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lactic acid/hemorràgia
L'enllaç es desa al porta-retalls
Pàgina 1 des de 221 resultats
The effects of dihydroxyphenyl lactic acid on alleviating blood-brain barrier injury following subarachnoid hemorrhage in rats.
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The influence of different resuscitation solution on lactic acid accumulation after hemorrhagic shock: a network meta-analysis.
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Study on preparation of fibrinogen-loaded poly (L-lactic) acid nano-fabrics and its haemostatic performance in swine traumatic haemorrhage models.
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Haemorrhage is the major cause of death in civilian trauma and the leading cause of preventable death in military trauma. It is very important to develop a haemostatic material with definite haemostatic effects. In this study, a nano-fabric membrane containing fibrinogen (Fbg) (2.5%, w/v) was
Lactic Acid Is an Independent Predictor of Mortality and Improves the Predictive Value of Existing Risk Scores in Patients Presenting With Acute Gastrointestinal Bleeding.
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Serum Lactic Acid Following Aneurysmal Subarachnoid Hemorrhage Is a Marker of Disease Severity but Is Not Associated With Hospital Outcomes.
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Background: Following aneurysmal subarachnoid hemorrhage, peripherally-drawn lactic acid has been associated with poor outcomes; however, its clinical significance is unknown. We investigated admission factors and patient outcomes associated with serum lactic acid in this population. Methods: This
Diaspirin cross-linked hemoglobin infusion did not influence base deficit and lactic acid levels in two clinical trials of traumatic hemorrhagic shock patient resuscitation.
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BACKGROUND
Diaspirin cross-linked hemoglobin (DCLHb) has demonstrated a pressor effect that could adversely affect traumatic hemorrhagic shock patients through diminished perfusion to vital organs, causing base deficit (BD) and lactate abnormalities.
METHODS
Data from two parallel, multicenter
[Research on pathogenic mechanisms of hemorrhages due to induced venous stasis. 3. Variations of O2 desaturation and blood lactic acid after venous stasis in relation to the positivity of the tourniquet test].
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[Experimental hemorrhagic fever (Junin virus). Blood sugar, lactic acid in the blood, serum glycolysis and osmotic fragility of erythrocytes].
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[Lactic acid as a prognostic marker in obstetric postpartum hemorrhage].
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Blood lactic acid levels in irreversible hemorrhagic and lethal endotoxin shock.
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[Protective effects of Salviae miltiorrhizae and anisodamine (654-2) against early lung injury in acute hemorrhagic necrotizing pancreatitis in the dog].
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Experimental study had been conducted on 18 healthy adult mongral dogs of both sex, weighting from 7.5-11 kg, randomly divided into four groups. Experimental models of acute hemorrhagic necrotizing pancreatitis (AHNP) were established by retrograde injecting 1 ml/kg of sodium taurocholate directly
[Effects of Astragalus membranaceus injection on nitric oxide and endothelin concentration of intestinal mucosa after hemorrhage shock-reperfusion in rats].
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OBJECTIVE
To observe the effects of Stragalus membranaceus injection on nitric oxide and endothelin levels of intestinal mucosa in reperfusion injury after hemorrhage shock.
METHODS
32 SD rats were randomly divided into four groups: normal group, model group, low dosage group, (treated with
[Acute, hemorrhagic, overgrowing vaginal inflammation caused by Streptococcus agalacitae--case study].
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The aim of this study was to discuss a case of an acute, hemorrhagic, overgrowing vaginal inflammation caused by Streptococcus agalacitae. Intravaginal globules based on methylcellulose and gelatin, containing a lactic acid and chitosan complex in mole relation 1:1 to 4:1 and, in succesion, an
Metabolic and hemodynamic effects of CO2 pneumoperitoneum in a controlled hemorrhage model.
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BACKGROUND
Intracavity infusion of fibrin sealant-based agents, as a novel modality to control internal bleeding, is associated with an increase of pneumoperitoneum (PP) pressure. The safe limit of such increase has not been well defined in hypovolemic subjects. The purpose of this study was to
Cerebral lactic acid delivery and uptake during and after ischemia in the piglet.
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To determine whether lactic acid is a cerebral substrate during and after partial ischemia in piglets, cerebral blood flow and arteriovenous differences of O2 and substrates were measured during control, after hemorrhagic hypotension, and 10 and 90 minutes after reperfusion with blood. During and
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