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Resolution of hydrops secondary to cytomegalovirus after maternal and fetal treatment with human cytomegalovirus hyperimmune globulin.
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BACKGROUND
Congenital cytomegalovirus (CMV) is a common infection with limited treatment options. Vertical transmission can lead to fetal death or long-term neurologic injury. We present a case wherein fetal hydrops resolved after maternal and fetal intravenous administration of CMV hyperimmune
Anti-thymocyte globulin induced non-cardiogenic pulmonary edema during renal transplantation.
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Non-cardiogenic pulmonary edema (NCPE) is a clinical syndrome characterized by simultaneous presence of severe hypoxemia, bilateral alveolar infiltrates on chest radiograph, without evidence of left atrial hypertension/congestive heart failure/fluid overload. The diagnosis of drugrelated NCPE relies
THE PERMEABILITY GLOBULINS OF HUMAN SERUM AND THE BIOCHEMICAL MECHANISM OF HEREDITARY ANGIONEUROTIC EDEMA.
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Deficiency in gamma globulin with edema and hypoproteinemia.
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Hereditary angioneurotic edema. II. Deficiency of inhibitor for serum globulin permeability factor and/or plasma kallikrein.
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Idiopathic hypoproteinemia; report of a case of transient edema, depression of plasma albumin and gamma globulin and eosinophilia.
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The persistence in the blood of the radioactive label of albumins, gamma globulins, and globulins of intermediate mobility. IV. Specific activity ratios between plasma and edema fluid of I 131 labeled albumin injected intravenously in nutritional and nephrotic hypoproteinemia.
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Nonimmune fetal hydrops: diagnosis and obstetrical management.
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As the incidence of Rh-isoimmunization has been decreasing with the availability of Rh immune globulin, the proportion of cases of fetal hydrops from nonimmune causes has increased. Evaluation of the fetus with hydrops requires an integrated approach, beginning with targeted ultrasound evaluation
The changing pattern of fetal hydrops.
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Fetal hydrops (hydrops fetalis) remains a significant cause of fetal and neonatal mortality. The decreased incidence of rhesus iso-immunisation due to prophylaxis with rhesus immune globulin (anti-D), improved antenatal ultrasound screening, and advances in neonatal intensive care have greatly
Acute dermatomyositis with subcutaneous generalized edema.
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The authors report a 40-year-old Caucasian man with relapsing muscle and skin involvement of dermatomyositis treated with high-dose corticosteroids, taken orally, and methotrexate and human gamma globulin, both administered intravenously. After 4 months of aggressive treatment, he presented with
Kawasaki disease associated with gallbladder hydrops.
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Kawasaki disease is an acute, febrile, systemic syndrome of unknown etiology which principally affects young children. We present a five-year-old boy, who developed an unusual finding, acute hydrops of the gallbladder, in addition to the characteristic signs and symptoms of Kawasaki disease.
[Acute non-immunologic hydrops fetalis with bilateral chylothorax in the 36th week of pregnancy].
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Since the introduction of anti-D gamma-globulin prophylaxis non-immune hydrops fetalis (NIHF) has become the relevant and more common form of fetal hydrops. A case of an NIHF with chylothorax, diagnosed in the 36th week of pregnancy, is discussed with regard to obstetrical and neonatological
Effects of antihistamines on the lung vascular response to histamine in unanesthetized sheep. Diphenhydramine prevention of pulmonary edema and increased permeability.
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To see whether antihistamines could prevent and reverse histamine-induced pulmonary edema and increased lung vascular permeability, we compared the effects of a 4-h intravenous infusion of 4 mug/kg per min histamine phosphate on pulmonary hemodynamics, lung lymph flow, lymph and plasma protein
[Obtaining the beta-globulin fractions from swine and cattle sera and a study of their immunological activity].
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The rivanol precipitation was used to obtain beta-globulin fractions from specific swine sera against edema disease, paratyph and Aujeszky's disease as well as from normal ovine and swine sera. Agar electrophoresis revealed that the preparations produced contained beta-globulin (86 per cent),
[The effect of neuroleptics on blood proteins during development of toxic cerebral edema-brain swelling].
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Simulation of toxic brain edema-swelling allowed one to analyze the dynamics of blood protein alteration in presence of various neuroleptics. Alterations in blood protein fractions correlated with dynamics of nervous tissue impairments. All the drugs studied exhibited similar antiswelling action
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