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hypothyroidism/kalij
Veza se sprema u međuspremnik
Page 1 od 262 rezultati
Treatment of amiodarone-induced hypothyroidism with potassium perchlorate.
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The antiarrhythmic drug, amiodarone, induces thyroid dysfunction, which is potentially dangerous in cardiac patients. After discontinuation of the drug it takes several months before euthyroidism is restored. The potent antithyroid drug, potassium perchlorate (KClO4), is used successfully to treat
Potassium perchlorate only temporarily restores euthyroidism in patients with amiodarone-induced hypothyroidism who continue amiodarone therapy.
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BACKGROUND
Amiodarone-induced hypothyroidism (AIH) may occur in patients with or without underlying thyroid disorders. In the latter, restoration of euthyroidism, after amiodarone discontinuation, can be facilitated and accelerated by a short course of potassium perchlorate (KClO4). However, it is
[Transitory efficacy of potassium perchlorate in hypothyroidism caused by amiodarone].
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We report a case of a patient with hypothyroidism due to amiodarone. The short-term administration (1 g/die for 10 days) of potassium perchlorate (KClO4) led to normalization of serum thyroid hormone concentrations and marked reduction of thyrotropic hormone. The reduction of KClO4 (400 mg/die) and
[Neonatal hypothyroidism induced by the ingestion of potassium iodide during pregnancy (author's transl)].
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This concerns a case of an infant born with goiter and hypothyroidism whose mother had taken potassium iodide during pregnancy in the form of a respiratory eupneic syrup. Laboratory tests revealed the etiology of the goiter by demonstrating that it concerned a transitory, acquired hormone production
[Hypothyroidism following intermittent administration of potassium iodide].
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Iodide-induced hypothyroidism with high TSH and low T4 under long term potassium iodide medication was observed in a toddler suffering from chronic wheezy bronchitis -- in spite of intermittent application. The pathophysiology of iodide-induced hypothyroidism and the apparent immaturity of
Subclinical hypothyroidism during potassium iodide therapy for lymphocutaneous sporotrichosis.
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This report describes a high school football player with an unusual ulcerative lesion on the upper leg that was treated initially as pyoderma gangrenosum, but later proved to be due to Sporothrix schenckii. His treatment with potassium iodide solution was complicated by the occurrence of subclinical
Renin and aldosterone in hypothyroidism: relation to excretion of sodium and potassium.
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Changes in plasma renin activity (PRA) and plasma aldosterone (PA) were studied together with the urinary excretion of sodium and potassium in patients with hypothyroidism. The basal levels of PRA and PA were significantly less than those in normal subjects. However, there was no significant
Lymphocutaneous Sporotrichosis Treated with Potassium Iodide with Development of Subclinical Hypothyroidism: Wolff-Chaikoff Effect?
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Sporotrichosis is a subcutaneous mycotic infection caused by Sporothrix schenckii that is acquired by traumatic implantation. The diagnosis is established by demonstration of fungal elements on histopathology and culture. Potassium iodide, azole antifungals, and terbinafine are the treatment options
Nitric oxide is involved in the hypothyroidism with significant morphology changes in female Wistar rats induced by chronic exposure to high water iodine from potassium iodate.
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Epidemiological studies indicated that chronic exposure to high water iodine is associated with primary hypothyroidism (PH) and subclinical hypothyroidism (SCH). However, the mechanism is not well understood. In this study, we explored whether chronic exposure to high water iodine from potassium
Short term administration of potassium perchlorate restores euthyroidism in amiodarone iodine-induced hypothyroidism.
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We studied the effect of potassium perchlorate (KClO4) in patients with hypothyroidism due to amiodarone. The short term administration of KClO4 to six such patients led to prompt restoration of euthyroidism, while the three untreated patients remained hypothyroid for 2-6 months. Since KClO4
Potassium adaptation in hypothyroidism: changes in transport adenosinetriphosphatases.
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To evaluate whether hypothyroidism alters the adaptive responses of renal transport adenosine-triphosphatases (ATPases) to modifications in dietary K content, we examined the activities of Na-K pump and H-K pump in hypothyroid rats under basal conditions and after dietary K changes. Hypothyroidism
[Contribution to a knowledge of heart disease caused by metabolic disorders in infancy; clinical and electrocardiographic aspects of the vitamin deficiency syndromes of early infancy, of hypothyroidism, and of disturbance of potassium equilibrium].
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Your article on the "A-B-C's of heart health" in the October 2008 "Women's Heart Advisor" emphasized the importance of vitamin D for heart health. I am taking Synthroid 88mcg for hypothyroidism, and I recently read in a medical report that vitamin D is known to antagonize thyroid function--that excessive vitamin D supplementation can contribute to a loss of potassium and suppress thyroid function. The article says patients should avoid sources of extra vitamin D if they have a hypothyroid condition. Can you please address this?
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Ulcerative subcutaneous zygomycosis: development of hypothyroidism induced by potassium iodide (Wolff-Chaikoff effect).
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Hypothyroidism and atypical spherocytic hemolytic anemia with high-sodium, low-potassium red cells.
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