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hyperventilation/slagtilfælde
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[Transcranial Doppler ultrasonography and hyperventilation test in assessment of cerebral vasoreactivity after ischemic stroke].
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In 36 patients 3 month after ischaemic stroke in regions supplied by MCA (Middle Cerebral Artery) physical examination, CT scanning and blood flow velocity recordings in ICA (Internal Carotid Artery) and MCA were performed. In both MCA blood flow velocity was measured in resting state and after 30
The influence of hyperventilation on the measurement of stroke volume using a CO2 rebreathing method.
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The influence of different degrees of hyperventilation on stroke volume measured with a CO2 rebreathing method was studied in seven normal subjects and seven patients with aortic regurgitation. Hyperventilation was initially performed with a rebreathing rate of 30 min-1 and a tidal volume
Electroencephalography hyperventilation and stroke in children with sickle cell disease.
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A recent study and report in which hyperventilation was used during electroencephalography (EEG) in 6 children with sickle cell disease (SCD) and seizures, without serious complication, prompted a cautionary response regarding the potential risks attending the practice of EEG hyperventilation in SCD
Confounding of Cerebral Blood Flow Velocity by Blood Pressure During Breath Holding or Hyperventilation in Transient Ischemic Attack or Stroke.
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Background and Purpose- Breath holding (BH) and hyperventilation are used to assess abnormal cerebrovascular reactivity, often in relation to severity of small vessel disease and risk of stroke with carotid stenosis, but responses may be confounded by blood pressure (BP) changes. We compared effects
Mechanism of hyperventilation in acute cerebrovascular accidents.
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Lumbar cerebrospinal fluid and arterial blood acid-base state were assessed in 19 patients within 24 hours of an acute cerebrovascular accident. Those with haemorrhage into the C.S.F. showed a lower C.S.F. pH and higher C.S.F. lactate than those without haemorrhage but the Pco(2), was similar in the
Hyperventilation-precipitated cerebrovascular accident in a patient with sickle cell anaemia.
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Hyperventilation exercise during electroencephalography precipitated a recurrence of right hemiplegia and aphasia in a patient with Hb SS disease. Although recovery of function started within hours of the event, full recovery has not occurred six months after. Hyperventilation provocative test
Prolonged artificial hyperventilation in cerebral apoplexy.
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Hyperventilation and vomiting due to posterior circulation stroke.
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Artifical hyperventilation in stroke.
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The development of hyperventilation in patients with chronic heart failure and Cheyne-Strokes respiration: a possible role of chronic hypoxia.
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OBJECTIVE
To analyze the relationship between daytime respiratory and cardiac function in patients with compensated chronic heart failure (CHF) with and without periodic breathing (PB) or Cheyne-Stokes respiration (CSR).
METHODS
We studied 132 patients (female, 13%; mean age, 53+/-8 years; body mass
Cerebral apoplexy (stroke) treated with or without prolonged artificial hyperventilation. 1. Cerebral circulation, clinical course, and cause of death.
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Cerebral apoplexy (stroke) treated with or without prolonged artificial hyperventilation. 2. Cerebrospinal fluid acid-base balance and intracranial pressure.
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Prolonged artificial hyperventilation in severe cerebral apoplexy. Clinical results and cerebrospinal fluid findings in a controlled study.
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Bi-hemispheric CBF and its CO2 reactivity of TIAs and completed strokes in ICA occlusions.
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Bi-hemispheric cerebral blood flow (CBF) measurements during rest and hyperventilation, with intra-arterial 133Xe injection method, were investigated in 19 cases, angiographically diagnosed as unilateral internal carotid artery (ICA) occlusion, including 8 cases with TIAs and 11 cases with completed
Altered cerebral vasoregulation in hypertension and stroke.
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BACKGROUND
Autoregulation of blood flow that maintains steady perfusion over the range of systemic blood pressure is compromised by stroke.
OBJECTIVE
To determine whether cerebral vasoregulation is impaired in stroke during orthostatic stress.
METHODS
Subjects included 30 control subjects, 30 with
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