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mitral valve insufficiency/edema
L'enllaç es desa al porta-retalls
Pàgina 1 des de 299 resultats
The role of ischemic mitral regurgitation in the pathogenesis of acute pulmonary edema.
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BACKGROUND
Acute mitral regurgitation may cause pulmonary edema, but the pathogenetic role of chronic ischemic mitral regurgitation, a dynamic condition, has not yet been characterized.
METHODS
We prospectively studied 28 patients (mean [+/-SD] age, 65+/-11 years) with acute pulmonary edema and left
Lobar pulmonary edema due to mitral regurgitation: diagnosis by echocardiography.
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One etiology of unilateral lobar pulmonary edema is mitral regurgitation. Echocardiography is able to demonstrate the retrograde flow of blood into the pulmonary veins and allows timely diagnosis and treatment planning. Correction of mitral regurgitation is followed by resolution of the radiographic
Radiographic features of cardiogenic pulmonary edema in dogs with mitral regurgitation: 61 cases (1998-2007).
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OBJECTIVE
To evaluate radiographic distribution of pulmonary edema (PE) in dogs with mitral regurgitation (MR) and investigate the association between location of radiographic findings and direction of the mitral regurgitant jet (MRJ).
METHODS
Retrospective case series.
METHODS
61 dogs with
Intermittent mitral regurgitation and pulmonary edema after aortic valve replacement.
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We report intermittent mitral valve regurgitation with 17 acute pulmonary edemas over a 16-month period after aortic valve replacement due to combined aortic valve disease in a 51-year-old man. The mechanism of mitral regurgitation was explained by the relatively large size of the prosthetic valve
[Traumatic mitral regurgitation with acute pulmonary edema].
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A case of a successful surgical treatment for traumatic mitral valve regurgitation is reported. A 44-year-old, small-statured female with cretinism had a traffic accident. Eleven days after the accident, she was admitted to our hospital with severe respiratory distress syndrome by acute pulmonary
[Unilateral pulmonary edema in two patients with mitral regurgitation].
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METHODS
Two patients were admitted with dyspnoea, the first after an infection, the second in association with an ischemic cardiomyopathy.
METHODS
Chest radiography displayed an interstitial infiltrate in the right upper lobe in the first patient and in the right lower lobe in the second one. In
Clinical images. Pulmonary edema without an obvious cause: handgrip-induced large dynamic changes in ischemic mitral regurgitation in the cath lab.
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We report a case of pulmonary edema experienced in a patient with severe left ventricular dysfunction without an obvious cause. Thus, we tested the hypothesis of a dynamic increase in the severity of (ischemic) mitral regurgitation. Right- and left-heart cardiac catheterization was performed before
Unilateral cardiogenic pulmonary edema associated with acute mitral regurgitation.
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Two cases of cardiogenic unilateral pulmonary edema are reported. Both patients presented to the emergency department with dyspnea, and chest radiography revealed unilateral infiltration, which mimics pulmonary disease. However, the patients were diagnosed with cardiogenic pulmonary edema, because
Aortic stenosis and severe mitral regurgitation in the fetus resulting in giant left atrium and hydrops: pathophysiology, outcomes, and preliminary experience with pre-natal cardiac intervention.
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OBJECTIVE
The objective of this article is to review anatomic, physiologic, and clinical features of fetuses and neonates with severe mitral regurgitation (MR) in conjunction with aortic stenosis (AS) and left ventricular (LV) and left atrial (LA) dilation and to present preliminary results of
Prevalence of unsuspected mitral regurgitation and left ventricular diastolic dysfunction in patients with coronary artery disease and acute pulmonary edema associated with normal or depressed left ventricular systolic function.
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To define the prevalence and role of left ventricular (LV) systolic dysfunction, LV diastolic dysfunction and mitral regurgitation (MR) in patients with acute pulmonary edema, 40 patients with coronary artery disease and acute pulmonary edema were prospectively evaluated within 36 hours of
[Regional lung edema in acute mitral valve insufficiency following chordae tendineae rupture with prolapse of the posterior mitral valve leaflet].
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An unilateral or predominantly lobar pulmonary edema is an unusual clinical or radiological finding, often misdiagnosed as one of the more common causes of focal lung disease. We report 2 cases of a regional pulmonary edema caused by the acute onset of a severe mitral insufficiency after the rupture
[Localized pulmonary edema in the right upper lobe--an important differential diagnostic hint for acute mitral insufficiency].
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Differential diagnosis of unilateral alveolar pulmonary infiltration includes various possibilities. Acutely developing mitral insufficiency, often without any prior cardiac symptoms, may be the cause of pulmonary edema localized exclusively in the right upper lobe. This unusual and often
Papillary Muscle Dysfunction Due to Coronary Slow-Flow Phenomenon Presenting with Acute Mitral Regurgitation and Unilateral Pulmonary Edema.
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Cardiogenic pulmonary edema usually presents with characteristic clinical features and bilateral infiltrates on the chest radiograph. Rarely, pulmonary edema may manifest unilaterally, leading to a mistaken diagnosis of a primary lung pathology. We present a 30-year-old man who developed acute
Pulmonary edema localized in the right upper lobe accompanying mitral regurgitation.
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Focal patterns of pulmonary edema are confusing and often mistaken for the more common causes of focal lung disease, pneumonia, infarction, or aspiration. The authors report four cases of right upper lobe edema secondary to mitral regurgitation. The pathogenesis believed to be responsible for this
Pulmonary edema of the right upper lobe associated with acute mitral regurgitation.
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To determine the association between mitral regurgitation and pulmonary edema localized in the right upper lobe, the authors reviewed 21 cases of mitral regurgitation secondary to dysfunction or rupture of the papillary muscle or rupture of the chordae tendineae cordis. The patients, 12 men and 9
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