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tubocurarine/vòmit
L'enllaç es desa al porta-retalls
6 resultats
Omitting antagonism of neuromuscular block: effect on postoperative nausea and vomiting and risk of residual paralysis. A systematic review.
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We have estimated the effect of omitting antagonism of neuromuscular block on postoperative nausea and vomiting. A systematic search (MEDLINE, EMBASE, Biological Abstracts, Cochrane library, reference lists and hand searching; no language restriction, up to March 1998) was performed for relevant
Nondepolarizing neuromuscular blockers inhibit the serotonin-type 3A receptor expressed in Xenopus oocytes.
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Molecular cloning and sequence comparison indicates a high degree of structural homology between muscle nicotinic acetylcholine (nACh) and serotonin-type 3 (5-HT(3A)) receptors, both members of the direct ligand-gated family of ion channels. Because of the structural similarities and common
Relationship between respiratory muscle strength and vital capacity during partial curarization in awake subjects.
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To determine the relationship between respiratory muscle strength and changes in normal vital capacity (VC), graded levels of muscle weakness were produced in six healthy supine male subjects by four successive doses of d-tubocurarine (0.05 mg/kg each). The maximal effect of d-tubocurarine abolished
Alfentanil as an adjuvant of balanced anaesthesia for tonsillectomy in adults.
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In a double-blind study, 80 adult patients, undergoing tonsillectomy, were randomly allocated to one of the four groups: d-tubocurarine (d-Tc) 50 micrograms/kg+alfentanil (Alf) 20 micrograms/kg, d-Tc 50 micrograms/kg+Alf 50 micrograms/kg, Alf 10 micrograms/kg+Alf 20 micrograms/kg, Alf 10
Labetalol as a hypotensive agent for middle ear microsurgery.
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Labetalol, an alpha- and beta-adrenergic receptor-blocking agent, was studied as a hypotensive agent during halothane (mostly 0.5 vol.%)-N2O-fentanyl-d-tubocurarine anaesthesia with a head-up tilt of 5 degrees in 41 patients undergoing middle ear microsurgery. After the mean initial dose of 0.3
Rapid anesthesia induction in combat casualties with full stomachs.
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A series of 50 combat-injured patients given crash intubation for rapid induction of anesthesia is presented. The technic consisted of injection of 3 mg of d-tubocurarine and 0.4 mg of atropine IV, preoxygenation by face mask for 3 minutes, and then injection of sodium thiopental or ketamine,
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