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ganglion cysts/febre

L'enllaç es desa al porta-retalls
Pàgina 1 des de 88 resultats

Hyperthermia and hypoxia increase tolerance of retinal ganglion cells to anoxia and excitotoxicity.

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OBJECTIVE Knowledge of the mechanisms by which retinal ganglion cells are damaged may provide information required to develop novel treatments for diseases that cause retinal ganglion cell death. The authors investigated whether the expression of the 72-kDa heat shock protein in cultured rat retinal

Peripapillary Retinal Nerve Fiber Layer and Ganglion Cell-Inner Plexiform Layer Thickness in Children with Familial Mediterranean Fever.

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OBJECTIVE To evaluate the thickness of the peripapillary retinal nerve fiber layer (RNFL) and retinal ganglion cell-inner plexiform layer (GCIPL) in children with familial Mediterranean fever (FMF). METHODS The study included 39 FMF patients and 36 healthy controls. After detailed ocular

Peripapillary retinal nerve fiber layer and ganglion cell-inner plexiform layer thickness in adult-onset familial Mediterranean fever.

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OBJECTIVE The purpose of the present study was to evaluate the thickness of the peripapillary retinal nerve fiber layer (RNFL) and retinal ganglion cell-inner plexiform layer (GCIPL) in adult-onset familial Mediterranean fever (FMF). METHODS Forty two adult-onset FMF patients and forty two healthy

Alterations in autonomic ganglion cells in fever, dehydration, and inanition.

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Meningitis after invasive treatment of the trigeminal ganglion: two case reports and a review of the literature.

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OBJECTIVE The objective of this case is to report the rare complication of a meningitis after an invasive treatment of the trigeminal ganglion. METHODS In this case report, two cases of meningitis due to oral streptococci are reported and a review of literature is presented. CONCLUSIONS This

Replication, tissue tropisms and transmission of yellow fever virus in Aedes albopictus.

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Experimental studies undertaken to ascertain the dynamics of yellow fever virus replication in an introduced strain (Houston) of the Asian mosquito, Aedes albopictus (Skuse), indicate that this species is an efficient vector of yellow fever virus. Replication of virus in Ae. albopictus could be

Retinal and Choroidal Thickness in Adult Patients with Familial Mediterranean Fever.

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We aimed to evaluate changes in retinal, choroidal, ganglion cell complex (GCC) and retinal nerve fiber layer (RNFL) thicknesses in genetically diagnosed adult patients with familial Mediterranean fever (FMF). A total of 50 eyes of 50 genetically diagnosed patients with FMF and 50 eyes of controls

Effects of Recovery Time during Magnetic Nanofluid Hyperthermia on the Induction Behavior and Efficiency of Heat Shock Proteins 72.

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In this study, we investigated the effects of recovery time during magnetic nanofluid hyperthermia (MNFH) on the cell death rate and the heat shock proteins 72 (HSP72) induction behavior in retinal ganglion cells (RGCs-5) to provide a possible solution for highly efficient ocular neuroprotection.

Induction of a heat shock gene (hsp70) in rabbit retinal ganglion cells detected by in situ hybridization with plastic-embedded tissue.

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Elevation of body temperature by 2-3 degrees C induces a 2.7 kilobase hsp70 mRNA species in the rabbit retina within 1 hr. In situ hybridization with thin sections derived from plastic-embedded tissue permitted a higher level of resolution of retinal cell types compared to procedures which involved

Traumatic intraosseous ganglion. A case report.

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The rapid development of an intraosseous ganglion following an intraarticular fracture of the distal radius in a patient suffering from familial Mediterranean fever is presented. The case supports the view that trauma may be an etiological factor in the genesis of intraosseous ganglia.
The induction of the heat shock protein, HSP72, was studied in the cochlea of guinea pigs and rats subjected to a hyperthermic stress. Analyses were done by immunoblotting and immunocytochemistry at 6 and 12 h after heat shock, using a commercially available monoclonal antibody (Amersham), and by in
Objective: The exact mechanism of phonophobia induced by subarachnoid hemorrhage (SAH) has not been understood well. This subject was investigated. Material and methods: This study was conducted on 25 rabbits. They divided into three groups: Five as control, five as SHAM, 20 as SAH
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