ketosis/epileptisk anfald

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Clinical Implications of Ketosis in Children with Benign Convulsions with Mild Gastroenteritis.

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The pathophysiologic mechanisms underlying benign convulsions with mild gastroenteritis (CwG) in children remain unclear. We investigated the incidence of ketosis in CwG and whether this is related to seizures.This retrospective study included children aged

Severe diabetic ketoacidosis complicated by hypocapnic seizure.

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In clinical practice, seizures independent of hypoglycemia are observed in patients with type 1 diabetes mellitus (T1DM) more frequently than expected by chance, suggesting a link. However, seizures during management of diabetic ketoacidosis (DKA) have generally been considered a bad prognostic

Hypophosphatemia-induced seizure in a child with diabetic ketoacidosis.

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We report an unusual case of hypophosphatemia-related seizure in a child with diabetic ketoacidosis (DKA). A 1-year-old type 1 diabetic boy with hyperglycemia, ketoacidosis, and dehydration was admitted to the pediatric intensive care unit. After having received fluid replacement using isotonic

Therapeutic ketosis with ketone ester delays central nervous system oxygen toxicity seizures in rats.

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Central nervous system oxygen toxicity (CNS-OT) seizures occur with little or no warning, and no effective mitigation strategy has been identified. Ketogenic diets (KD) elevate blood ketones and have successfully treated drug-resistant epilepsy. We hypothesized that a ketone ester given orally as

Dietary fat, ketosis, and seizure resistance in rats on the ketogenic diet.

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OBJECTIVE Fat is the major component of the ketogenic diet (KD), yet no studies have examined whether the type of fat used in the diet can be optimized to provide additional benefits. The purpose of the present experiments was to compare the efficiency of different fats in inducing ketosis and

Seizures and movement disorders induced by hyperglycemia without ketosis in elderly.

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BACKGROUND Non-ketotic hyperglycemia (NKHG) may increase the probability of seizures and movement disorders. METHODS We describe a series of 14 elders admitted for seizures and movement disorders linked to NKHG. RESULTS Twelve patients developed motor seizures and two others movement disorders.

Ketonemia and seizures: metabolic and anticonvulsant effects of two ketogenic diets in childhood epilepsy.

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Metabolic effects of a new ketogenic regimen in which ketonemia is induced by feeding of medium chain triglycerides (MCT) are described, and comparisons are made with effects of the standard high fat ketogenic diet. Eighteen children maintained on the MCT diet for 3 months to 4 years failed to show

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ketosis/epileptisk anfald

Clinical Implications of Ketosis in Children with Benign Convulsions with Mild Gastroenteritis.

Severe diabetic ketoacidosis complicated by hypocapnic seizure.

Hypophosphatemia-induced seizure in a child with diabetic ketoacidosis.

Therapeutic ketosis with ketone ester delays central nervous system oxygen toxicity seizures in rats.

Dietary fat, ketosis, and seizure resistance in rats on the ketogenic diet.

Seizures and movement disorders induced by hyperglycemia without ketosis in elderly.

Ketonemia and seizures: metabolic and anticonvulsant effects of two ketogenic diets in childhood epilepsy.

Convulsions during diabetic ketosis; a case report.

Re: Dietary fat, ketosis, and seizure resistance in rats on the ketogenic diet.

The effect of ketosis of EEG and on susceptibility to seizure.

Focal seizures as a manifestation of hyperglycemia without ketoacidosis. A case report.

[Convulsions during a coma caused by hyperglycemia and hyperosmolarity, without acetonemia].

Subcortical reversible T2-weighed hypointensities in seizures secondary to diabetic ketoacidosis.

[Vomiting with acetonuria immediately following an asthmatic seizure; interpretation].

[Epileptic seizures of the supplementary motor area in hyperglycemia without ketosis].

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