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Side 1 fra 481 resultater
Peptic ulcer disease and the tar and nicotine yield of currently smoked cigarettes.
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In order to examine the association of smoking cigarettes with a lower tar and nicotine yield to peptic ulcer disease, a common disease associated in past studies with cigarette smoking. We used questionnaire information provided by 9009 current regular cigarette smokers. The percentage who ever had
Risk factors of duodenal ulcer bleeding: the role of smoking and nicotine.
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Several studies have shown that cigarette smoking affects duodenal ulcer (DU) recurrence. To verify any correlation between smoking and complications of ulcer disease, we studied 33 DU smokers, 16 DU ex-smokers and 87 DU non-smokers for up to 48 months, recording age, sex, family history of ulcer,
Natural history of peptic ulcer disease in Poland. Part IV. Dietary habits, mode of eating, coffee, tea, tobacco and alcohol, addiction's.
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In the "Ulcer" research programme 5813 male and female patients with peptic ulcer of the upper gastrointestinal tract were analysed paying attention to the quality of their diet and mode of meal taking. Weight loss was found to occur in these patients and information was gathered concerning alcohol,
Acute effects of smoking during modified sham feeding in duodenal ulcer patients. An analysis of nicotine, acid secretion, gastrin, catecholamines, epidermal growth factor, prostaglandin E2, and bile acids.
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Smoking is associated with an increased incidence of duodenal ulcer with a high relapse rate, and smokers tend to be slow healers. The etiology responsible for this remains unknown, and there is general disagreement as to whether smoking affects gastric secretion. The aim of the present study was to
[Particular features of the clinical course of ulcer in tobacco farmers from Kyrgyzstan].
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The article describes the results of the dispensary follow-up of patients with stomach and duodenal ulcers residing in tobacco farming districts in Osh Region (Kyrgyzstan). It turned out that tobacco farmers suffer from ulcers more frequently with a more severe course of the disease and various
Association between recurrent aphthous ulcers and tobacco habits.
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The study is a complication of data on oral mucosal lesions collected among 20 333 people aged 15 yr and over. The aim was to calculate the correlation between the prevalence of recurrent aphthous ulcers and various tobacco habits. The prevalence of aphthous ulcers in the population was 17.7%
Tobacco use as contributory factor in peptic ulcer disease.
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OBJECTIVE
To find out the association of tobacco as a contributory factor in patients presenting with peptic ulcer disease (PUD).
METHODS
A descriptive case study.
METHODS
Department of Medicine, Postgraduate Medical Institute, Lady Reading Hospital, Peshawar, from May 2001 to April 2002.
METHODS
A
[Effect of nicotine on duodenal ulcer].
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In 30 non-smokers and smokers each with and endoscopically ascertained duodenal ulcer the healing of the ulcer, the rate of recidivations, the clinical symptomatology and the duration of the unability to work were examined. In these cases in the group of patients with use of nicotine a delayed
Effects of nicotine on activity and stress-induced gastric ulcers in rats.
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Nicotine is known to influence locomotor activity. The alkaloid also intensifies gastric ulcer formation in stressed rats. The effects of nicotine on locomotor activity in relation to gastric lesions induced by restraint at 4 degrees C for 2 h (stress) were, therefore, studied. Ten-day treatment
Tobacco cigarette smoke aggravates gastric ulcer in rats by attenuation of ulcer margin hyperemia.
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We tested the hypothesis that attenuation of the hyperemia at the margin of acetic acid-induced gastric ulcer in rats by tobacco cigarette smoke will increase the size of the ulcer in the acute and the healing stages. Compared with the adjacent mucosa, blood flow measured by hydrogen gas clearance
The anti-ulcer drug sucralfate does not affect gastric nicotine levels.
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OBJECTIVE
It has been claimed that sucralfate can overcome the negative effects of nicotine in patients with peptic ulcer disease, although the possible mechanism being unknown. This study was performed in order to test whether sucralfate was capable of binding intragastric nicotine, thus making it
Nicotine and ascorbic acid effects on cold-restraint ulcers in rats.
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Rats were orally administered 1-ascorbic acid, nicotine 1-ascorbic acid and nicotine, or distilled water for 10 days. Following this treatment they were fasted for 24 h and then restrained in a cold environment for 2 h. Nicotine alone produced significantly more gastric ulcers than any other
Recurrent aphthous ulcers among tobacco users- hospital based study.
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BACKGROUND
Current evidence shows ambiguous relationship between tobacco use and the occurrence of aphthous.
OBJECTIVE
We studied the relationship between the occurrence of recurrent aphthous ulcers and various forms of tobacco usage.
METHODS
A hospital based case control study was carried out in a
Nicotine and gastric ulcers in stress.
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Chronic nicotine treatment worsens stomach mucosal damage by cold (4 degrees C) and restraint (stress): it dose- and time-dependently intensifies stress-evoked gastric glandular ulceration, mast cell degranulation and motility. Nicotine 50 micrograms/ml drinking water, given ad libitum to female
Tobacco cigarette smoke attenuates duodenal ulcer margin hyperemia in the rat. Comparison of IAP clearance and hydrogen gas clearance techniques for measurements of gastrointestinal blood flow.
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The hyperemia at the duodenal ulcer margin is important for ulcer healing. We studied the effect of tobacco cigarette smoke on the hyperemia at the margin of mepirizole-induced duodenal ulcer. Duodenal mucosal blood flow values measured by iodo[14C]antipyrine (IAP) autoradiography and hydrogen gas
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