraradicular inflammation induced by mechanical compression using in vivo model. OBJECTIVE The relationship between the intraradicular edema and nerve fiber degeneration induced by mechanical compression was determined in the nerve root. BACKGROUND Recently

Synapse involvement of the dorsal horn in experimental lumbar nerve root compression: a light and electron microscopic study.

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METHODS This study was aimed at investigating changes in the dorsal horn of the lumbar cord induced by mechanical compression using an in vivo model. OBJECTIVE To determine the effect of axonal flow disturbance in the dorsal horns induced by nerve root compression. BACKGROUND Few studies have looked

Anterograde-propagation of axonal degeneration in the visual system of wlds mice characterized by diffusion tensor imaging.

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To evaluate the feasibility of using diffusion tensor imaging (DTI) to characterize the temporospatial profile of axonal degeneration and its relation to blood-brain barrier (BBB) permeability. Longitudinal DTI was performed in Wallerian degeneration slow (WldS) mice following retinal ischemia. In

Biosynthesis of long-chain alcohols by developing and regenerating rat sciatic nerve.

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Cell-free preparations of rat sciatic nerve were found to catalyze the reduction of fatty acid to alcohol in the presence of NADPH as reducing cofactor. The reductase was membrane-bound and associated primarily with the microsomal fraction. When fatty acid was the substrate, ATP, coenzyme A (CoA),

Revascularization of tissue-engineered nerve grafts and invasion of macrophages.

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Nonneural derived nerve conduits fail to support regeneration over larger gaps due to lacking viable Schwann cells. Thus, tissue engineering of nerves is focusing on implantation of viable Schwann cells into suitable scaffolds. We established grafts made from acellular muscles and veins,

Direct immunofluorescence findings in peripheral nerve from patients with diabetic neuropathy.

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Direct immunofluorescence examination was performed on peripheral nerve from 16 patients with diabetes mellitus and 53 additional patients with peripheral neuropathy of diverse cause. Six nerves from patients with diabetes mellitus yielded positive findings: 4 had granular and lamellar deposition of

Imaging of cauda equina edema in lumbar canal stenosis by using gadolinium-enhanced MR imaging: experimental constriction injury.

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OBJECTIVE It has been reported that disturbance of blood flow arising from circumferential compression of the cauda equina by surrounding tissue plays a major role in the appearance of neurogenic intermittent claudication (NIC) associated with lumbar spinal canal stenosis (LSCS). We created a model

Cerebrospinal fluid and plasma apolipoproteins in patients with multiple sclerosis.

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Apolipoprotein (apo) E is synthesized by cells of the central and peripheral nervous systems, and its synthesis and secretion in the peripheral nervous systems, and its synthesis and secretion in the peripheral nervous system of animals are greatly stimulated following Wallerian degeneration. It has

Wld(S) ameliorates renal injury in a type 1 diabetic mouse model.

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Diabetic nephropathy (DN) is the leading cause of end-stage kidney disease worldwide. The purpose of this study is to investigate whether the Wld(S) (slow Wallerian degeneration; also known as Wld) gene plays a renoprotective role during the progression of DN. Diabetes was induced in 8-wk-old male

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