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spermatic cord torsion/protease

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Effect of dexmedetomidine on testicular torsion/detorsion damage in rats.

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OBJECTIVE We assessed the antioxidant activity of dexmedetomidine (DEX) during an ischemic period in a rat model of testicular torsion/detorsion (T/DT) by using biochemical and histopathological methods. METHODS Wistar Albino male rats weighing 250-300 g were divided into three groups: sham (group

Vardenafil reduces testicular damage following ischemia/reperfusion injury in rats.

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We investigated the effect of intraperitoneal vardenafil (1 mg/kg) administration during an ischemic period in a rat model of testicular torsion/detorsion (T/D). Twenty-one adult Wistar rats were equally randomized into a control group, a T/D group and a vardenafil group. The control group was

Effects of gabexate mesilate on ischemia-reperfusion-induced testicular injury in rats.

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The aim of this study was to determine the effects of a synthetic serine protease inhibitor, gabexate mesilate (GM), in rats with ischemia-reperfusion (I-R) damage due to unilateral testicular torsion. Thirty male Sprague-Dawley rats were separated into three groups, each containing ten rats. A sham
BACKGROUND The aim of the present study was to investigate the underlying mechanisms in the preventive effects of intravenous anesthetics on testicular ischemia-reperfusion injury. METHODS Forty male Wistar Albino rats were randomly assigned to four groups of 10 rats each. Anesthesia was induced and

Protective effect of hydrogen sulfide on experimental testicular ischemia reperfusion in rats.

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AIM
Testicular torsion is an urgent urological condition. Ischemia-reperfusion (I/R) processes that occur after detorsion as a treatment for torsion are caused by testicular injury. The purpose of our study is investigating the protecting effect of hydrogen sulfide

Role of caspases in male infertility.

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Apoptosis is characterized by a variety of changes resulting in the recognition and phagocytosis of apoptotic cells. Caspases (cysteinyl aspartate-specific proteinases) play a central role in the regulation of apoptosis in the human seminiferous epithelium. They are expressed as inactive proenzymes
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