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5 hydroxytryptamine/cefalàlgia
L'enllaç es desa al porta-retalls
Pàgina 1 des de 192 resultats
Platelet substance P and 5-hydroxytryptamine in migraine and tension-type headache.
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Levels of substance P (SP) and 5-hydroxytryptamine (5-HT) in platelets were measured in 25 patients with migraine, 31 patients with tension-type headache (TH) and 27 healthy controls. The mean concentration of SP in platelets was 355.3 pg/10(9) platelets in patients with migraine, 290.8 pg/10(9)
[Tension-type headache: 5-hydroxytryptamine and related substances].
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There are many reports concerning platelet dysfunction and abnormalities of platelet 5-hydroxytryptamine (5-HT) in migrainous patients. In recent years, platelets are considered good models of monoamine neurons. Levels of neurotransmitters in platelets are suggested to reflect those in the nervous
Salivary substance P, 5-hydroxytryptamine, and gamma-aminobutyric acid levels in migraine and tension-type headache.
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Substance P, 5-hydroxytryptamine, and gamma-aminobutyric acid levels in saliva were measured in 55 patients with migraine during headache attacks (15 men and 40 women, average age 37.6 years), 36 patients with migraine in interictal periods (8 men and 28 women, average age 43.9 years), 48 patients
Concentration and uptake of 5-hydroxytryptamine in platelets from cluster headache and migraine patients.
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Concentrations of 5-hydroxytryptamine (5-HT) in platelets were determined in 33 cluster headache patients (17 males) and in 34 migraine patients (16 males) outside attacks. The 5-HT uptake into platelets was measured and the kinetic constants Vmax and Km determined in 26 cluster patients (14 males)
Kinetics of platelet 5-hydroxytryptamine uptake in headache patients.
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Platelet 5-hydroxytryptamine (5-HT) uptake was measured in asymptomatic headache patients attending a specialist migraine clinic, and in hospital staff who did not suffer from regular or severe headache. Current levels of anxiety and depression were assessed in all subjects using the Hospital
5-Hydroxytryptamine receptor agonists for the abortive treatment of vascular headaches block mast cell, endothelial and platelet activation within the rat dura mater after trigeminal stimulation.
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Antidromic stimulation of small caliber trigeminal axons causes neurogenic inflammation in the dura mater and tongue as evidenced by marked increases in mast cell activation, protein extravasation, as well as in the numbers of endothelial cytoplasmic vesicles, endothelial microvilli and platelet
5-Hydroxytryptamine levels and platelet aggregation responses in subjects with acute migraine headache.
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Blood 5-hydroxytryptamine levels were reduced during migraine attacks in patients who had not taken any drugs but the aggregation responses to 5-hydroxytryptamine of the blood platelets from these patients were similar to responses of platelets from migrainous subjects between attacks. This confirms
Headaches, depression and 5-hydroxytryptamine.
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5-hydroxytryptamine supersensitivity as a new theory of headache and central pain: a clinical pharmacological approach with p-chlorophenylalanine.
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5-Hydroxytryptamine in platelets of patients with muscle contraction headache.
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Headache as possible expression of deficiency of brain 5-hydroxytryptamine (central denervation supersensitivity).
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Blood platelet 5-hydroxytryptamine accumulation and migraine.
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The active uptake of 5-hydroxytryptamine (5-HT) by platelets from migraine patients was measured and the results compared with 5-HT uptake of platelets from normal subjects. No significant differences in platelet 5-HT uptake were found between migraine patients and control subjects. A significant
5-Hydroxytryptamine (5-HT) and the initiation of migraine: new perspectives.
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The hypothesis that 5-hydroxytryptamine (5-HT) acting through 5-HT2C receptors is a key factor in the initiation of migraine has been re-evaluated in the light of recent basic and clinical scientific developments. The key findings are that nitric oxide is an important trigger for migraine, that
Platelet activation in muscle contraction headache and migraine.
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In migraine, the role of platelets is regarded as an important factor. We investigated plasma beta-thromboglobulin (BTG), platelet factor 4 (PF4), and 5-hydroxytryptamine (5-HT) in migraine patients and muscle contraction headache (MCH) patients during headache-free periods. The mean values of the
Increased 11-dehydrothromboxane B2 in migraine: platelet hyperfunction in patients with migraine during headache-free period.
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Several disturbances in platelet function have been reported in migraine and tension-type headache (TH). The plasma 11-dehydrothromboxane B2 (11-dTXB2) is free from artifactual increase during blood sampling, and it can be a reliable indicator of thromboxane A2 (TXA2) production in vivo. TXA2 is a
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