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rhabdomyolysis/hypoxia
Odkaz je uložen do schránky
Strana 1 z 83 Výsledek
Rhabdomyolysis and hypoxia associated with prolonged propofol infusion in children.
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Přihlášení Registrace
Propofol, a new anesthetic, is now used more commonly to sedate patients in the intensive care unit. Propofol's rapid elimination has popularized its use to induce and maintain hypnosis in patients with refractory status epilepticus. It is also associated with occasional severe metabolic acidosis
Evidence for sustained renal hypoxia and transient hypoxia adaptation in experimental rhabdomyolysis-induced acute kidney injury.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
BACKGROUND
Indirect evidence suggests that hypoxia contributes to the pathophysiology of rhabdomyolysis-induced acute kidney injury (AKI). However, the cellular location and kinetics of hypoxia, as well as potential hypoxia adaptation are unclear.
METHODS
Rhabdomyolysis was induced in rats by IM
Rhabdomyolysis-induced acute kidney injury under hypoxia and deprivation of food and water.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
BACKGROUND
To investigate the renal pathophysiologyin rhabdomyolysis-induced acute kidney injury (AKI) in rats under hypoxia and deprivation of food and water (HDFW), thus broadening the knowledge about rhabdomyolysis-induced AKI in massive earthquake.
METHODS
Male Wistar rats weighing 200-230g were
Rhabdomyolysis and severe haemolytic anaemia, hepatic dysfunction and intestinal osteopathy due to hypophosphataemia in a patient after Billroth II gastrectomy.
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Přihlášení Registrace
Hypophosphataemic syndromes lead to appreciable morbidity and mortality. A deficiency or lack of phosphate leads to tissue hypoxia and disruption of cellular function, which may cause severe clinical complications. We present various manifestations of hypophosphataemia; in all cases, diagnosis was
[Metabolic myopathy causing rhabdomyolysis].
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Přihlášení Registrace
Rhabdomyolysis is a potential life-threatening condition characterized by a rapid destruction of striated muscle cells. Causes include trauma, muscle hypoxia, drugs, malignant hyperthermia, infections, toxins and metabolic myopathies. We report a case of a 52-year-old man with hereditary
Blood cell exchange in the treatment of exercise-induced rhabdomyolysis in a patient with sickle cell trait.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
We report the use of red blood cell exchange (RBCex) to treat rhabdomyolysis complicated by acute kidney injury in a 16-year-old African-American female with sickle cell trait (SCT). Treatment with aggressive fluid and electrolyte management failed to stem the rise in her creatine kinase, and RBCex
Rhabdomyolysis following status asthmaticus.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
A case of rhabdomyolysis following an asthmatic attack is reported. A 71-year-old man was admitted because of wheezing and hypoxemia. Brown urine was present on admission. Although these symptoms completely disappeared with the treatment with aminophylline, salbutamol and corticosteroid, transiently
Cutaneous eruptions in coma patients with nontraumatic rhabdomyolysis.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
BACKGROUND
Rhabdomyolysis has been described most commonly after muscle injury but may also result from coma due to alcohol intake or drug abuse. Its clinical findings usually occur as muscular pain and swelling, but these symptoms are also seen in as many as 60% of patients with nontraumatic
[Myocardial involvement in rhabdomyolysis caused by acute heroin intoxication].
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Přihlášení Registrace
It is well known that heroin overdose may cause rhabdomyolysis. Sometimes a myocardial involvement complicates its course. We report a case of heroin intoxication followed by rhabdomyolysis associated with myocardial injury, with symptoms, laboratory findings, ECG and echocardiography features of
Non-traumatic rhabdomyolysis with gluteal ulcer.
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Přihlášení Registrace
The case is presented of a 34-year-old woman with rhabdomyolysis due to massive intake of nimetazepam, a benzodiazepine hypnotic. On admission, the patient had numerous blisters all over the body. One of the blisters in the gluteal region developed into a deep ulcer accompanied by muscle necrosis
Unreliable Early Neuroprognostication After Severe Carbon Monoxide Poisoning Is Likely Due to Cytopathic Hypoxia: A Case Report and Discussion.
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Přihlášení Registrace
A 17-year-old girl was found unconscious in a running vehicle. She developed very severe acute respiratory distress syndrome (which was treated with rescue high-frequency oscillation), hemodynamic instability, acute kidney injury, rhabdomyolysis, and remained comatose with a Glasgow Coma Scale score
Successful management of a dog that had severe rhabdomyolysis with myocardial and respiratory failure.
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Přihlášení Registrace
CASE DESCRIPTION - A 6-year-old castrated male Llewelyn Setter was evaluated because of an acute onset of myalgia and respiratory distress. CLINICAL FINDINGS - Physical examination revealed a stiff stilted gait, swollen muscles that appeared to cause signs of pain, panting, and ptyalism. The dog had
Capture-induced exertional rhabdomyolysis in the Shortfin Mako Shark, Isurus oxyrinchus.
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Přihlášení Registrace
Electrolyte disturbances in foals with severe rhabdomyolysis.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
Marked electrolyte abnormalities characterized by profound hyperkalemia, hyponatremia, hypocalcemia, and hyperphosphatemia were noted in 4 neonatal foals with acute rhabdomyolysis and pigmenturia. In 2 foals, rhabdomyolysis developed 4-6 days after admission for dysmaturity, and in 2 foals,
Severe hypokalemia, rhabdomyolysis, muscle paralysis, and respiratory impairment in a hypertensive patient taking herbal medicines containing licorice.
Články mohou překládat pouze registrovaní uživatelé
Přihlášení Registrace
A 93 year-old hypertensive woman was found to have severe hypokalemia (as low as 1.3 mEq/L) and developed paralysis of the all extremities associated with metabolic alkalosis, hypoxemia, hypercapnea, extremely high levels of creatine phosphokinase (up to 9280 U/L), myoglobin and myoglobinuria
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