arteriosclerosis/phosphatase

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[Changes in the total calcium content and in the activity of alkaline phosphatase and gamma-glutamyltransferase in the blood serum of rats with Mönckeberg's experimental arteriosclerosis].

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Experiments on rats (single-action, six-days' hyperepinephrinemia and monoiodacetatic acid intoxication) were performed to study the calcium concentration, alkaline phosphatase and gamma-glutamyltransferase activities in serum at Monckeberg's medial calcific sclerosis modelment. It is established

[Alkaline phosphatase and ATPase activity in the walls of the cerebral vessels in hypertension and arteriosclerosis with disorders of the cerebral circulation].

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In hypertensive disease and atherosclerosis without acute disorders of cerebral circulation it was established that in the cerebral vessel walls there was a high activity of alkaline phosphatase and adenosintriphosphtase. In the symmetrical areas of the subcortical nodes differences in the activity

[Specific phosphatase (5-nucleotidase, ATP-pyrophosphatase) and aspecific phosphatase in normal arterial walls, human arteriosclerosis, experimental adrenalin arterial pathology].

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Serum hepatocyte growth factor as a possible indicator of arteriosclerosis.

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OBJECTIVE To investigate the possible involvement of hepatocyte growth factor in arteriosclerotic lesions, by studying the relationship between serum concentrations of hepatocyte growth factor and grades of retinal arteriosclerosis. METHODS We measured the blood pressure, body mass index, serum

Lipid phosphate phosphatase inhibitors locally amplify lysophosphatidic acid LPA1 receptor signalling in rat brain cryosections without affecting global LPA degradation.

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BACKGROUND Lysophosphatidic acid (LPA) is a signalling phospholipid with multiple biological functions, mainly mediated through specific G protein-coupled receptors. Aberrant LPA signalling is being increasingly implicated in the pathology of common human diseases, such as arteriosclerosis and

Graft permeabilization facilitates gene therapy of transplant arteriosclerosis in a rabbit model.

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BACKGROUND Smooth muscle cell (SMC) replication plays a central role in the pathogenesis of transplant arteriosclerosis. One strategy to eliminate dividing cells is to express a herpesvirus thymidine kinase (tk) gene that phosphorylates the nucleoside analogue ganciclovir into a toxic form leading

Mitogen-activated protein kinase phosphatase-1 in rat arterial smooth muscle cell proliferation.

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Smooth muscle cell proliferation and migration is important in arteriosclerosis. In this process, cytokines and growth factors are upregulated and bind to their respective receptors, which in turn stimulate mitogen-activated protein (MAP) kinases. MAP kinases then relay signals to the nucleus that

FAL1 regulates endothelial cell proliferation in diabetic arteriosclerosis through PTEN/AKT pathway.

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OBJECTIVE This study aims to investigate the role of FAL1 in the occurrence and progression of diabetic arteriosclerosis and its underlying mechanism. METHODS FAL1 expression in coronary artery disease (CAD) tissues, normal artery tissues, and tumor necrosis factor-α (TNF-α)-induced endothelial

Which parameter is more influential on the development of arteriosclerosis in hemodialysis patients?

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Arteriosclerosis is characterized by stiffening of arteries. The incremental elastic modulus (Einc) measurement is a good marker of arterial wall stiffness. Metabolic, inflammatory and hemodynamic alterations cause structural changes and vascular complications in end stage renal disease. The aim of

Melatonin Attenuates Aortic Endothelial Permeability and Arteriosclerosis in Streptozotocin-Induced Diabetic Rats: Possible Role of MLCK- and MLCP-Dependent MLC Phosphorylation.

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The development of diabetic macrovascular complications is a multifactorial process, and melatonin may possess cardiovascular protective properties. This study was designed to evaluate whether melatonin attenuates arteriosclerosis and endothelial permeability by suppressing the myosin light-chain

Effect of occlusal disorder on the gingiva in rats with experimental arteriosclerosis. Enzyme histochemical and biochemical study.

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This histochemical study showed that experimentally arteriosclerotic rats, subjected to an occlusal stress of six weeks' duration, showed an inflammation-related increase of chloride-activated arginine aminopeptidase and acid phosphatase activity in the basal cells of the gingival epithelium. The

[Characteristics of metabolism in patients with arteriosclerosis obliterans and arteritis].

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Lipid, carbohydrate and protein metabolic parameters, such as triglycerides, total and alpha-cholesterol, total protein and albumin, urea nitrogen, creatinine, glucose and the activity of alanine and aspartate aminotransferases, alkaline phosphatase, total lactate dehydrogenase and creatine

[Histochemical characteristics of the vascular-capillary bed of the brain during aging and in arteriosclerosis].

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Using the alkaline phosphatase test, the authors studied the capillaries of 41 fields in clinically healthy people aged 22-64 years and animals (dogs, cats, rats) and also in individuals aged 45 to 64 years with mild atherosclerosis in the area of the median cerebral artery trunk. It has been

Rho-kinase inhibitors.

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Pulmonary arterial hypertension (PAH) is a fatal disease with poor prognosis characterized by progressive elevation of pulmonary arterial pressure and vascular resistance due to pulmonary artery hyperconstriction and remodeling; however, the precise mechanism of PAH still remains to be elucidated.

Recent progress in the treatment of pulmonary arterial hypertension: expectation for rho-kinase inhibitors.

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Pulmonary arterial hypertension (PAH) is a disease with poor prognosis characterized by progressive elevation of pulmonary arterial pressure and vascular resistance due to pulmonary artery hyperconstriction and remodeling. However, the precise mechanism of PAH still remains to be elucidated.

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