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hepatomegaly/edema

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Massive hepatomegaly is a common finding in hydrops fetalis (HF) arising from hemoglobinopathies. It has been suggested that extramedullary hematopoiesis, which is markedly increased in response to anemia, plays a principal role in hepatomegaly via sinusoidal obstruction and distortion of the

Hepatomegaly and edema.

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[Hydrops of the gallbladder associated with Epstein-Barr virus infection].

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A 50-year-old male developed Hydrops of Gallbladder during the course of Epstein-Barr virus infection. The patient had a history of acute encephalitis one month prior to admission. Physical examination revealed jaundice and hepatomegaly. Liver function tests were abnormal and the white blood count

Fetal cytomegalovirus infection associated with cerebral hemorrhage, hydrops fetalis, and echogenic bowel: case report.

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We describe some fetal ultrasound findings associated with intrauterine cytomegalovirus (CMV) infection. We report a 38-year-old gravida 3, para 2 at 16 weeks of gestation who underwent ultrasound examination for anomaly screening. The scan revealed an extensive irregular echogenic area in the fetal

[Clinical aspects of stasis hepatomegaly].

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The clinical characteristics of 100 patients diagnosed of congestive hepatomegaly have been reviewed in order to analyze some of the clinical and analytical parameters. The protocol for the study consisted of ten variables: sex, age, quality and size of the hepatomegaly, presence of hepatojugular

Nonimmune hydrops fetalis secondary to premature closure of the foramen ovale.

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Premature closure of the foramen ovale is a rare cardiac anomaly. Severe right-sided heart failure accounts for the hepatomegaly, ascites, pleural and pericardial effusions, and generalized edema of these hydropic fetuses. Premature closure of the foramen ovale should be a part of the differential
A syndrome which is known as plasma cell dyscrasia with polyneuropathy and various endocrine manifestations or plasma cell dyscrasia with polyneuropathy, organomegaly, endocrinopathy, abnormal M protein and skin changes is very interesting because this syndrome has miscellaneous manifestations such

Antenatal-onset infantile cortical hyperostosis and nonimmune hydrops.

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Infantile cortical hyperostosis antenatal onset is an uncommon disease characterized by polyhydramnios, anasarca or hydrops, pulmonary hypoplasia, hepatomegaly, bowed hyperostotic long bones, and a poor prognosis. Sonographically the intrauterine manifestations may be similar to those of

Comparative efficacy and safety of bumetanide and furosemide in long-term treatment of edema due to congestive heart failure.

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Bumetanide and furosemide were compared for efficacy in reducing edema due to congestive heart failure in 28 patients (21 receiving bumetanide and seven receiving furosemide) in a long-term study for periods from one week to 18 months. In both groups the patients showed decreases in body weight,

Edema, anemia, hypoproteinemia, and acrodermatitis enteropathica: an uncommon initial presentation of cystic fibrosis.

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Cystic fibrosis is a genetic disorder characterized by chronic obstructive pulmonary disease, pancreatic exocrine deficiency, and abnormally high sweat electrolyte concentrations. Less frequently, the presenting features in infants may include edema, anemia, hypoproteinemia, and acrodermatitis

[Insulin edema in hepatic glycogenosis].

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BACKGROUND Hepatic glycogenosis is a rare syndrome, which includes poorly controlled diabetes mellitus, hepatomegaly, delayed puberty, and growth delay. Insulin edema is sometimes associated. METHODS An 18-year-old woman presented with diffuse edema, hepatomegaly, amenorrhea, uncontrolled diabetes,

Epidemic dropsy--a clinical study of the Delhi outbreak.

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BACKGROUND A major outbreak of epidemic dropsy occurred in Delhi, India, in August-September 1998, due to the consumption of contaminated mustard oil. METHODS The clinical data of 212 adult patients of epidemic dropsy who presented to our hospital is analysed. RESULTS Pitting pedal oedema (100%),

Pulmonary congestion and edema (marble spleen disease) of chickens produced by group II avian adenovirus.

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"Marble spleen disease" of chickens was diagnosed in 22-week-old chickens. Total mortality was 8.9%. Deaths occurred over a period of 2 months. Gross lesions included pulmonary congestion, splenomegaly, hepatomegaly, and congestion of egg follicles. Microscopic lesions included pulmonary congestion
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