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pyruvate kinase/infarkt
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[A clinical and experimental study on early diagnosis of myocardial infarct extension by measuring the activity of serum pyruvate kinase].
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To explore the early diagnostic effects of serum pyruvate kinase (PK) on myocardial infarct extension (MIE), the activity of serum PK, glutamate oxalacetate transaminase (GOT), creatine kinase (CK) and its isoenzyme (CK-MB) were measured in 102 cases of acute myocardial infarction (AMI). The results
Serum enolase and pyruvate kinase activities in the diagnosis of myocardial infarction.
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Sequential determinations of serum enolase and pyruvate kinase (PK) activities were carried out in addition to routine enzyme assays on 191 patients admitted to a coronary care unit. The upper reference values established in 61 healthy laboratory staff members were 21 U/1 for enolase and 76 U/1 for
HIF1 mediates a switch in pyruvate kinase isoforms after myocardial infarction.
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Alternative splicing of RNA is an underexplored area of transcriptional response. We expect that early changes in alternatively spliced genes may be important for responses to cardiac injury. Hypoxia inducible factor 1 (HIF1) is a key transcription factor that rapidly responds to loss of oxygen
[Pyruvate kinase in acute myocardial infarct].
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Serum pyruvate kinase in acute myocardial infarction.
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Pyruvate kinase isoenzymes in progressive muscular dystrophy and in acute myocardial infarction.
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[Pyruvate kinase deficiency, infectious mononucleosis, hemolytic anemia with cold autoantibodies and massive splenic infarction].
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[Clinical significance of determining plasma pyruvate kinase (PK) activity in acute myocardial infarction (author's transl)].
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Activity of phosphoglycerate mutase and its isoenzymes in serum after acute myocardial infarction.
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Aims/background-In humans there are three phosphoglycerate mutase (PGM, EC 5.4.12.1) isoenzymes (MM, MB and BB) which have similar distribution and developmental pathways to creatine kinase (CK, EC 2.7.3.2) isoenzymes. Total serum PGM activity increases in acute myocardial infarction with the same
Lipid metabolism, cerebral metabolic rate, and some related enzyme activities after brain infarction in rats.
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Multiple infarcts were produced in cerebral hemispheres of rats by injecting calibrated 50-micron microspheres into the left internal carotid artery, and alterations in lipid and energy metabolism were evaluated 24 hours later in the embolized hemisphere. Total phospholipid content was decreased by
Pyruvate Kinase M2 Increases Angiogenesis, Neurogenesis, and Functional Recovery Mediated by Upregulation of STAT3 and Focal Adhesion Kinase Activities After Ischemic Stroke in Adult Mice.
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Ischemic stroke remains a serious threat to human life. Generation of neuronal and vascular cells is an endogenous regenerative mechanism in the adult brain, which may contribute to tissue repair after stroke. However, the regenerative activity is typically insufficient for significant therapeutic
Effects of acupuncture on glycometabolic enzymes in multi-infarct dementia rats.
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Acupuncture has exhibited therapeutic effects on vascular dementia in our previous research. The mechanism of its anti-dementia effects involves energy metabolism. For brain cells, glucose metabolism is almost the only source of energy, and glucose metabolism disorders are early signs of dementia.
High-energy phosphotransfer in the failing mouse heart: role of adenylate kinase and glycolytic enzymes.
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OBJECTIVE
To measure the activity of the key phosphotransfer enzymes creatine kinase (CK), adenylate kinase (AK), and glycolytic enzymes in two common mouse models of chronic heart failure.
RESULTS
C57BL/6 mice were subjected to transverse aortic constriction (TAC), myocardial infarction induced by
[The spleen in non-malignant haematological disorders].
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The spleen functions as a filter of the circulating blood, removing aging or abnormal red blood cells, intraerythrocyte inclusions as well as foreign particals. As the spleen is composed of lymphocytic tissue, circulatory elements and mononuclear phagocytic cells it plays an important role in the
Inhibition of red blood cell-induced platelet aggregation in whole blood by a nonionic surfactant, poloxamer 188 (RheothRx injection).
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RheothRx Injection, an aqueous solution of a nonionic block copolymer (poloxamer 188) formulated for intravenous administration, was investigated as an inhibitor of red blood cell (RBC)-induced platelet aggregation at plasma concentrations of 0.05-5mgmL-1. Platelet aggregation was determined by
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