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neuroblastoma/edema
Veza se sprema u međuspremnik
Page 1 od 76 rezultati
[Feto-placental anasarca secondary to a neuroblastoma. Prenatal diagnosis].
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The authors describe a very rare case of feto-placental anasarca of non-immunological cause (AFPNI) which was secondary to a fetal neuroblastoma. The diagnosis was suspected before birth thanks to ultrasound. After birth a further ultrasound study as well as an anatomo-pathological study were
A catecholamine-secreting neuroblastoma leading to hydrops fetalis.
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A case of fetal neuroblastoma of the right adrenal gland, with rapid development of hydrops fetalis due to catecholamine-induced cardiomyopathy, is reported. A fetus with a right suprarenal mass detected during ultrasonography at 32 weeks gestation progressively developed into hydrops fetalis by
Hydramnios with hydrops fetalis and disseminated fetal neuroblastoma.
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A white primigravida with acute toxemia and hydramnios delivered a hydropic male infant who died. A neuroblastoma of the right adrenal gland was found with metastic spread to the liver and other organs. The placental capillaries contained tumor cells. Pathogenesis of hydrops, hydramnios, and
Congenital neuroblastoma presenting as hydrops fetalis.
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Metastatic fetal neuroblastoma with non immune fetal hydrops.
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Association of hydrops fetalis with congenital neuroblastoma.
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Fetal neuroblastoma with subsequent fetal supraventricular tachycardia and hydrops.
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13-cis-retinoic acid-associated bone marrow edema in neuroblastoma.
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Fatal Congenital Retroperitoneal Neuroblastoma Diagnosed by Fetal Magnetic Resonance Imaging.
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Congenital extra-adrenal neuroblastoma is a rare condition, which typically has a favorable prognosis. We present a unique case of extra-adrenal retroperitoneal neuroblastoma diagnosed by fetal magnetic resonance imaging, which ultimately leads to fetal hydrops and neonatal death.
Hypertensive retinopathy as the initial presentation of neuroblastoma.
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UNASSIGNED
To describe a case of a patient who presented with hypertensive retinopathy and was found to have neuroblastoma.
UNASSIGNED
Neuroblastoma has three main ocular presentations. As a primary disease, it can present with a paraneoplastic syndrome in the form of opsoclonus, or it can present
Raccoon eyes and the MIBG super scan: scintigraphic signs of neuroblastoma in a case of suspected child abuse.
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The authors report on an infant suspected of having been abused, who presented with periorbital edema and ecchymoses (clinical "raccoon eyes"). The pattern of the nuclear medicine bone scan suggested neuroblastoma rather than trauma. Both the bone scan and the subsequent MIBG scan revealed multiple
Effect of the ostreolysin A/pleurotolysin B pore-forming complex on neuroblastoma cell morphology and intracellular Ca²⁺ activity.
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Ostreolysin A (OlyA) and pleurotolysin B (PlyB), isolated from edible oyster mushrooms, form a cytolytic complex (OlyA/PlyB) in membrane cells that causes respiratory arrest. This study evaluated the mechanisms underlying cytotoxic OlyA/PlyB activity in neuroblastoma NG108-15 cells. Confocal
A model of bacterial translocation in neuroblastoma-bearing mice.
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OBJECTIVE
The aim of this study was to establish a model of bacterial translocation (BT) in neuroblastoma-bearing mice.
METHODS
A suspension of 1 x 10(6) cells of the murine neuroblastoma cell line C1300 was injected subcutaneously into the thighs of 8-week-old female A/J mice, which were then
Nonimmunologic hydrops fetalis: a clinicopathological study of 50 autopsy cases.
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Fifty cases of nonimmunologic hydrops fetalis found in Japanese infants are reported. Nonimmunologic hydrops fetalis is associated with various pathological conditions, twin transfusion syndrome including acardiac monsters, fetal heart diseases, congenital cystic adenomatoid malformation, pulmonary
Kinin generation from exogenous kininogens at the surface of retinoic acid-differentiated human neuroblastoma IMR-32 cells after stimulation with interferon-γ.
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Bradykinin-related peptides, kinins, ubiquitously occur in the nervous system and together with other pro-inflammatory mediators contribute to pathological states of that tissue such as edema and chronic pain. In the current work we characterized the kinin-forming system of neuronal cells obtained
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