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diabetic ketoacidosis/potassium
L'enllaç es desa al porta-retalls
Pàgina 1 des de 265 resultats
Effect of Potassium Infusions on Serum Levels in Children during Treatment of Diabetic Ketoacidosis.
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A retrospective study was done to determine the effect of potassium (K + ) infusions on serum levels in children admitted to the pediatric intensive care unit (PICU) with diabetic ketoacidosis (DKA). Eighty-two percent of 92 cases studied received 40 mEq/L K + infusion over the
Does potassium concentration measured on blood gas analysis agree with serum potassium in patients with diabetic ketoacidosis?
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OBJECTIVE
The aims of this study were to define the maximum clinically acceptable difference between potassium concentrations on different samples and to determine the degree of agreement between potassium concentration measured on blood gas analysis and serum for patients with diabetic ketoacidosis
Hypocalcemia, hypomagnesemia, and transient hypoparathyroidism during therapy with potassium phosphate in diabetic ketoacidosis.
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The effects of intravenous administration of potassium phosphate in the treatment of diabetic ketoacidosis were studied in nine children, ages 9 9/12 to 17 10/12 yr. During phosphate infusion (20--40 meq/L of fluid), all children maintained normal serum concentrations of phosphorus. Transient
Diabetic ketoacidosis: difference between potassium determined by blood gas analysis versus plasma measurement.
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OBJECTIVE
To evaluate the accuracy of potassium concentrations measured by blood gas analysis (PBG) compared with laboratory serum potassium (LSP), in the initial care of patients with diabetic ketoacidosis (DKA).
METHODS
Fifty three patients with diabetes mellitus were evaluated in a retrospective
Immediate plasma potassium levels in treating diabetic ketoacidosis.
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A patient presented with diabetic ketoacidosis and severe hypokalemia (less than 2.0 mmol/L [less than 2.0 mEq/L]). The availability of immediate plasma potassium levels using a blood gas analyzer (Radiometer) prevented the use of potentially hazardous therapy. Potassium levels should be determined
Nonadherence to potassium replacement protocol leads to prolonged management of diabetic ketoacidosis.
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Introduction Diabetic ketoacidosis is a life-threatening condition that requires prompt management. Objectives We aimed to assess the impact of adherence to potassium replacement protocol according to the guidelines of Diabetes Poland on the duration of diabetic ketoacidosis (DKA) treatment.
[Treatment of extreme hyperkalemia caused by diabetic ketoacidosis, potassium-sparing diuretics and potassium substitutes].
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Grave hyperkalemic is a serious metabolic disorder. Its treatment fell into the fields of urgent medicine because of the risk of malignant cardiac arrhythmias that can be fatal for the patient. The article deals with the treatment of a 49-year-old female patient with decompensated liver cirrhosis
Correlation of acidosis-adjusted potassium level and cardiovascular outcomes in diabetic ketoacidosis: a systematic review.
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Determinants of plasma potassium levels in diabetic ketoacidosis.
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The classic proposal of intracellular K+ for extracellular H+ exchange as responsible for the hyperkalemia of diabetic ketoacidosis (DKA) has been questioned because experimentally induced organic anion acidosis fails to produce hyperkalemia. It has been suggested, instead, that the elevated serum
[The value of potassium phosphate supplements in the treatment of diabetic ketoacidosis. Effects upon 2.3-diphosphoglycerate and tissue oxygenation (author's transl)].
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Amongst 8 diabetics with severe ketoacidosis and treated identically, 4 received intravenous supplements of potassium phosphate (25 mmol PO4/I) for 24 hours. This resulted in blood 2.3-diphosphoglycerate and in vivo P50 levels which were invariably higher than in the control subjects and a fall in
A paper which changed clinical practice (slowly). Jacob Holler on potassium deficiency in diabetic acidosis (1946).
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It is often said that the introduction of insulin into clinical medicine made a 'dramatic' difference to the mortality resulting from diabetic coma. This is true in the sense that before 1922 it was almost uniformly fatal, but until the 1950s the mortality in many large hospitals was as high as
The mineral and water composition of human red blood cells in diabetic acidosis and during recovery with special reference to changes in potassium content.
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Paralysis due to reduced serum potassium concentration during treatment of diabetic acidosis; report of case treated with 33 grams of potassium chloride intravenously.
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Potassium deficiency associated with diabetic acidosis.
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Potassium and phosphate in diabetic acidosis.
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