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flavanoid/krvarenje

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Role of the indigenous drug saptamrita lauha in hemorrhagic retinopathies.

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A clinical study was done on 48 eyes to assess the role of an indigenous drug, Saptamrita Lauha, in the absorption of hemorrhages and prevention of their recurrence in retinopathic eyes. Rapid absorption of retinal hemorrhages was observed in both treated groups: diabetic and hypertensive (mean
To study flavanoids extracted from onion (FEO) on the number of activated microglia and the release of proinflammatory factors in intracerebral hemorrhage (ICH) model rat at different time points, and to explore its possible mechanism for treating

Antiviral actions of flavanoid-derived compounds on dengue virus type-2.

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Dengue viruses, mosquito-borne members of the Flaviviridae family, are the causative agents of dengue fever and its associated complications, dengue haemorrhagic fever and dengue shock syndrome. To date, more than 2.5 billion people in over 100 countries are at risk of infection, and approximately

The teratogenicity and the action mechanism of gallic acid relating with brain and cervical muscles.

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Gallic acid (3,4,5-trihydroxybenzoic acid) (GA) and other flavanoids are extensively used in nutraceuticals because of their antioxidant and antiinflammatory properties. While examining whether GA is effective in alleviating valproic-acid-induced teratogenesis in a chicken embryo model (CEM), we
OBJECTIVE To investigate efficacy and side effects of Cissus quadrangularis L. and micronised purified flavanoid fraction (MPFF) in treatment of hemorrhoids. METHODS A prospective double blind randomized study was designed in acute hemorrhoidal patients from three hospitals. In each hospital, eighty

Acute mental stress and hemostasis: When physiology becomes vascular harm.

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Stress-induced activation of the sympathoadrenal medullary system activates both the coagulation and fibrinolysis system resulting in net hypercoagulability. The evolutionary interpretation of this physiology is that stress-hypercoagulability protects a healthy organism from excess bleeding should

ACUTE MENTAL STRESS AND HEMOSTASIS: WHEN PHYSIOLOGY BECOMES VASCULAR HARM.

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Stress-induced activation of the sympathoadrenal medullary system activates both the coagulation and fibrinolysis system resulting in net hypercoagulability. The evolutionary interpretation of this physiology is that stress-hypercoagulability protects a healthy organism from excess bleeding should
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