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leukocytosis/hypoxia
Veza se sprema u međuspremnik
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False hypoxemia induced by leukocytosis.
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Severe hypoxemia in a patient with massive leukocytosis.
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Evaluation of leukocytosis, bacteremia, and portal vein partial oxygen tension in clinically normal dogs and dogs with portosystemic shunts.
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OBJECTIVE
To assess the relationship of WBC counts and partial oxygen tension in the portal vein and caudal vena cava with portal bacteremia, bacteria in the liver, and postoperative morbidity and mortality in dogs with portosystemic shunts.
METHODS
Prospective study.
METHODS
12 clinically normal
[Spurious hypoxemia due to hyperleukocytosis. Report of one case].
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BACKGROUND
Arterial gasometry is considered the gold standard for establishing a diagnosis of respiratory failure of any etiology. However, there are some circumstances in which it loses specificity, making necessary to consider other tests such as pulse oximetry to adequately determine hypoxemia.
[An autopsy case of systemic lupus erythematosus complicating leukocytosis, amegakaryocytic thrombocytopenia, interstitial pneumonitis, and pleulitis].
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A 52-year-old female was admitted to our hospital in August 1988, for evaluation of purpura and gingival bleeding. Hematologic examination showed mild leukocytosis (12,400/microliter) and severe thrombocytopenia (1,000/microliter). On bone marrow examination, megakaryocyte count was normal and the
Spurious Laboratory Values in Patients With Leukocytosis.
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Physiological derangements such as hypoxemia and hyperkalemia are medical emergencies that warrant prompt interventions to prevent further patient clinical deterioration. However, in patients with myeloproliferative diseases or malignancies that result in extreme leukocytosis, hypoxemia and
Endothelin-1-induced macrophage inflammatory protein-1beta expression in monocytic cells involves hypoxia-inducible factor-1alpha and AP-1 and is negatively regulated by microRNA-195.
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Patients with sickle cell disease (SCD) exhibit a chronic inflammatory state manifested by leukocytosis and increased circulating levels of proinflammatory cytochemokines. Our studies show that placenta growth factor levels are high in SCD, and placental growth factor induces the release of the
PRECORDIAL PAIN, LEUKOCYTOSIS AND BICYTOPENIA IN A TEENAGER WITH SYSTEMIC JUVENILE IDIOPATHIC ARTHRITIS UNDER IMMUNOSUPPRESSIVE THERAPY.
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[Iron metabolism regulatory mechanisms in initial leukocytosis in patients with acute leukemia].
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OBJECTIVE
To study levels of hepsidine (Hp), hypoxia-inducible factor-1alpha (HIF-1alpha), erythropoietin (EP) and ferritin in patients with acute leukemia (AL), effects of protein fractions of homogenate of blastic cells (BC) on regulatory proteins studied.
METHODS
Depending on leukocyte count in
[Intrauterine hypoxia and sudden infant death syndrome].
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Sudden infant death syndrome (SIDS) or crib or cot death are synonyms for the sudden, unexpected and unexplained death of an infant. The incidence of SIDS has been estimated to be from 1-2% to 3%. Protracted intrauterine hypoxia or recurrent hypoxic insults during fetal life undoubtedly influence
Leukopenia, hypoxia, and complement function with different hemodialysis membranes.
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Complement activation during exposure of plasma to cuprophan has been postulated to cause leukopenia and hypoxia in hemodialysis patients. To determine if hypoxia is related to leukopenia and if complement activation leads to a depletion of functional complement components, we dialyzed four patients
Normal arterial oxygen saturation with the ear oximeter in patients with leukemia and leukocytosis.
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Leukemic patients with severe leukocytosis experience a spurious lowering of the arterial oxygen tension if the arterial blood samples are not immediately analyzed. Arterial oxygen tensions were measured in three patients with acute leukemia; actual oxygen saturation was simultaneously measured with
Correlation between leukocytosis and necropsy findings in dogs with immune-mediated hemolytic anemia: 34 cases (1994-1999).
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OBJECTIVE
To determine whether severity of leukocytosis correlates with severity of postmortem lesions in dogs with immune-mediated hemolytic anemia (IMHA).
METHODS
Retrospective study.
METHODS
34 dogs with IMHA that had CBC performed within 48 hours prior to death and complete necropsy
Leukocyte larceny: a cause of spurious hypoxemia.
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Multiple blood specimens with different leukocyte counts from two patients with extreme leukocytosis secondary to leukemia and unexplained hypoxemia were tonometered with a gas of known oxygen concentration and the decay of oxygen tension (PO2) was measured over time. The decay in PO2 in the first 2
Inhaled carbon monoxide reduces leukocytosis in a murine model of sickle cell disease.
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Carbon monoxide (CO) has anti-inflammatory properties. We previously reported that acute treatments with inhaled CO inhibit vascular inflammation and hypoxia-induced vasoocclusion in sickle cell disease mouse models. Therefore, we hypothesized that chronic CO inhalation would decrease vascular
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