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[Cerebrospinal fluid chlorine-soluble proteins and lipoproteins in the differential diagnosis of cerebral infarct and brain tumor].
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Results of determining the chemical composition and the electrophoretic spectrum of chlorine-soluble proteins and lipoproteins in the cerebrospinal fluid of patients with cerebral infarctions and brain tumours are presented. The determinations showed non-identity of those proteins in the patients
Myocardial infarction, acute ischemic stroke, and hyperglycemia triggered by acute chlorine gas inhalation.
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Chlorine is one of the most common substances involved in toxic inhalation. Until now, several accidental exposures have been reported. The damage to the respiratory tract in the immediate phase after exposure to chlorine is well defined. Death occurs particularly due to pulmonary edema with
[Clinical regularities of myocardial infarction repair].
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A total of 136 patients with trans- and 200 with non-transmural myocardial infarction (MI) were examined over time for chlorine-soluble mucoprotein (MP), serum hexoses (SH), their fraction-hexoses of glycosaminoglycans (H-GAG), and hexoses of glycoproteins (H-GP), serum, plasma and urine
[Role of inflammation markers in the evaluation of the clinical course of myocardial infarct].
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The authors examined the time course of changes in the levels of chlorine soluble mucoprotein and glycosaminoglycans as markers of an acute period of myocardial infarction. A significant correlation was established between the mass of the infarcted myocardium and the severity of its acute reaction
Engineered heart tissue transplantation alters electrical-conduction function in rats with myocardial infarction.
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OBJECTIVE
To investigate how affects electrical-conduction function following myocardial infarction (MI) in rats.
METHODS
Thirty SD rats of either sex (220-250 g) were anesthetized using chlorine hydrate and were randomly divided into three groups: control group, MI group and transplantation group.
In vivo chlorine-35, sodium-23 and proton magnetic resonance imaging of the rat brain.
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In this study we demonstrate the feasibility of combined chlorine-35, sodium-23 and proton magnetic resonance imaging (MRI) at 9.4 Tesla, and present the first in vivo chlorine-35 images obtained by means of MRI. With the experimental setup used in this study all measurements could be done in one
Temporal Trends in Cardiovascular Hospital Discharges Following a Mass Chlorine Exposure Event in Graniteville, South Carolina.
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Background: On January 6, 2005, a train derailed in Graniteville, South Carolina, releasing nearly 60,000 kg of toxic chlorine gas. The disaster left nine people dead and was responsible for hundreds of hospitalizations and outpatient visits in the subsequent weeks. While chlorine gas
[The diagnosis and reparative processes in a protracted course of myocardial infarct].
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A study of 20 patients with protracted myocardial infarction and 142 patients with transmural myocardial infarction showing a routine course revealed that the clinico-biochemical equivalent of prolonged course of reparative processes in protracted myocardial infarction are up to 2 months registered
[Study on improving the speed and quality of making rat myocardial infarction model by coronary artery ligation].
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OBJECTIVE
To establish the methods for improving the speed and quality of making rat myocardial infarction model by coronary artery ligation.
METHODS
With the precondition of having no effect on rat myocardial infarction size: (1) To compare the thoracotomy time, the ligation time of the way of
Migrainous Infarction in a Patient With Sporadic Hemiplegic Migraine and Cystic Fibrosis: A 99mTc-HMPAO Brain SPECT Study.
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Genetic mutations of sporadic hemiplegic migraine (SHM) are mostly unknown. SHM pathophysiology relies on cortical spreading depression (CSD), which might be responsible for ischemic brain infarction. Cystic fibrosis (CF) is caused by a monogenic mutation of the chlorine transmembrane conductance
Potential for chlorine gas-induced injury in the extrapulmonary vasculature.
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Exposure to chlorine gas (Cl(2)) primarily causes injury to the lung and is characterized by inflammation and oxidative stress mediated by reactive chlorine species. Reducing lung injury and improving respiratory function are the principal therapeutic goals in treating individuals exposed to Cl(2)
[The role of post-translational modification of fibrinogen in the pathogenesis of thrombosis].
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Fibrinogen is a precursor of fibrin, which is the main component of the blood clot. The opposite of coagulation is fibrinolysis. The proper functioning of both systems allow to maintain a hemostasis. Increasing level of fibrinogen is an important risk factor for myocardial infarction or ischemic
Cardiovascular risk markers in hemodialysis patients.
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Cardiovascular events are the main cause of death in hemodialysis patients. Nevertheless, acute myocardial infarction may be misdiagnosed in uremic patients, because typical markers have a high rate of false positivity. A recent two-year prospective study showed that predialytic high serum
[Cardiovascular risk markers in hemodialysis].
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OBJECTIVE
Although maintenance dialysis prevents death from uremia, patient survival remains an important issue. Cardiovascular (CV) events have been considered the main cause of death in hemodialysis (HD) patients. Some authors demonstrated an expected remaining life span of < or =2 yrs in HD
Serum and cerebrospinal fluid magnesium levels, Glasgow Coma Scores, and in-hospital mortality in patients with acute stroke.
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The aim of this study was to determine the relationship between serum and cerebrospinal fluid (CSF) magnesium(Mg+2) levels, Glasgow Coma Scores (GCS), and 7-day mortality in acute stroke patients. Patients with acute ischemic or hemorrhagic stroke arriving within the first 3 h of symptoms were
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