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turpentine/moždani udar

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ČlanciKliničkim ispitivanjimaPatenti
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A study on the differential protein profiles in liver cells of heat stress rats with and without turpentine treatment.

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BACKGROUND Heat stress (HS) and related illnesses are a major concern in military, sports, and fire brigadiers. HS results in physiologic responses of increased temperature, heart rate and sweating. In heat stroke, inflammatory response plays an important role and it is evidenced that turpentine (T)

In vivo effects of hyperthermia on the functional and conformational characteristics of antithrombin.

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BACKGROUND High temperatures produce in vitro transitions of antithrombin to its inactive latent and polymeric forms. Accordingly, high body temperatures might contribute in vivo to conformational changes in antithrombin associated with increased thrombotic risk. METHODS We assessed the in vivo

Pre-existing inflammatory state compromises heat tolerance in rats exposed to heat stress.

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This study investigated the roles of endotoxemia and heat-induced tissue damage in the pathology of heat stroke. In groups of eight, male Wistar rats were treated with heat exposure only (HE), or heat exposure with turpentine (T+HE), dexamethasone (D+HE), and turpentine and dexamethasone combined
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