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Side 1 fra 48 resultater
Quinidine-induced thrombocytopenia with pulmonary hemorrhage.
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Examination of an elderly man with quinidine sulfate-induced thrombocytopenia complicated by pulmonary hemorrhage failed to identify any underlying pulmonary disease contributing to the bleeding All bleeding ceased, and pulmonary infiltrates disappeared after the platelet count returned to normal.
Intracranial hemorrhage associated with quinidine induced thrombocytopenia.
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Quinidine is commonly used in the treatment of atrial and ventricular arrhythmias. Such patients are at increased risk for embolic strokes and may require concurrent anticoagulation therapy. We report here the occurrence of intracranial hemorrhage as a complication of thrombocytopenia in two
Cumulative effects of quinidine and aspirin on bleeding time and platelet alpha 2-adrenoceptors: potential mechanism of bleeding diathesis in patients receiving this combination.
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We observed quinidine-induced prolongation of bleeding time without thrombocytopenia in three subjects. In addition, we noticed a cumulative prolongation of bleeding time by a combination of quinidine and aspirin. We postulated that because both quinidine and aspirin inhibit epinephrine-induced
Hemorrhagic diathesis related to quinidine therapy.
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[Case of thrombocytopenia with hemorrhage following quinidine therapy].
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[Thrombopenic complications with hemorrhage, due to quinidine therapy].
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A second case of quinidine-induced thrombocytopenia with pulmonary hemorrhage.
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Quinidine-induced hypoprothrombinemic hemorrhage in patients on chronic warfarin therapy.
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Oral hemorrhage after the use of quinidine: report of case.
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Possible quinidine-induced hemorrhage in a patient on warfarin sodium.
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Managing chronic atrial fibrillation: a Markov decision analysis comparing warfarin, quinidine, and low-dose amiodarone.
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OBJECTIVE
To compare the relative risks and benefits of several clinical strategies for managing patients with chronic atrial fibrillation.
METHODS
Five recent randomized controlled trials of warfarin in atrial fibrillation, 6 randomized controlled trials of quinidine, and 13 longitudinal studies of
Immunoreactions involving platelets. IV. Studies on the pathogenesis of thrombocytopenia in drug purpura using test doses of quinidine in sensitized individuals; their implications in idiopathic thrombocytopenic purpura.
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Regulated intravenous doses of quinidine were given to patients with the antibody of quinidine purpura to produce controlled thrombocytopenia without clinical sequelae. The degree of thrombocytopenia and the rate at which it developed were dependent on the relative plasma concentration of quinidine
[Fatal thrombocytopenia triggered by quinidine].
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A woman aged 60 years receiving anticoagulation treatment on account of artificial mitral and aortic valve prostheses developed severe thrombocytopenia three weeks after the commencement of quinidine treatment. The results of investigations suggested severe thrombocytopenia precipitated by
Fatal quinidine-induced thrombocytopenia following open-heart surgery.
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Three patients developed quinidine-induced thrombocytopenia within 3 months of our initiating quinidine therapy after open-heart surgery. One patient recovered from thrombocytopenic purpura after quinidine was discontinued. The 2 other patients presented with thrombocytopenic purpura and gingival
Intravenous immune globulin for the treatment of presumed quinidine-induced thrombocytopenia.
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Quinidine is cited as one of the most frequent causes of drug-induced thrombocytopenia. This case report describes the use of high-dose intravenous gamma globulin to rapidly reverse quinidine-induced thrombocytopenia in a patient at increased risk for bleeding because of the presence of an
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