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dystonia/canya de sucre
L'enllaç es desa al porta-retalls
4 resultats
Delayed dystonia with striatal CT lucencies induced by a mycotoxin (3-nitropropionic acid).
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We describe a clinical syndrome of delayed dystonia in children subsequent to initial gastrointestinal symptoms and acute noninflammatory encephalopathy. The syndrome was caused by the ingestion of mildewed sugarcane containing the Arthrinium-produced mycotoxin, 3-nitropropionic acid (3-NPA). In the
Moldy sugarcane poisoning--a case report with a brief review.
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A five-year-old girl developed an acute encephalopathy after eating a piece of moldy sugarcane. Delayed symptomatic dystonia was the main effect; cranial CT scans revealed bilateral lenticular lucencies. This case is typical of moldy sugarcane poisoning cases previously reported only in China.
Neurologic diseases associated with use of plant components with toxic potential.
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Epidemics of neurotoxic disease in developing regions of the world are often associated with dietary dependence on plant components with inherent toxic potential or which have spoiled and become contaminated with mycotoxins. Diseases triggered by plant toxins include lathyrism and cassavism, types
Consistent striatal damage in rats induced by 3-nitropropionic acid and cultures of arthrinium fungus.
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Arthrinium fungi were cultivated from the samples of mildewed sugarcane which caused acute encephalopathy and delayed dystonia in children. The Arthrinium cultures (AC) contained 5 mg/ml 3-nitropropionic acid (3-NPA) after being inactivated and concentrated. A neuropathological study was carried out
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