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lysophosphatidylcholine/hypoxia
L'enllaç es desa al porta-retalls
Pàgina 1 des de 32 resultats
The role of free fatty acids in hypoxia-induced injury to renal proximal tubule cells.
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Phospholipase activation with resulting phospholipid breakdown and lipid byproduct accumulation may play a critical role in hypoxic cell injury. To explore this role, mildly hypoxic rabbit renal proximal tubules (PT) in suspension were treated in vitro with exogenous phospholipase A2 (PLA2). This
Myelin lipids of the rat brain in experimental hypoxia.
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The pattern of cerebral myelin lipids was studied in Wistar rats exposed to mild hypoxia (7% of oxygen in a respiratory mixture) for 30 min. The most pronounced change involved a marked increase in cholesterol esters and lysophosphatidylcholine content in the myelin fraction. The results obtained
Lysophosphatidylcholine accumulation in cardiomyocytes requires thrombin activation of Ca2+-independent PLA2.
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Lysophosphatidylcholine (LPC) accumulates during ischemia or following thrombin stimulation of cardiac myocytes. We determined whether LPC accumulation reflects increased LPC production via phospholipase A2 (PLA2) activation, inhibition of LPC catabolism, or a combination of both.
[State of pulmonary surfactant and ultrastructure of the aerohematic barrier in acute hypoxia].
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Experiments on 8 dogs with acute respiratory hypoxia that lasted 1.5-2 hours were made to study ultrastructure of the aerohematic barrier, lung surface activity and the composition of individual phospholipid fractions in bronchoalveolar washings. The studies were performed with the use of
Hypoxia-inducible factor 2α plays a critical role in the formation of alveoli and surfactant.
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Alveolarization of the developing lung is an important step toward the switch from intrauterine life to breathing oxygen-rich air after birth. The distal airways structurally change to minimize the gas exchange path, and Type II pneumocytes increase the production of surfactants, which are required
Lysophospholipase and the metabolism of lysophosphatidylcholine in isolated bovine rod outer segments.
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Incubation of bovine rod outer segments (ROS) with radiolabeled palmitic acid (16:0) and lysophosphatidylcholines (lysoPC) radiolabeled in either the fatty acid or the choline group indicated the presence of a lysophospholipase activity that is unaffected by Ca2+. In the presence of ATP, Mg2+ and
Lysophosphatidylcholine and Cellular Potassium Loss in Isolated Rabbit Ventricle.
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BACKGROUND: Lysophospholipids such as lysophosphatidylcholine (LPC) have many direct electrophysiological effects on cardiac muscle and have been implicated as a cause of lethal ventricular arrhythmias during acute myocardial ischemia. Because extracellular K(+) accumulation is also a key
Possible origins of PAF-acether and lyso-PAF-acether in rat lung alveoli secondary to hypoxia.
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After 4 h hypoxia, platelet activating factor (PAF-acether or 1-O-alkyl-2-acetyl-sn-glycero-3-phosphocholine) and its deacetylated derivative, lyso-PAF-acether, accumulate in rat lung surfactant, the latter in a 1000-fold excess (Prévost, M.C., Cariven, C., Simon, M.F., Chap, H. and Douste-Blazy, L.
Effect of hypoxia on phospholipid metabolism in porcine pulmonary artery endothelial cells.
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The effect of exposure of porcine pulmonary artery endothelial cells to hypoxic (0% O2) and normoxic (20% O2) conditions for 24 and 48 h on phospholipid metabolism was studied. Sonicates prepared from endothelial cells that were exposed to 24 h of hypoxia showed significant increases in
Adipocyte-derived Lysophosphatidylcholine Activates Adipocyte and Adipose Tissue Macrophage Nod-Like Receptor Protein 3 Inflammasomes Mediating Homocysteine-Induced Insulin Resistance.
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The adipose Nod-like receptor protein 3 (NLRP3) inflammasome senses danger-associated molecular patterns (DAMPs) and initiates insulin resistance, but the mechanisms of adipose inflammasome activation remains elusive. In this study, Homocysteine (Hcy) is revealed to be a DAMP that activates
Hypoxia-mediated impaired erythrocyte Lands' Cycle is pathogenic for sickle cell disease.
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Although Lands' cycle was discovered in 1958, its function and cellular regulation in membrane homeostasis under physiological and pathological conditions remain largely unknown. Nonbiased high throughput metabolomic profiling revealed that Lands' cycle was impaired leading to significantly elevated
Changes in enzyme activities of glycerolipid metabolism of guinea-pig cerebral hemispheres during experimental hypoxia.
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Hypoxic treatment causes changes of some enzymatic activities involved in the glycerolipid metabolism in subcellular fractions of guinea pig cerebral hemispheres. The activity of lysophosphatidylcholine acyltransferase, choline phosphotransferase, glycerol-3-phosphate acyltransferase(s), as well as
Deletion of the metabolic transcriptional coactivator PGC1β induces cardiac arrhythmia.
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OBJECTIVE
Peroxisome proliferator-activated receptor-γ coactivators PGC1α and PGC1β modulate mitochondrial biogenesis and energy homeostasis. The function of these transcriptional coactivators is impaired in obesity, insulin resistance, and type 2 diabetes. We searched for transcriptomic, lipidomic,
Electrophysiologic effects of a short-chain acyl carnitine, L-propionylcarnitine, on isolated canine Purkinje fibers.
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The effects of L-propionylcarnitine (PC), a derivative of L-carnitine, on action potentials of canine Purkinje fibers in vitro, were studied under acidic conditions (pH 6.9), using conventional microelectrode techniques. The concentrations of 10(-5) M to 3 X 10(-3) M had no significant effect on
Amphipathic lipid metabolites and their relation to arrhythmogenesis in the ischemic heart.
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Myocardial ischemia is associated with profound electrophysiologic derangements which occur within minutes and are rapidly reversible with reperfusion, suggesting that subtle and reversible biochemical alterations within or near the sarcolemma contribute. Our efforts have concentrated on two
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