avitaminosis/hypoxia

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Iodide metabolism during anoxia, nephrectomy, trauma, avitaminoses and starvation in the rat.

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Pulmonary hypertension associated with scurvy and vitamin deficiencies in an autistic child.

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Restricted dietary intake is common among children with behavioral issues. Here we report a case of a severely autistic child who presented initially with limp but who soon developed cough, tachypnea, hypoxia, and tachycardia. An echocardiogram revealed evidence of pulmonary hypertension (PH) with

[Vitamins and oxidative stress].

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The central and local stress limiting systems, including the antioxidant defense system involved in defending the organism at the cellular and systemic levels from excess activation response to stress influence, leading to damaging effects. The development of stress, regardless of its nature [cold,

[The transketolase activity in erythrocytes as a criterion for thiamine balance in the body].

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In deficiency of thiamin the transketolase activity was decreased in patients and in experimental animals. The data obtained showed the high sensitivity of transketolase from erythrocytes to hypovitaminosis B1. At the same time, in diseases, especially accompained by hypoxia (and also by

Environmental aspects of congenital scoliosis.

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Growing evidence has proved that many aspects of our lifestyle and the environment contribute to the development of congenital disease. Congenital spinal deformities are due to anomalous development of the vertebrae including failure of formation and segmentation during embryogenesis. The causes of

Virus myocarditis: a critique of the literature from clinical, electrocardiographic, and pathologic standpoints.

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Concurrent viral infection and myocarditis presumably indicate viral myocarditis. The electrocardiographic and pathologic changes developing during acute infection may, however, result from changes not produced by the infection itself, eg, fever, tachycardia, ischemia, potassium depletion, vitamin

ZO proteins and redox-dependent processes.

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CONCLUSIONS ZO-1, ZO-2, and ZO-3 are scaffold proteins of the tight junction (TJ) that belong to the MAGUK protein family characterized for exhibiting PDZ, SH3, and GuK domains. ZO proteins are present only in multicellular organisms, being the placozoa the first to have them. ZO proteins associate

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