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hyperammonemia/seizures
L'enllaç es desa al porta-retalls
Pàgina 1 des de 262 resultats
Hyperammonemia as a Marker of Subclinical Seizures After Traumatic Cardiac Arrest.
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This report details the presence of hyperammonemia in a patient who sustained cardiac arrest after a traumatic amputation. Serum ammonia levels may rise due to numerous etiologies; however, few reports detail its usefulness in diagnosing subclinical seizures. In this case, we successfully utilized
Transient hyperammonemia associated with postictal state in generalized convulsion.
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Previous studies revealed that transient hyperammonemia was noted after generalized convulsion. This study was undertaken to analyze the association between postictal state and serum ammonia levels. Adult patients presenting to the emergency department with seizures were included. Serum ammonia and
Hyperammonemia is associated with generalized convulsion.
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OBJECTIVE
The possibility that hyperammonemia may be associated with generalized convulsion (GC) was retrospectively investigated.
METHODS
Subjects comprised 17 patients with GC who were transported to our department and underwent analysis of serum biochemistry, including ammonia, since October
Hyperammonemia in idiopathic epileptic seizure.
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OBJECTIVE
It is known that patients with convulsion often present hyperammonemia. The elevation of ammonia levels in convulsion is considered to occur along with extensive muscle contractions, but the details remain unclear. In emergency pathologies, such as cardiopulmonary arrest or hemorrhagic
Hyperinsulinism-hyperammonemia Syndrome in an Infant with Seizures.
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Hyperinsulinism-hyperammonemia syndrome (HI/HA) is the second most common form of persistent hyperinsulinemic hypoglycemia of infancy (PHHI). The main clinical characteristics of HI/HA syndrome are repeated episodes of symptomatic hypoglycemia, but not usually severe. Consequently, children with
Seizures and diagnostic difficulties in hyperinsulinism-hyperammonemia syndrome.
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Hyperinsulinism/hyperammonemia (HI/HA) syndrome is a rare disorder presented with recurrent hypoglycemia and elevated serum ammonia, which may lead to development delays, permanent neurologic damages, if it remains underdiagnosed. It is caused by activating mutations in the GLUD1 gene which encodes
Inhibition of acute hyperammonemia-induced convulsions by systemically administered gamma aminobutyric acid in rats.
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The present study has investigated the effects of intraperitoneally administered gamma aminobutyric acid (GABA) on ammonium chloride-induced hyperammonemia and convulsions in rats. Systemically administered GABA did not alter the concentration of GABA in the brain of control as well as
Intractable absence seizures in hyperinsulinism-hyperammonemia syndrome.
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A girl with intractable absence seizures and facial myoclonia at age 7 years was eventually diagnosed with hyperinsulinism-hyperammonemia syndrome because of hypoglycemia, hyperinsulinism, hyperammonemia, and the results of an oral l-leucine loading test. Her seizures occurred even during periods of
Transient hyperammonemia in seizures: a prospective study.
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OBJECTIVE
To investigate the incidence and duration of transient hyperammonemia in seizures and to verify the significant confounders related to transient hyperammonemia in seizures.
METHODS
One hundred twenty-one noncirrhotic adult patients with seizures admitted to the emergency department were
Postictal transient hyperammonemia as an indicator of seizure disorder.
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OBJECTIVE
Transient hyperammonemia can occur after episodes of seizure. To verify that transient hyperammonemia is a unique finding in seizure patients, we compared the change in blood ammonia concentration during the postictal period with that of other patients who suffered consciousness
Familial hyperinsulinism-hyperammonemia syndrome in a family with seizures: case report.
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Hyperinsulinism-hyperammonemia (HI/HA) syndrome is the second most frequent cause of congenital hyperinsulinism (CHI) and it is characterized by recurrent symptomatic hypoglycemia and persistent hyperammonemia. We describe the familial case of a 2-year-old child and her 32-year-old mother who,
Risk Factors Associated With Hyperammonemia Following Unprovoked Convulsive Seizures
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This report describes a unique case of recurrent transient hyperammonemia (THA) following a first-time occurrence of generalized tonic-clonic seizure in a young adult, who went on to develop post-stroke epilepsy. Although this phenomenon has been described in recent literature, we report not only
Generalized convulsion resulted in hyperammonemia during treatment with tranexamic acid for hemoptysis.
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BACKGROUND
Tranexamic acid is commonly used to treat various kinds of bleeding disorders. It has been shown to cause severe convulsions in animal experiments.
OBJECTIVE
We report a patient who experienced a single convulsive seizure that resulted in transient hyperammonemia during treatment with
Valproate-induced hyperammonemia and seizures: perioperative concerns.
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Hyperornithinemia, hyperammonemia, and homocitrullinuria. A new disorder of amino acid metabolism associated with myoclonic seizures and mental retardation.
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