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osteomalacia/seizures
L'enllaç es desa al porta-retalls
Pàgina 1 des de 61 resultats
Fractures caused by epileptic seizures and epileptic osteomalacia.
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A case of several severe fractures in one patient following epileptic seizures is reported. The patient suffered from epileptic osteomalacia and responded well to vitamin D treatment. The cause of anticonvulsant-induced osteomalacia and its treatment are discussed.
Phenytoin induced severe disabling osteomalacia in a young male with seizure disorder.
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While the use of anti epileptic drugs (AEDs) for a long period is a known risk factor for bone loss and pathological fractures, yet the physicians are not yet sensitized to this possibility. It is now believed that the patients who have fractures due to long-term treatment with anticonvulsants have
Prevention of rickets and osteomalacia in the UK: political action overdue.
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The consequences of vitamin D and dietary calcium deficiency have become a huge public health concern in the UK. The burden of disease from these deficiencies includes rickets, and hypocalcaemic seizures, dilated cardiomyopathy and mostly occult myopathy and osteomalacia. The increasing burden of
Intractable muscle pain syndrome, osteomalacia, and axonopathy in long-term use of phenytoin.
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Muscle pain syndrome is a complicated situation in that even an extensive work-up may overlook the primary etiology. A patient with multiple medical problems, including a history of partial gastrectomy due to trauma, a below-knee amputation from arteriosclerotic disease, and chronic phenytoin
Intractable seizures and metabolic bone disease secondary to celiac disease.
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Celiac disease (CD) is Gluten sensitive enteropathy with a wide spectrum of severity and protean clinical manifestations. Patients with atypical (non-diarrhoeal) presentations are missed as the diagnosis of Celiac Disease is not considered. We present three young girls (ages 18, 19, 23 at
Osteomalacia should be sought and treated before withdrawal of anticonvulsant therapy in UK Asians.
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Individuals from the Asian sub-continent in the United Kingdom are at particular risk of developing osteomalacia. We report a Gujarati woman who developed osteomalacia whilst taking anticonvulsant drugs; withdrawal of anticonvulsant therapy was followed by a seizure complicated by femoral neck
Spot the silent sufferers: A call for clinical diagnostic criteria for solar and nutritional osteomalacia.
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Osteomalacia and rickets result from defective mineralization when the body is deprived of calcium. Globally, the main cause of osteomalacia is a lack of mineral supply for bone modeling and remodeling due to solar vitamin D and/or dietary calcium deficiency. Osteomalacia occurs when existing bone
Multi-trauma secondary to hypocalcaemia-induced seizure: A case report.
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UNASSIGNED
Fractures are known sequelae of seizures. We present a young male with bilateral acetabula and surgical neck of humerus (SNOH), right neck of femur (NOF) and thoracolumbar fractures in the context of a hypocalcaemic seizure secondary to severe malnutrition, secondary hyperparathyroidism
Hypocalcemic seizures and secondary bilateral femoral fractures in an adolescent with primary vitamin D deficiency.
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Nutritional rickets and osteomalacia are reemerging in Western societies, particularly in young children and in adolescents of African or Asian descent. Hypocalcemic seizures resulting from vitamin D deficiency are rare in adolescents, whereas fractures caused by seizures without evidence of direct
Nutritional Rickets and Osteomalacia in the Twenty-first Century: Revised Concepts, Public Health, and Prevention Strategies.
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Nutritional rickets and osteomalacia are common in dark-skinned and migrant populations. Their global incidence is rising due to changing population demographics, failing prevention policies and missing implementation strategies. The calcium deprivation spectrum has hypocalcaemic (seizures, tetany
Congenital rickets presenting with hypocalcaemic seizures.
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At Ataturk University Hospital, eight infants who presented with hypocalcaemic seizures were subsequently found to have rickets. Their mothers had osteomalacia. Neither mothers nor infants received vitamin D supplementation. Maternal vitamin D deficiency and non-supplementation in the infants were
Vitamin D prophylaxis and the lowered incidence of fractures in anticonvulsant rickets and osteomalacia.
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Small doses of vitamin D can probably prevent catastrophic skeletal demineralization in patients taking antiseizure medication. Moderate doses of vitamin D can reverse this degree of demineralization once it has occurred. Prophylactic vitamin D therapy was associated with a lowered incidence of
Incidence of fractures of the vertebral spine in epileptic patients.
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The incidence of vertebral fractures in 87 epileptic outpatients was 16 per cent. The occurrence of vertebral fractures in epileptics was not correlated to age, state of osteomalacia or to type or number of epileptic seizures.
Incidental rickets in the emergency department setting.
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Vitamin D deficiency rickets is a childhood osteomalacia, with impaired skeletal development and potentially skeletal deformities. The radiographic findings of rickets are many but include widening, fraying, and cupping of the metaphysis. Developmental delay and related complications of seizure and
Effects of phenytoin and carbamazepine on calcium transport in Caco-2 cells.
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Adverse effects of anti-seizure/anti-epileptic medications on bone density have been observed and reported since the early 1960s. Phenytoin and carbamazepine are two commonly prescribed anti-epileptic drugs most frequently associated with osteomalacia including fractures, bone demineralization, and
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