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wattakaka volubilis/antioxidant

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Antioxidant and free-radical-scavenging effects of fruits of Dregea volubilis.

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This study evaluated the in vitro antioxidant potential of petroleum ether (60-80°C), chloroform, and methanol extract of the fruits of Dregea volubilis Benth (Asclepiadaceae). The different antioxidant assays, including total antioxidant activity, reducing power, free radical, super oxide anion

In vitro and in vivo antitumor activity of a methanol extract of Dregea volubilis leaves with its antioxidant effect.

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BACKGROUND In India, Dregea volubilis (L.f.) Benth. ex Hook.f. (Asclepediaceae), a large twining shrub with a woody vine, is used to treat tumors traditionally. OBJECTIVE This study evaluated the in vitro and in vivo antitumor activity of the methanol extract of Dregea volubilis leaves (MEDV) and

Antioxidant activity of Wattakaka volubilis (Linn. f.) leaf extract in carbon tetrachioride induced mice.

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Plants are natural source of antioxidants which ameliorate a variety of diseases. In this study, we determined the in vitro and in vivo antioxidant activity of methanolic, polyphenolic and sapogenin mixture leaf extracts of Wattakaka volubilis (Linn.f.) Stapf, an ayurvedic medicinal plant. The in

Protection against selenite cataract in rat lens by drevogenin D, a triterpenoid aglycone from Dregea volubilis.

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Dregea volubilis is a woody climbing plant commonly found in the hotter parts of India. The leaves are edible and used as a green vegetable, while the plant extract has been used traditionally to treat several diseases including eye ailments. Drevogenin D is a triterpenoid aglycone that has been
Wattakaka volubilis (L.f) (Stapf.) is well known for its rich steroid pregnane contents and this plant is known for its potential use as alternative biological activities. However, little is known about its neuroprotective effect in ischemia-reperfusion (I/R)-induced cerebral injury. Hence, the

Drevogenin D prevents selenite-induced oxidative stress and calpain activation in cultured rat lens.

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OBJECTIVE Selenite-induced cataractogenesis is mediated by oxidative stress, accumulation of calcium and activation of lenticular calpains. Calpains are a super family of Ca2+ dependent proteases, which are involved in lens protein proteolysis and insolubilization. Many inhibitors could prevent
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