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hypertrichosis/edema
L'enllaç es desa al porta-retalls
Pàgina 1 des de 49 resultats
Polyneuropathy, skin hyperpigmentation, edema, and hypertrichosis in localized osteosclerotic myeloma.
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1 61-year-old man had osteosclerotic myeloma that was localized in the eleventh thoracic vertebral body and associated with sensorimotor polyneuropathy, skin hyperipigmentation, edema, hypertrichosis, gynecomastia, and white nails. Cases of osteosclerotic myeloma with and without polyneuropathy in
[Marked endocrine abnormalities in polyneuritis with skin hyperpigmentation, hypertrichosis, edema and increased lambda type immunoglobulin A: a case report].
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A syndrome which is known as plasma cell dyscrasia with polyneuropathy and various endocrine manifestations or plasma cell dyscrasia with polyneuropathy, organomegaly, endocrinopathy, abnormal M protein and skin changes is very interesting because this syndrome has miscellaneous manifestations such
Use of SEPs to localize degeneration in a rare polyneuropathy: studies on polyneuropathy associated with pigmentation, hypertrichosis, edema, and plasma cell dyscrasia.
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Peripheral and cental nerve conduction were studied in five patients who had polyneuropathy associated with pigmentation, hypertrichosis, edema, and plasma cell dyscrasia by using the short-latency somatosensory evoked potential (SEP) to electrical stimulation of the median nerve at the wrist. The
[Membranoproliferative glomerulonephritis associated with "chronic polyneuropathy with skin hyperpigmentation, hypertrichosis, edema and abnormal immunoglobulins" (author's transl)].
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[A necropsy case of myeloma (IgA, lambda type) with skin pigmentation, hypertrichosis, edema and polyneuropathy. Congo-red positive deposition in the dermis and peripheral nerve (author's transl)].
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[The pathology of the peripheral nervous system in two autopsy cases of osteosclerotic myeloma associated with polyneuropathy, skin hyperpigmentation, hypertrichosis and edema (author's transl)].
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[Polyneuropathy with skin hyperpigmentation, hypertrichosis, edema and choked disc--with special reference to biochemical, endocrinological, immunological and neurohistological studies].
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[Skin pigmentation deposits, hypertrichosis of the forearms and thighs, arthralgia of the knee, edema of the posterior of the legs, bloating, muscular atrophy and general weakness (bone radiography)--craniotomy for the treatment of arachnoiditis of the optic chiasm: (multiple myeloma)].
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Transaldolase deficiency: a new cause of hydrops fetalis and neonatal multi-organ disease.
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Transaldolase (TALDO) deficiency is a newly recognized metabolic disease, which has been reported so far in 2 patients presenting with liver failure and cirrhosis. We report a new sibship of 4 infants born to the same consanguineous parents; all presented at birth or in the antenatal period with
Paradoxical effects of hair removal systems: a review.
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Although a variety of lasers have proven to be clinically effective for long-term hair removal, the use of these lasers has also been associated with undesirable side effects, such as hyper- and hypopigmentation, crusting, erythema, and edema. One notable side effect that seems to be underreported
[A case of Crow-Fukase syndrome showing improvement following excision and irradiation of bone lesions].
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A 57-year-old woman suffering from pleural and pericardial effusion, pulmonary hypertention, lymphadenopathy, hepatosplenomegaly, edema, hypertrichosis, small hemangioma and polyneuropathy was diagnosed as Crow-Fukase syndrome. Osteoctomy of the left second rib and irradiation of this rib and the
Efficacy and tolerance of the new long-acting vasodilator RO 12-4713 in hypertensive patients not managed on beta blockers and diuretics.
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The new vasodilator Roche 12-4713, an oxodiazolo-pyrimidine with long-acting direct arteriolar dilating properties, was tested in 13 patients (ages 33-66 years, mean 53 +/- 7 SD) previously uncontrolled on a beta blocker-diuretic combination. The addition of Ro 12-4713 (30-180 mg per day) for 134
[A case of Crow-Fukase syndrome with increased serum interleukin-6].
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We experienced a 47-year-old Japanese female with polyneuropathy, edema, hypertrichosis, hyperpigmentation, and white nail, which were diagnostic as having Crow-Fukase syndrome. Laboratory and radiological evaluation showed neither plasma cell dyscrasia nor monoclonal gammopathy. Increased factor
Dermatologic manifestations of acromegaly: A case in point and a focused review.
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Acromegaly is a systemic syndrome caused by overproduction of growth hormone. Cutaneous, endocrine, cardiovascular, skeletal, and respiratory systems are affected. Cutaneous changes in acromegaly relate to overexpression of growth hormone and insulin-like growth factor 1 on skin cells and adnexa.
Skin manifestations in acromegaly.
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Cutaneous changes in acromegaly result from excess GH and IGF-1 action on skin cells and adnexae. Skin puffiness due to dermal glycosaminoglycan accumulation and edema are most prominent in the face, hands and feet. Oily skin with large pores, hypertrichosis, and excessive sweating are common
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