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Page 1 od 478 rezultati
Cerebellar ataxia a unique initial presentation of Legionnaires' disease.
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We present a case of Legionnaires' disease complicated by cerebellar ataxia. A 60-year-old man was diagnosed with Legionnaires' disease by urine antigen after presenting to the hospital with a main problem of headache and gait instability. He also had a productive cough, as well as nausea, vomiting
Paraneoplastic syndrome manifesting as chronic cerebellar ataxia in a child with Hodgkin disease.
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An 8-year-old boy had nausea and vomiting associated with nystagmus, ataxia, and dysarthria of acute onset. Three years later he had a mass in the anterior mediastinum as a result of Hodgkin disease of mixed cellularity. This association of paraneoplastic cerebellar degeneration with Hodgkin disease
Treatment for speech disorder in Friedreich ataxia and other hereditary ataxia syndromes.
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BACKGROUND
Hereditary ataxia syndromes can result in significant speech impairment, a symptom thought to be responsive to treatment. The type of speech impairment most commonly reported in hereditary ataxias is dysarthria. Dysarthria is a collective term referring to a group of movement disorders
Capecitabine-associated cerebellar ataxia.
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OBJECTIVE
A case of capecitabine-associated cerebellar ataxia is presented.
CONCLUSIONS
A 65-year-old white woman with stage IV colorectal cancer with liver metastasis was started on a chemotherapy regimen of capecitabine, oxaliplatin, and bevacizumab, given every three weeks. She tolerated the
The cannabinoid CB1 antagonist AM 251 produces food avoidance and behaviors associated with nausea but does not impair feeding efficiency in rats.
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BACKGROUND
A growing body of evidence suggests that cannabinoid CB1 receptor antagonists have potential therapeutic utility as appetite suppressants. However, the specific mechanisms underlying the reduction in food intake produced by these drugs are not well understood.
OBJECTIVE
Considering the
Non-familial periodic ataxia responding to acetazolamide.
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A 44-year-old man presented with symptoms of periodic ataxia, dysarthria, nausea and excessive sweating during the last twenty years. These symptoms could be provoked by physical or emotional stress and disappeared after bedrest for several hours. No other members of his family were known to have
Clinical and molecular correlations in spinocerebellar ataxia type 6: a study of 24 Dutch families.
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BACKGROUND
Autosomal dominant cerebellar ataxias (ADCAs), or spinocerebellar ataxias (SCAs), are a heterogeneous group of neurodegenerative disorders. Mild CAG repeat expansions in the alpha(1A) voltage-dependent calcium channel gene are associated with SCA type 6 (SCA6).
OBJECTIVE
To obtain further
Acute cerebellar ataxia with abnormal MRI lesions after varicella vaccination.
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A 2-year-old boy, with the primary difficulties of nausea and vomiting, developed a staggering gait and dysarthria 10 days after varicella vaccination. Magnetic resonance imaging demonstrated multiple areas of high signal intensity in the white matter of the cerebellum, predominantly in the
Transient cerebellopontine demyelinisation revealed by MRI in acute cerebellar ataxia.
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An eight year old boy was admitted to our ward with a history of abrupt onset of rapidly progressive gait disorder, nausea, vertigo and vomiting. The clinical as well as the laboratory findings suggested the diagnosis of acute cerebellar ataxia. Magnetic resonance imaging (MRI), however, showed
Downward vertical gaze palsy as a prominent manifestation of episodic ataxia type 2: a case report.
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OBJECTIVE
Episodic ataxia type 2 (EA2) is an inherited autosomal dominant disorder characterized by intermittent ataxia, nausea, vomiting, dysarthria, or nystagmus. We report a case of EA2, which downward gaze palsy exists as a common sign in all her attacks. Responsiveness of EA2 to acetazolamide
Prolonged vertigo and ataxia after mandibular nerve block for treatment of trigeminal neuralgia.
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Common complications of neurolytic mandibular nerve block are hypoesthesia, dysesthesia, and chemical neuritis. We report a rare complication, prolonged severe vertigo and ataxia, after neurolytic mandibular blockade in a patient suffering from trigeminal neuralgia. Coronoid approach was used for
[MRI and SPECT findings in a case of metronidazole-induced reversible acute cerebellar ataxia].
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A 69-year-old man was referred to our department because of acute onset nausea, vomiting, dysphagia, dysarthria and gait disturbance. He had a 50-day-history of amebic dysentery and had been treated with 1,500 mg metronidazole per day. Neurological examination revealed dysphagia, ataxic speech,
A sleep modulated Channelopathy: a novel CACNA1A pathogenic variant identified in episodic Ataxia type 2 and a potential link to sleep alleviated migraine.
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[Acute cerebellar ataxia with sympathotonic orthostatic hypotension following Epstein-Barr virus infection--a case report].
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A 21-year-old man with acute cerebellar ataxia and sympathotonic orthostatic hypotension, following Epstein-Barr (EB) virus infection, was reported. He noticed unsteady gait 2 weeks after the development of cough, nausea and vomiting. On admission, he was unable to sit and walk due to truncal ataxia
[Dyskinesia after treatment with droperidol for preventing postoperative nausea and vomiting].
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This case presents a young woman with extrapyramidal reactions after the use of a small dose (0.625 mg) of droperidol. In combination with dexamethason (8 mg) droperidol was given as the patient underwent laparoscopic cholecystectomy in order to prevent postoperative nausea and vomiting. The patient
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