myalgia/epileptisk anfald

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Seizures and Basal Ganglia Calcification Caused by Hypoparathyroidism

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Seizures, not a typical feature of hypoparathyroidism, may present along with myalgia and tetany in patients of hypoparathyroidism. Thorough history and examination, derangement of biochemical parameters such as hypocalcemia, hyperphosphatemia, and inappropriately low levels of parathyroid hormone,

Intractable muscle pain syndrome, osteomalacia, and axonopathy in long-term use of phenytoin.

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Muscle pain syndrome is a complicated situation in that even an extensive work-up may overlook the primary etiology. A patient with multiple medical problems, including a history of partial gastrectomy due to trauma, a below-knee amputation from arteriosclerotic disease, and chronic phenytoin

Botulinum toxin for painful spasms from focal seizures: theoretical considerations and case report.

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BACKGROUND Botulinum toxin (BTX) has been used to treat muscle overactivity and can reduce pain from muscle spasm. There are no previous reports to our knowledge of using BTX to treat the pain from intractable focal motor seizures. METHODS We present a case of chronic, focal, muscle pain, and spasm

Does rocuronium-sugammadex reduce myalgia and headache after electroconvulsive therapy in patients with major depression?

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BACKGROUND We aimed to compare the effects of succinylcholine and rocuronium-sugammadex on development of myalgia and headache after electroconvulsive therapy (ECT). METHODS Forty-five patients undergoing ECT were enrolled in the study. Anesthesia induction was provided with propofol 1 mg/kg

The Incidence and Predictors of Headache and Myalgia in Patients After Electroconvulsive Therapy (ECT).

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BACKGROUND Electroconvulsive therapy (ECT) is a safe and effective mode of therapy for a wide variety of psychiatric disorders. However, it is associated with some disturbing side effects, such as nausea and vomiting, dental and tongue injury, confusion, dizziness, headache, and

Correlates of myalgia in electroconvulsive therapy.

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Myalgias are common in patients treated with electroconvulsive therapy (ECT). The mechanism of this side effect is unknown. Two commonly postulated etiologies are the motor activity during the convulsion and the fasciculations induced by succinylcholine. If the former phenomenon accounts for most of

Effects of preemptive intravenous paracetamol and ibuprofen on headache and myalgia in patients after electroconvulsive therapy: A placebo-controlled, double-blind, randomized clinical trial.

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The aim of this study is to determine the efficacy of preemptive analgesia with paracetamol and ibuprofen to reduce the intensity and incidence of headache and myalgia after electroconvulsive therapy (ECT).Sixty patients with major depression who were

Cytokine dermatitis and febrile seizure from imiquimod.

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Cytokine dermatitis is a well-known and common clinical adverse effect of imiquimod 5% cream (Aldara, 3M). Data from initial Phase III clinical trials reveal a minority of study drug patients experience systemic adverse effects, including fever, arthralgia, headache, myalgia, and lymphadenopathy.

Modification of tonic-clonic convulsions by atracurium in multiple-monitored electroconvulsive therapy.

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OBJECTIVE To determine the effect of two different doses of atracurium on the modification of tonic-clonic convulsions in multiple-monitored electroconvulsive therapy (MMECT). To compare recovery time and adverse reactions of these doses. METHODS Clinical study. Anesthesiologist was blinded in the

The effect of ketamine versus thiopental sodium on hemodynamic profile and seizure duration during electroconvulsive therapy in patients with major depressive disorder: A randomized cross-over clinical trial

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Background: Electroconvulsive therapy (ECT) is an effective treatment in major depressive disorder (MDD). Earlier studies suggest that ketamine has antidepressive effects and prolongs seizure duration in favor of therapeutic efficacy of ECT. A great concern with the use of ketamine is its

[Rhabdomyolysis related to statin and seizures: report of 3 cases].

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OBJECTIVE To investigate the clinical features and prognosis of rhabdomyolysis related to seizure attacks and use of statin. METHODS The medical records of 3 patients with established diagnosis of rhabdomyolysis were analyzed and the related literatures were reviewed. RESULTS All the 3 patients had

Fluoxetine overdose in a teenager resulting in serotonin syndrome, seizure and delayed onset rhabdomyolysis.

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A 14-year-old young adult took an overdose of 1.2 g of fluoxetine, a selective serotonin reuptake inhibitor (SSRI) that he had been prescribed for depression. He had a generalised tonic/clonic seizure at 6 hours postingestion.After the seizure, he developed signs consistent with serotonin syndrome:

Severe, self-limiting lactic acidosis and rhabdomyolysis accompanying convulsions.

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A 26 year old man with no previous history of convulsions presented in status epilepticus and severe lactic acidosis. He regained consciousness and the acidosis resolved after several hours of conservative management without intravenous bicarbonate, but he developed severe myalgia associated with

Bromide toxicosis (bromism) in a dog treated with potassium bromide for refractory seizures.

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A 4-year-old German Shepherd Dog was evaluated because of chronic hind limb lameness and recurrent seizures. Diagnostic evaluation of the dog confirmed rheumatoid arthritis and idiopathic epilepsy. The rheumatoid arthritis was treated with prednisone and piroxicam. The seizures were treated with

Unilateral seizures in a patient with hairy cell leukemia treated with interferon.

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A patient is described who developed unilateral seizures whilst being treated with recombinant interferon for hairy cell leukemia. Special features included the relatively low dose of interferon, the focal aspect of the epilepsy and the high resistance to anticonvulsants. Oligoclonal banding of

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